نتایج جستجو برای: syn dienvelope

تعداد نتایج: 6083  

Journal: :The Journal of biological chemistry 2012
Bruno Fauvet Martial K Mbefo Mohamed-Bilal Fares Carole Desobry Sarah Michael Mustafa T Ardah Elpida Tsika Philippe Coune Michel Prudent Niels Lion David Eliezer Darren J Moore Bernard Schneider Patrick Aebischer Omar M El-Agnaf Eliezer Masliah Hilal A Lashuel

Since the discovery and isolation of α-synuclein (α-syn) from human brains, it has been widely accepted that it exists as an intrinsically disordered monomeric protein. Two recent studies suggested that α-syn produced in Escherichia coli or isolated from mammalian cells and red blood cells exists predominantly as a tetramer that is rich in α-helical structure (Bartels, T., Choi, J. G., and Selk...

2018
Simona Daniele Daniela Frosini Deborah Pietrobono Lucia Petrozzi Annalisa Lo Gerfo Filippo Baldacci Jonathan Fusi Chiara Giacomelli Gabriele Siciliano Maria Letizia Trincavelli Ferdinando Franzoni Roberto Ceravolo Claudia Martini Ubaldo Bonuccelli

Neurodegenerative disorders (NDs) are characterized by abnormal accumulation/misfolding of specific proteins, primarily α-synuclein (α-syn), β-amyloid1-42 (Aβ1-42) and tau, in both brain and peripheral tissues. In addition to oligomers, the role of the interactions of α-syn with Aβ or tau has gradually emerged. Nevertheless, despite intensive research, NDs have no accepted peripheral markers fo...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2016
Jianhua Xu Xin-Sheng Wu Jiansong Sheng Zhen Zhang Hai-Yuan Yue Lixin Sun Carmelo Sgobio Xian Lin Shiyong Peng Yinghui Jin Lin Gan Huaibin Cai Ling-Gang Wu

UNLABELLED α-Synuclein (α-syn) missense and multiplication mutations have been suggested to cause neurodegenerative diseases, including Parkinson's disease (PD) and dementia with Lewy bodies. Before causing the progressive neuronal loss, α-syn mutations impair exocytosis, which may contribute to eventual neurodegeneration. To understand how α-syn mutations impair exocytosis, we developed a mous...

2014
Hien T. Tran Charlotte Hiu-Yan Chung Michiyo Iba Bin Zhang John Q. Trojanowski Kelvin C. Luk Virginia M.Y. Lee

Accumulation of misfolded alpha-synuclein (α-syn) into Lewy bodies (LBs) and Lewy neurites (LNs) is a major hallmark of Parkinson's disease (PD) and dementia with LBs (DLB). Recent studies showed that synthetic preformed fibrils (pffs) recruit endogenous α-syn and induce LB/LN pathology in vitro and in vivo, thereby implicating propagation and cell-to-cell transmission of pathological α-syn as ...

Journal: :Journal of neuropathology and experimental neurology 2000
V Askanas W K Engel R B Alvarez J McFerrin A Broccolini

Alpha-synuclein (alpha-syn) is an important component of neuronal and glial inclusions in brains of patients with several neurodegenerative disorders. Sporadic inclusion-body myositis (s-IBM) is the most common progressive muscle disease of older patients. Its muscle phenotype shows several similarities with Alzheimer disease brain. A distinct feature of s-IBM pathology is specific vacuolar deg...

2010
Hideya Mizuno Nobuhiro Fujikake Keiji Wada Yoshitaka Nagai

α-Synuclein (α-Syn) is a major component of protein inclusions known as Lewy bodies, which are hallmarks of synucleinopathies such as Parkinson's disease (PD). The α-Syn gene is one of the familial PD-causing genes and is also associated with an increased risk of sporadic PD. Numerous studies using α-Syn expressing transgenic animals have indicated that α-Syn plays a critical role in the common...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2011
Beate Winner Roberto Jappelli Samir K Maji Paula A Desplats Leah Boyer Stefan Aigner Claudia Hetzer Thomas Loher Marçal Vilar Silvia Campioni Christos Tzitzilonis Alice Soragni Sebastian Jessberger Helena Mira Antonella Consiglio Emiley Pham Eliezer Masliah Fred H Gage Roland Riek

The aggregation of proteins into oligomers and amyloid fibrils is characteristic of several neurodegenerative diseases, including Parkinson disease (PD). In PD, the process of aggregation of α-synuclein (α-syn) from monomers, via oligomeric intermediates, into amyloid fibrils is considered the disease-causative toxic mechanism. We developed α-syn mutants that promote oligomer or fibril formatio...

Journal: :Molecular medicine reports 2015
Xiaofeng Zhao Xicui Sun Song Cai Dongzhi Ran Ying Yan Zhong Pei

α-Synuclein (α-Syn) is hypothesized to have a critical role in sporadic and genetic cases of Parkinson's disease (PD) in which Lewy bodies, as the hallmark of PD, are formed from abnormal aggregates of α-Syn. To determine the role of α-Syn in the motor and cognitive dysfunction observed in PD, a Drosophila melanogaster model was established to investigate the electrophysiological and ethologica...

Journal: :Human molecular genetics 2011
Xianpeng Liu Yong Joo Lee Liang-Chun Liou Qun Ren Zhaojie Zhang Shaoxiao Wang Stephan N Witt

Hydroxyurea (HU) inhibits ribonucleotide reductase (RNR), which catalyzes the rate-limiting synthesis of deoxyribonucleotides for DNA replication. HU is used to treat HIV, sickle-cell anemia and some cancers. We found that, compared with vector control cells, low levels of alpha-synuclein (α-syn) protect S. cerevisiae cells from the growth inhibition and reactive oxygen species (ROS) accumulati...

2007
Vrizlynn L. L. Thing Morris Sloman Naranker Dulay

In this paper, we analyze the stateless SYNSYN&ACK and SYN-FIN/RST detection mechanisms for TCP SYN attacks. We indicate the inherent vulnerability of the SYN-FIN/RST detection mechanism caused by the computation of the RST packet counts. We indicate why SYNSYN&ACK is a more efficient and reliable detection mechanism than SYN-FIN/RST. We come up with ‘Bot Buddies’ for TCP SYN attacks and explai...

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