نتایج جستجو برای: fatal cardiomyopathy

تعداد نتایج: 82571  

Journal: :Human molecular genetics 1998
F Degoul H Brulé C Cepanec M Helm C Marsac J Leroux R Giegé C Florentz

A growing number of mutated mitochondrial tRNA genes have been found associated with severe human diseases. To investigate the potential interference of such mutations with the primordial function of tRNAs, i.e. their aminoacylation by cognate aminoacyl-tRNA synthetases, a human mitochondrial in vitro aminoacylation system specific for isoleucine has been established. Both native tRNAIleand iso...

Journal: :Archives of pathology & laboratory medicine 2014
Christina G Loporcaro David J Tester Joseph J Maleszewski Teresa Kruisselbrink Michael J Ackerman

Annually, the sudden death of thousands of young people remains inadequately explained despite medicolegal investigation. Postmortem genetic testing for channelopathies/cardiomyopathies may illuminate a potential cardiac mechanism and establish a more accurate cause and manner of death and provide an actionable genetic marker to test surviving family members who may be at risk for a fatal arrhy...

2015
Weina Cui Albert Jang Pengyuan Zhang Brian Thompson DeWayne Townsend Joseph M. Metzger Jianyi Zhang Yaoliang Tang

BACKGROUND Duchenne muscular dystrophy (DMD) is the most common fatal form of muscular dystrophy characterized by striated muscle wasting and dysfunction. Patients with DMD have a very high incidence of heart failure, which is increasingly the cause of death in DMD patients. We hypothesize that in the in vivo system, the dystrophic cardiac muscle displays bioenergetic deficits prior to any func...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2014
Helena M Viola Abbie M Adams Stefan M K Davies Susan Fletcher Aleksandra Filipovska Livia C Hool

Duchenne muscular dystrophy is a fatal X-linked disease characterized by the absence of dystrophin. Approximately 20% of boys will die of dilated cardiomyopathy that is associated with cytoskeletal protein disarray, contractile dysfunction, and reduced energy production. However, the mechanisms for altered energy metabolism are not yet fully clarified. Calcium influx through the L-type Ca(2+) c...

Round heart disease is a disease of unknown etiology in turkey. The objective of this study was to examinethe effect of different amounts of sodium chloride in inducing cardiomyopathy in turkey poults. There are some reports that hypoxia and furazolidone can induce cardiomyopathy in turkey. Two hundred 2-day-old turkey poults were wing-banded and placed randomly into 6 groups and 24 replicates ...

2017
J Hordeaux L Dubreil C Robveille J Deniaud Q Pascal B Dequéant J Pailloux L Lagalice M Ledevin C Babarit P Costiou F Jamme M Fusellier Y Mallem C Ciron C Huchet C Caillaud M-A Colle

Pompe disease is a lysosomal storage disorder caused by acid-α-glucosidase (GAA) deficiency, leading to glycogen storage. The disease manifests as a fatal cardiomyopathy in infantile form. Enzyme replacement therapy (ERT) has recently prolonged the lifespan of these patients, revealing a new natural history. The neurologic phenotype and the persistence of selective muscular weakness in some pat...

2013
Melek Zekiye Ulucam

Arrhythmogenic right ventricular dysplasia is an inherited fatal cardiomyopathy causing fat and connective tissue infiltration of right ventricle. In advanced disease, it may also involve left ventricle. Heart failure, arrhythmias and sudden death are main clinical components. Many different genetic mutations can cause this cardiomyopathy. All mutations lead to the formation of the disease are ...

Journal: :Circulation 2001
F Ichida S Tsubata K R Bowles N Haneda K Uese T Miyawaki W J Dreyer J Messina H Li N E Bowles J A Towbin

BACKGROUND Mutations in the gene G4.5 result in a wide spectrum of severe infantile cardiomyopathic phenotypes, including isolated left ventricular noncompaction (LVNC), as well as Barth syndrome (BTHS) with dilated cardiomyopathy (DCM). The purpose of this study was to investigate patients with LVNC or BTHS for mutations in G4.5 or other novel genes. METHODS AND RESULTS DNA was isolated from...

Journal: :International heart journal 2015
Masataka Yoshinaga Daiji Yoshikawa Hideki Ishii Akihiro Hirashiki Takahiro Okumura Aki Kubota Shinichi Sakai Ken Harada Fuji Somura Tomofumi Mizuno Wakaya Fujiwara Hiroatsu Yokoi Mutsuharu Hayashi Junichi Ishii Yukio Ozaki Toyoaki Murohara Yukihiko Yoshida Tetsuya Amano Hideo Izawa

Hypertrophic cardiomyopathy (HCM) has various morphological and clinical features. A decade has passed since the previous survey of the epidemiological and clinical characteristics of Japanese HCM patients. The Aichi Hypertrophic Cardiomyopathy (AHC) Registry is based on a prospective multicenter observational study of HCM patients. The clinical characteristics of 42 ambulant HCM patients follo...

Journal: :American journal of physiology. Heart and circulatory physiology 2013
Shikha Mishra Jian Guan Eva Plovie David C Seldin Lawreen H Connors Giampaolo Merlini Rodney H Falk Calum A MacRae Ronglih Liao

Systemic amyloid light-chain (AL) amyloidosis is associated with rapidly progressive and fatal cardiomyopathy resulting from the direct cardiotoxic effects of circulating AL light chain (AL-LC) proteins and the indirect effects of AL fibril tissue infiltration. Cardiac amyloidosis is resistant to standard heart failure therapies, and, to date, there are limited treatment options for these patie...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید