نتایج جستجو برای: dystrophic epidermolysis

تعداد نتایج: 5839  

Journal: :Annals of dermatology 2009
Gyo Shin Kang Woo Tae Ko Jae Hong Kim Sung Min Choi Ae Suk Kim Dong Hoon Kim Moo Kyu Suh

Dystrophic epidermolysis bullosa (DEB) is a rare group of heritable mechanobullous disorders that are characterized by blistering and scarring of the skin and mucosae and these lesions are induced by minor trauma, DEB is also associated with nail dystrophy. DEB can be inherited either in an autosomal recessive or dominant fashion. Regardless of the mode of inheritance, DEB is caused by defects ...

2017
Sofia von Bischhoffshausen Dinka Ivulic Paola Alvarez Victor C. Schuffeneger Juan Idiaquez Constanza Fuentes Pilar Morande Ignacia Fuentes Francis Palisson David L. H. Bennett Margarita Calvo

Small fibres in the skin are vulnerable to damage in metabolic or toxic conditions such as diabetes mellitus or chemotherapy resulting in small fibre neuropathy and associated neuropathic pain. Whether injury to the most distal portion of sensory small fibres due to a primary dermatological disorder can cause neuropathic pain is still unclear. Recessive dystrophic epidermolysis bullosa (RDEB) i...

Journal: :The Journal of investigative dermatology 2008
Tracy Wong Luke Gammon Lu Liu Jemima E Mellerio Patricia J C Dopping-Hepenstal John Pacy George Elia Rosemary Jeffery Irene M Leigh Harshad Navsaria John A McGrath

Recessive dystrophic epidermolysis bullosa (RDEB) is a severe inherited skin-blistering disorder caused by mutations in the COL7A1 gene that lead to reduced type-VII collagen and defective anchoring fibrils at the dermal-epidermal junction (DEJ). Presently there are no effective treatments for this disorder. Recent mouse studies have shown that intradermal injections of normal human fibroblasts...

Journal: :The Journal of investigative dermatology 2001
P B Cserhalmi-Friedman M C Garzon E Guzman A Martinez-Mir W K Chung K Anyane-Yeboa A M Christiano

Peter B Cserhalmi-Friedman, Maria C Garzon, Edwin Guzman, Amalia Martinez-Mir, Wendy K Chung, Kwame Anyane-Yeboa and Angela M Christiano Department of Dermatology, New York, New York, U.S.A. Department of Genetics and Development, New York, New York, U.S.A. Department of Pediatrics, Division of Clinical Genetics, College of Physicians and Surgeons, Columbia University, New York, New York, U.S.A...

Journal: :Pediatric dentistry 2000
A Kostara G J Roberts M Gelbier

PURPOSE This study was performed to compare Dental Age (DA) of children with Dystrophic Epidermolysis Bullosa recessiva (DEBr) with the DA of healthy children. METHODS Orthopantomographs (OPG's) of children with DEBr were compared with those of healthy children. Dental maturity was estimated using Dermirjian's method. A total of 48 pairs of OPG's were compared. RESULTS There was a considera...

Journal: :Clinical genetics 2006
B Drera D Castiglia N Zoppi R Gardella G Tadini G Floriddia N De Luca C Pedicelli S Barlati G Zambruno M Colombi

Dystrophic epidermolysis bullosa (DEB) pruriginosa (DEB-Pr) is a rare variant of DEB due to COL7A1 dominant and recessive mutations, which is characterized by severe itching and lichenoid or nodular prurigo-like lesions, mainly involving the extremities. Less than 30 patients have been described showing variable disease expression, and frequently, delayed age of onset. We report the clinical an...

2017
Patricia Peking Ulrich Koller Blanca Duarte Rodolfo Murillas Susanne Wolf Tobias Maetzig Michael Rothe Thomas Kocher Marta García Gabriele Brachtl Axel Schambach Fernando Larcher Julia Reichelt Johann W. Bauer Eva M. Murauer

Functional impairment or complete loss of type VII collagen, caused by mutations within COL7A1, lead to the severe recessive form of the skin blistering disease dystrophic epidermolysis bullosa (RDEB). Here, we successfully demonstrate RNA trans-splicing as an auspicious repair option for mutations located in a wide range of exons by fully converting an RDEB phenotype in an ex vivo pre-clinical...

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