نتایج جستجو برای: permeabilization

تعداد نتایج: 2702  

Journal: :Biochimica et Biophysica Acta (BBA) - Biomembranes 2001

2012
Cláudia S. F. Queiroga Ana S. Almeida Helena L. A. Vieira

MITOCHONDRIA PRESENT TWO KEY ROLES ON CELLULAR FUNCTIONING: (i) cell metabolism, being the main cellular source of energy and (ii) modulation of cell death, by mitochondrial membrane permeabilization. Carbon monoxide (CO) is an endogenously produced gaseoustransmitter, which presents several biological functions and is involved in maintaining cell homeostasis and cytoprotection. Herein, mitocho...

Journal: :Molecular cell 2015
Thibaud T Renault Konstantinos V Floros Rana Elkholi Kelly-Ann Corrigan Yulia Kushnareva Shira Y Wieder Claudia Lindtner Madhavika N Serasinghe James J Asciolla Christoph Buettner Donald D Newmeyer Jerry E Chipuk

Proapoptotic BCL-2 proteins converge upon the outer mitochondrial membrane (OMM) to promote mitochondrial outer membrane permeabilization (MOMP) and apoptosis. Here we investigated the mechanistic relationship between mitochondrial shape and MOMP and provide evidence that BAX requires a distinct mitochondrial size to induce MOMP. We utilized the terminal unfolded protein response pathway to sys...

Journal: :Biochemical Society transactions 2014
Ana M Villamil Giraldo Hanna Appelqvist Thomas Ederth Karin Öllinger

Lysosomes are acidic organelles essential for degradation, signalling and cell homoeostasis. In addition, they play a key role in cell death. Permeabilization of the lysosomal membrane and release of hydrolytic enzymes to the cytosol accompanies apoptosis signalling in several systems. The regulatory mechanism of lysosomal stability is, however, poorly understood. Lipophilic or amphiphilic comp...

Journal: :Molecular biology of the cell 2002
Gemma Way Nicholas Morrice Carl Smythe Antony J O'Sullivan

After permeabilization with the pore-forming toxin streptolysin-O mast cells can be triggered to secrete by addition of both calcium and a GTP analogue. If stimulation is delayed after permeabilization, there is a progressive decrease in the extent of secretion upon stimulation, eventually leading to a complete loss of the secretory response. This loss of secretory response can be retarded by t...

2009
Kathleen M. Averette Matthew R. Pratt Yanan Yang Sara Bassilian Julian P. Whitelegge Joseph A. Loo Tom W. Muir Kenneth A. Bradley

NOD-like receptors (NLRs) are a group of cytoplasmic molecules that recognize microbial invasion or 'danger signals'. Activation of NLRs can induce rapid caspase-1 dependent cell death termed pyroptosis, or a caspase-1 independent cell death termed pyronecrosis. Bacillus anthracis lethal toxin (LT), is recognized by a subset of alleles of the NLR protein Nlrp1b, resulting in pyroptotic cell dea...

Journal: :Developmental cell 2008
Ann Cassidy-Stone Jerry E Chipuk Elena Ingerman Cheng Song Choong Yoo Tomomi Kuwana Mark J Kurth Jared T Shaw Jenny E Hinshaw Douglas R Green Jodi Nunnari

Mitochondrial fusion and division play important roles in the regulation of apoptosis. Mitochondrial fusion proteins attenuate apoptosis by inhibiting release of cytochrome c from mitochondria, in part by controlling cristae structures. Mitochondrial division promotes apoptosis by an unknown mechanism. We addressed how division proteins regulate apoptosis using inhibitors of mitochondrial divis...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید