نتایج جستجو برای: runx3

تعداد نتایج: 745  

2014
Giselle Sek Suan Nah Boon-Hui Tay Sydney Brenner Motomi Osato Byrappa Venkatesh

The cyclostomes (jawless vertebrates), comprising lampreys and hagfishes, are the sister group of jawed vertebrates (gnathostomes) and are hence an important group for the study of vertebrate evolution. In mammals, three Runx genes, Runx1, Runx2 and Runx3, encode transcription factors that are essential for cell proliferation and differentiation in major developmental pathways such as haematopo...

Journal: :Molecular medicine reports 2013
Ming Li Shi-Yun Tan Jun Zhang Hong-Xia You

Paeonol, a major phenolic component of the root bark of Paeonia moutan, is known to exhibit antitumor effects. However, the underlying mechanisms remain unknown. In the present study, the effects of paeonol on cell viability, intracellular calcium concentration and the expression of runt‑related transcription factor 3 (RUNX3) were analyzed in LoVo human colon cancer cells. Results revealed that...

2015
HONG JIANG WEI-WEI YU LU-LU WANG YANG PENG

MicroRNAs (miRNAs) are abnormally expressed in various types of cancer. miR-130a expression and function in gastric cancer has yet to be elucidated. The aim of the present study was to identify the miR-130a expression and function in gastric cancer. miR-130a expression was examined in gastric cancer cell lines and tissues by RT-qPCR. The diagnostic and prognostic significance of miR-130a in gas...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2015
Stefanie C S Schmidt Sizun Jiang Hufeng Zhou Bradford Willox Amy M Holthaus Peter V Kharchenko Eric C Johannsen Elliott Kieff Bo Zhao

Epstein-Barr Virus (EBV) conversion of B-lymphocytes to Lymphoblastoid Cell Lines (LCLs) requires four EBV nuclear antigen (EBNA) oncoproteins: EBNA2, EBNALP, EBNA3A, and EBNA3C. EBNA2 and EBNALP associate with EBV and cell enhancers, up-regulate the EBNA promoter, MYC, and EBV Latent infection Membrane Proteins (LMPs), which up-regulate BCL2 to protect EBV-infected B-cells from MYC proliferati...

Journal: :Arthritis and rheumatism 2013
Maria Apel Steffen Uebe John Bowes Emiliano Giardina Eleanor Korendowych Kristina Juneblad Francesca Pasutto Arif B Ekici Ross McManus Pauline Ho Ian N Bruce Anthony W Ryan Frank Behrens Beate Böhm Heiko Traupe Jörg Lohmann Christian Gieger Heinz-Erich Wichmann Leonid Padyukov Oliver Fitzgerald Gerd-Marie Alenius Neil J McHugh Giuseppe Novelli Harald Burkhardt Anne Barton André Reis Ulrike Hüffmeier

OBJECTIVE Psoriatic arthritis (PsA) is a common inflammatory joint disease distinct from other chronic arthritides and frequently accompanied by psoriasis vulgaris. In a first genome-wide association study (GWAS), we were able to identify several genetic risk factors. However, even combined with previously identified factors, the genetic contribution to disease was not fully explained. Therefor...

2005
Dan R. Littman Tarik Möröy Ichiro Taniuchi Viktor Steimle Christine J. Aldrian Andreas Wuerch Baerbel Grueter Michaela Petter Takeshi Egawa Kirsten Laule-Kilian Yvonne Ludwig Hidehiro Fukuyama Hedda Wardemann Ralph Waldschuetz Marc Ehlers

Journal: :iranian journal of pediatric hematology and oncology 0
mehdi azad department of medical laboratory sciences, faculty of allied medicine, qazvin university of medical sciences, qazvin, i farshad forooghi department of immunology, qazvin university of medical sciences, qazvin, iranسازمان اصلی تایید شده: دانشگاه علوم پزشکی قزوین (qazvin university of medical sciences) hassan ehteram department of pathology, school of medicine, kashan university of medical sciences, kashan, iranسازمان اصلی تایید شده: دانشگاه علوم پزشکی قزوین (qazvin university of medical sciences) mousa vatanmakan department of hematology, faculty of allied medicine, tehran university of medical sciences, tehran, iranسازمان اصلی تایید شده: دانشگاه علوم پزشکی کاشان (kashan university of medical sciences) hajar nasiri hematology-oncology and stem cell transplantation research center, tehran university of medical science, tehran, iranسازمان اصلی تایید شده: دانشگاه علوم پزشکی تهران (tehran university of medical sciences) naser mobarra stem cell research center, department of biochemistry, school of medicine, golestan university of medical sciences, gorسازمان اصلی تایید شده: دانشگاه علوم پزشکی تهران (tehran university of medical sciences)

background: stem cell differentiation is a process composed of vast variety of factors which are controlled by a network of certain mechanisms. this study aims to determine the possible role of dna methylation, a potent regulator of vhl, ecad and runx3 genes during erythroid differentiation driven by mir-451. materials and methods: to determine the methylation status of promoters and the expres...

Journal: :Journal of immunology 2011
Kojo Arita Shota Endo Tomonori Kaifu Kohji Kitaguchi Akira Nakamura Hidetaka Ohmori Kazuyoshi Kohu Masanobu Satake Toshiyuki Takai

Cells in the immune system are regulated positively or negatively by sets of receptor pairs that conduct balanced, activating, or inhibitory intracellular signaling. One such receptor pair termed paired Ig-like receptor (PIR) is composed of the inhibitory PIR-B and its activating isoform, PIR-A. Upon binding to their shared ligand, MHC class I molecules, these receptors control the threshold fo...

2018
Hamid Sayar Yan Liu Rui Gao Mohammad Abu Zaid Larry D. Cripe Jill Weisenbach Katie J. Sargent Mehdi Nassiri Lang Li Heiko Konig Attaya Suvannasankha Feng Pan Rajasubramaniam Shanmugam Chirayu Goswami Reuben Kapur Mingjiang Xu H. Scott Boswell

Co-occurrence of Flt3ITD and TET2 mutations provoke an animal model of AML by epigenetic repression of Wnt pathway antagonists, including RUNX3, and by hyperexpression of ID1, encoding Wnt agonist. These affect HOXA over-expression and treatment resistance. A comparable epigenetic phenotype was identified among adult AML patients needing novel intervention. We chose combinations of targeted age...

2014
Zhenfei Wang Rinuo Dao Luri Bao Yanhua Dong Haiyang Wang Pengyong Han Yongli Yue Haiquan Yu

The tumour suppressor gene silencing and proto-oncogene activation caused by epigenetic alterations plays an important role in the initiation and progression of cancer. Re-establishing the balance between the expression of tumour suppressor genes and proto-oncogenes by epigenetic modulation is a promising strategy for cancer treatment. In this study, we investigated whether cancer cells can be ...

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