نتایج جستجو برای: hla dq8

تعداد نتایج: 40088  

2012
B. Admou L. Essaadouni K. Krati K. Zaher M. Sbihi L. Chabaa B. Belaabidia A. Alaoui-Yazidi

The nonclassic clinical presentation of celiac disease (CD) becomes increasingly common in physician's daily practice, which requires an awareness of its many clinical faces with atypical, silent, and latent forms. Besides the common genetic background (HLA DQ2/DQ8) of the disease, other non-HLA genes are now notably reported with a probable association to atypical forms. The availability of hi...

Journal: :Journal of the Canadian Association of Gastroenterology 2021

2014
Jihane Romanos Anna Rosén Vinod Kumar Gosia Trynka Lude Franke Agata Szperl Javier Gutierrez-Achury Cleo C van Diemen Roan Kanninga Soesma A Jankipersadsing Andrea Steck Georges Eisenbarth David A van Heel Bozena Cukrowska Valentina Bruno Maria Cristina Mazzilli Concepcion Núñez Jose Ramon Bilbao M Luisa Mearin Donatella Barisani Marian Rewers Jill M Norris Anneli Ivarsson H Marieke Boezen Edwin Liu Cisca Wijmenga Cristian Scerri Tunde Koltai Sanja Kolaček Zrinka Mišak Slaven Abdović Sibylle Koletzko Gertraud Osiander Katharina Werkstetter Eckart Mummert Ilma R Korponay-Szabo Judit Gyimesi Raanan Shamir Corina Hartman Enzo Bravi Marco Poles Renata Auricchio G Gianna Giovamma Limongelli Luigi Greco Riccardo Troncone Vincenzo Villanacci Jacques G Bindels Ronald Brand Bibi Funke Kupper Caroline E Hogen Esch Erica G Hopman Frits Koning Yvonne Kooy-Winkelaar Chantal te Marvelde Hein Putter Els Stoopman Sabine Vriezinga Ludvig M Sollid Melinda Ráki Ania Chmielewska Piotr Dziechciarz Małgorzata Pieścik-Lech Hania Szajewska Anna Szaflarska-Szczepanik Gemma Castillejo Amalia Capilla Vicente Varea Carmen Ribes-Koninckx Anna Lopez Paula Crespo Eva Martinez Isabel Polanco Lotta Högberg Lars Stenhammar Annelie Carlsson Charlotta Webb Solveig Hammarroth Olle Hernell Carina Lagerqvist Anna Myléus Katrina Nordyke Fredrik Norström Olof Sandström Stig Wall Eva Karlsson

BACKGROUND The majority of coeliac disease (CD) patients are not being properly diagnosed and therefore remain untreated, leading to a greater risk of developing CD-associated complications. The major genetic risk heterodimer, HLA-DQ2 and DQ8, is already used clinically to help exclude disease. However, approximately 40% of the population carry these alleles and the majority never develop CD. ...

2014
Justin L. McCarville Yotam Nisemblat Heather J. Galipeau Jennifer Jury Rinat Tabakman Ad Cohen Esmira Naftali Bela Neiman Efrat Halbfinger Joseph A. Murray Arivarasu N. Anbazhagan Pradeep K. Dudeja Alexander Varvak Jean-Christophe Leroux Elena F. Verdu

Celiac disease (CD) is an autoimmune disorder in individuals that carry DQ2 or DQ8 MHC class II haplotypes, triggered by the ingestion of gluten. There is no current treatment other than a gluten-free diet (GFD). We have previously shown that the BL-7010 copolymer poly(hydroxyethyl methacrylate-co-styrene sulfonate) (P(HEMA-co-SS)) binds with higher efficiency to gliadin than to other proteins ...

Journal: :Revista de Gastroenterología de México (English Edition) 2019

Journal: :The Journal of clinical investigation 2003
Giuseppina Pennesi Mary J Mattapallil Shu-Hui Sun Dody Avichezer Phyllis B Silver Zaruhi Karabekian Chella S David Paul A Hargrave J Hugh McDowell W Clay Smith Barbara Wiggert Larry A Donoso Chi-Chao Chan Rachel R Caspi

Experimental autoimmune uveitis (EAU) is a disease of the neural retina induced by immunization with retinal antigens, such as interphotoreceptor retinoid-binding protein (IRBP) and arrestin (retinal soluble antigen, S-Ag). EAU serves as a model for human autoimmune uveitic diseases associated with major histocompatibility complex (HLA) genes, in which patients exhibit immunological responses t...

Journal: :Arthritis Research 2000
Louise Berg Johan Rönnelid Carani B Sanjeevi Jon Lampa Lars Klareskog

INTRODUCTION Despite much work over past decades, whether antigen-specific immune reactions occur in rheumatoid arthritis (RA) and to what extent such reactions are directed towards joint-specific autoantigens is still questionable. One strong indicator for antigenic involvement in RA is the fact that certain major histocompatibility complex (MHC) class II genotypes [human leucocyte antigen (HL...

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