نتایج جستجو برای: olig2

تعداد نتایج: 604  

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2007
Brahim Nait-Oumesmar Nathalie Picard-Riera Christophe Kerninon Laurence Decker Danielle Seilhean Günter U Höglinger Etienne C Hirsch Richard Reynolds Anne Baron-Van Evercooren

In multiple sclerosis (MS), oligodendrocyte and myelin destruction lead to demyelination with subsequent axonal loss. Experimental demyelination in rodents has highlighted the activation of the subventricular zone (SVZ) and the involvement of progenitor cells expressing the polysialylated form of neural cell adhesion molecule (PSA-NCAM) in the repair process. In this article, we studied the dis...

2017
Jichao Yuan Hongfei Ge Wei Liu Haitao Zhu Yaxing Chen Xuan Zhang Yang Yang Yi Yin Weixiang Chen Wanjiang Wu Yunfeng Yang Jiangkai Lin

Neural stem/progenitor cells (NSPCs) are an important source of cells for cell replacement therapy after nerve injury. How to induce NSPCs differentiation towards neurons and oligodendrocytes is a challenging issue in neuroscience research. In the present study, we polarized microglia into M1 and M2 phenotype, used their supernatants to induce NSPCs differentiation, and investigated the effects...

2017
Tao Deng Yuri Postnikov Shaofei Zhang Lillian Garrett Lore Becker Ildikó Rácz Sabine M. Hölter Wolfgang Wurst Helmut Fuchs Valerie Gailus-Durner Martin Hrabe de Angelis Michael Bustin

An interplay between the nucleosome binding proteins H1 and HMGN is known to affect chromatin dynamics, but the biological significance of this interplay is still not clear. We find that during embryonic stem cell differentiation loss of HMGNs leads to down regulation of genes involved in neural differentiation, and that the transcription factor OLIG2 is a central node in the affected pathway. ...

2014
Fereshteh Pourabdolhossein Sabah Mozafari Ghislaine Morvan-Dubois Javad Mirnajafi-Zadeh Alejandra Lopez-Juarez Jacqueline Pierre-Simons Barbara A. Demeneix Mohammad Javan

BACKGROUND Inhibitory factors have been implicated in the failure of remyelination in demyelinating diseases. Myelin associated inhibitors act through a common receptor called Nogo receptor (NgR) that plays critical inhibitory roles in CNS plasticity. Here we investigated the effects of abrogating NgR inhibition in a non-immune model of focal demyelination in adult mouse optic chiasm. METHODO...

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