نتایج جستجو برای: ileitis

تعداد نتایج: 630  

Journal: :Journal of immunology 2008
Rie Kajino-Sakamoto Maiko Inagaki Elisabeth Lippert Shizuo Akira Sylvie Robine Kunihiro Matsumoto Christian Jobin Jun Ninomiya-Tsuji

Recent studies have revealed that TAK1 kinase is an essential intermediate in several innate immune signaling pathways. In this study, we investigated the role of TAK1 signaling in maintaining intestinal homeostasis by generating enterocyte-specific constitutive and inducible gene-deleted TAK1 mice. We found that enterocyte-specific constitutive TAK1-deleted mice spontaneously developed intesti...

Journal: :American journal of physiology. Gastrointestinal and liver physiology 2012
P B Ernst L D Erickson W M Loo K G Scott E B Wiznerowicz C C Brown F J Torres-Velez M S Alam S G Black M McDuffie S H Feldman J L Wallace G W McKnight I T Padol R H Hunt K S Tung

SAMP1/YitFcs mice serve as a model of Crohn's disease, and we have used them to assess gastritis. Gastritis was compared in SAMP1/YitFcs, AKR, and C57BL/6 mice by histology, immunohistochemistry, and flow cytometry. Gastric acid secretion was measured in ligated stomachs, while anti-parietal cell antibodies were assayed by immunofluorescence and enzyme-linked immunosorbent spot assay. SAMP1/Yit...

2014
Cátia Vieira Maria Teresa Magalhães-Cardoso Fátima Ferreirinha Isabel Silva Ana Sofia Dias Julie Pelletier Jean Sévigny Paulo Correia-de-Sá

Purinergic signalling is remarkably plastic during gastrointestinal inflammation. Thus, selective drugs targeting the "purinome" may be helpful for inflammatory gastrointestinal diseases. The myenteric neuromuscular transmission of healthy individuals is fine-tuned and controlled by adenosine acting on A(2A) excitatory receptors. Here, we investigated the neuromodulatory role of adenosine in TN...

Journal: :American journal of physiology. Gastrointestinal and liver physiology 2002
Paula Miceli Gerald P Morris Wallace K MacNaughton Stephen Vanner

The efferent secretomotor activity of capsaicin-sensitive nerves was monitored during the evolution of 2,4,6-trinitrobenzenesulfonic acid (TNBS)-induced ileitis in the guinea pig by recording changes in short-circuit current (DeltaI(sc)) in response to capsaicin, substance P (SP), and carbachol. Submucosal-mucosal preparations mounted in standard Ussing chambers were studied at time 0, at 8 h, ...

Journal: :Journal of the Japanese Association for Infectious Diseases 1974

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