نتایج جستجو برای: collagen induced arthritis

تعداد نتایج: 1103161  

2012
Fei Bao Pei Wu Na Xiao Frank Qiu Qing-Ping Zeng

BACKGROUND Rheumatoid arthritis (RA) is an inflammatory articular disease with cartilage and bone damage due to hyperplasic synoviocyte invasion and subsequent matrix protease digestion. Although monoclonal antibodies against tumor necrosis factor alpha (TNFα) have been approved for clinical use in patients with RA, desired therapeutic regimens suitable for non-responders are still unavailable ...

2013
Naoto Horai Takaharu Nagaoka Itsuro Higuchi Hayato Kasai Takako Yoshioka Yoshihisa Umekita Koichiro Fukuzaki Ryoichi Nagata Atsuro Miyata Kazuhiro Abeyama

BACKGROUND Not only joint destruction but also muscle wasting due to rheumatoid cachexia has been problem in terms of quality of life of patients with rheumatoid arthritis (RA). In the present study, we performed histopathological examination and assessed relationships between characteristic parameters relating to muscle and joint swelling in a collagen-induced arthritis (CIA) model using cynom...

2017
Marije K. Verheul Michel P. M. Vierboom Bert A. ’t Hart Rene E. M. Toes Leendert A. Trouw

BACKGROUND Rheumatoid factor (RF), anti-citrullinated protein antibodies (ACPA) and anti-carbamylated protein (anti-CarP) antibodies are rheumatoid arthritis (RA)-associated autoantibodies. Besides their presence in human serum, anti-CarP antibodies have also been described in rodent models of arthritis, while ACPA are not consistently detectable. Data on these RA-associated autoantibodies in p...

Journal: :Arthritis Research & Therapy 2008
Melina Daans Rik JU Lories Frank P Luyten

INTRODUCTION Rheumatoid arthritis is a chronic systemic autoimmune disease affecting peripheral joints and leading to loss of joint function. The severity and outcome of disease are dependent on the balance between inflammatory/destructive and homeostatic or repair pathways. Increasing evidence suggests a role for bone morphogenetic protein (BMP) signaling in joint homeostasis and disease. ME...

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