BACKGROUND Atrioventricular (AV) nodal reentry is a relatively common cause of regular, narrow QRS tachycardia. The underlying basis for this arrhythmia is functional (and anatomic) duality of pathways in the region of the AV node, although the exact boundaries of the reentrant circuit have not been convincingly defined. During the more common type of AV nodal reentry (seen in approximately 90%...

The mechanisms underlying the development of ventricular fibrillation (VF) during early myocardial ischemia were assessed by use of a computerized three-dimensional mapping system capable of recording simultaneously from 232 intramural recording sites throughout the entire feline heart in vivo. Occlusion of the proximal left anterior descending coronary artery led to ventricular tachycardia (VT...

ndovascular management of acute and chronic aortic dissection was pioneered early last decade, with reported survival rates as high as 100%, and is fast becoming an established treatment modality for complicated aortic dissection. The conventional endovascular approach involves the closure of the primary entry tear by endograft with induction of false lumen thrombosis and subsequent aortic remo...

Since their development at the end of 50s, panel methods were widely used for fast simulation aerospace objects reentry. Although improvements proposed continuum regime formulations, bridging functions usually employed in transitional did not go through major changes since then. With current interest designing Very Low Earth Orbit satellites and more efficient reentry vehicles, a greater level ...

Reentry in nervous systems is the ongoing bidirectional exchange of signals along reciprocal axonal fibers linking two or more brain areas. The hypothesis that reentrant signaling serves as a general mechanism to couple the functioning of multiple areas of the cerebral cortex and thalamus was first proposed in 1977 and 1978 (Edelman, 1978). A review of the amount and diversity of supporting exp...

Arrhythmia Mechanisms and Therapy. An understanding of the mechanisms responsible for experimental and clinical arrhythmias has led to the development of mechanism-specific therapies for ventricular tachycardia. These include: (1) beta blockade for catecholamine-sensitive ventricular tachycardia; (2) verapamil for ventricular tachycardia possibly due to triggered activity; (3) pacing, isoproter...