نتایج جستجو برای: hiv عفونتهای قارچی سایتوکینهای il

تعداد نتایج: 350334  

2016
Kaku A. So-Armah Janet P. Tate Chung-Chou H. Chang Adeel A. Butt Mariana Gerschenson Cynthia L. Gibert David Leaf David Rimland Maria C. Rodriguez-Barradas Matthew J. Budoff Jeffrey H. Samet Lewis H. Kuller Steven G. Deeks Kristina Crothers Russell P. Tracy Heidi M. Crane Mohammad M. Sajadi Hilary A. Tindle Amy C. Justice Matthew S. Freiberg

BACKGROUND HIV infection and biomarkers of inflammation [measured by interleukin-6 (IL-6)], monocyte activation [soluble CD14 (sCD14)], and coagulation (D-dimer) are associated with morbidity and mortality. We hypothesized that these immunologic processes mediate (explain) some of the excess risk of mortality among HIV infected (HIV+) versus uninfected people independently of comorbid diseases....

Journal: :Molecular pharmacology 2006
Patrick T Ronaldson Reina Bendayan

In this work, we examined the ability of gp120, a human immunodeficiency virus-1 (HIV-1) viral envelope glycoprotein, to trigger the innate immune response in astrocytes, an HIV-1 brain cellular target, and we investigated the functional expression of the ATP-binding cassette membrane transporter P-glycoprotein (P-gp) in primary cultures of rat astrocytes treated with gp120 or cytokines [tumor ...

پایان نامه :وزارت بهداشت، درمان و آموزش پزشکی - دانشگاه علوم پزشکی و خدمات بهداشتی درمانی تهران 1368

چکیده ندارد.

پایان نامه :وزارت بهداشت، درمان و آموزش پزشکی - دانشگاه علوم پزشکی و خدمات بهداشتی درمانی تهران 1369

چکیده ندارد.

2011
Abigail Eastburn Rebecca Scherzer Andrew R. Zolopa Constance Benson Russell Tracy Tri Do Peter Bacchetti Michael Shlipak Carl Grunfeld Phyllis C. Tien

BACKGROUND Whether HIV viremia, particularly at low levels is associated with inflammation, increased coagulation, and all-cause mortality is unclear. METHODS The associations of HIV RNA level with C-reactive protein (CRP), fibrinogen, interleukin (IL)-6 and mortality were evaluated in 1116 HIV-infected participants from the Study of Fat Redistribution and Metabolic Change in HIV infection. H...

Journal: :The Journal of infectious diseases 2001
A Weinberg D A Wohl R J Barrett C van der Horst

Cytomegalovirus (CMV)-immune recovery was characterized in human immunodeficiency virus (HIV)-infected patients receiving highly active antiretroviral therapy. CMV lymphocyte proliferation (LP), responder-cell frequency (RCF), and interferon (IFN)-gamma and interleukin (IL)-2 secretion were studied in CMV-seropositive HIV-infected patients and in CMV-seropositive HIV-uninfected control subjects...

Journal: :The Journal of infectious diseases 2014
Carey L Shive Joseph C Mudd Nicholas T Funderburg Scott F Sieg Benjamin Kyi Doug A Bazdar Davide Mangioni Andrea Gori Jeffrey M Jacobson Ari D Brooks Jeffrey Hardacre John Ammori Jacob D Estes Timothy W Schacker Benigno Rodriguez Michael M Lederman

BACKGROUND Systemic inflammation has been linked to a failure to normalize CD4(+) T-cell numbers in treated human immunodeficiency virus (HIV) infection. Although inflammatory cytokines such as interleukin 6 (IL-6) are predictors of disease progression in treated HIV infection, it is not clear how or whether inflammatory mediators contribute to immune restoration failure. METHODS We examined ...

Journal: :Intervirology 2014
Juan C Hernandez Eicke Latz Silvio Urcuqui-Inchima

BACKGROUND/AIMS Inflammasomes are multimolecular complexes that regulate caspase-1. They act as sensors for endogenous and exogenous signals, and mediate the processing of pro-IL-1β into its secreted, biologically active form. The NLRP3 inflammasome and IL-1β are particularly interesting because they are required for efficient control of viral infections. Indeed, HIV-1 induces expression of NLR...

Journal: :Journal of Immunology 2023

Abstract Background: HIV-associated neurocognitive dysfunction persists despite suppressive antiretroviral therapy (ART). Extracellular vesicles (EVs) are emerging modulators of immunological responses. We have previously shown that EVs expressing monocyte activation and neuronal markers associated with HIV cognitive impairment (CI). Whether these EV changes observed in people (PWH) influence i...

Journal: :Journal of immunology 2008
Marcel F Nold Claudia A Nold-Petry Gregory B Pott Jarod A Zepp Milene T Saavedra Soo-Hyun Kim Charles A Dinarello

IL-32, a proinflammatory cytokine that activates the p38MAPK and NF-kappaB pathways, induces other cytokines, for example, IL-1beta, IL-6, and TNF-alpha. This study investigated the role of endogenous IL-32 in HIV-1 infection by reducing IL-32 with small interfering (si)RNA in freshly infected PBMC and in the latently infected U1 macrophage cell line. When PBMC were pretreated with siRNA to IL-...

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