نتایج جستجو برای: bax inhibitor 1 like protein

تعداد نتایج: 4108528  

Journal: :Molecular cell 2009
Fernanda Lisbona Diego Rojas-Rivera Peter Thielen Sebastian Zamorano Derrick Todd Fabio Martinon Alvaro Glavic Christina Kress Jonathan H Lin Peter Walter John C Reed Laurie H Glimcher Claudio Hetz

Adaptation to endoplasmic reticulum (ER) stress depends on the activation of an integrated signal transduction pathway known as the unfolded protein response (UPR). Bax inhibitor-1 (BI-1) is an evolutionarily conserved ER-resident protein that suppresses cell death. Here we have investigated the role of BI-1 in the UPR. BI-1 expression suppressed IRE1alpha activity in fly and mouse models of ER...

Journal: :Journal of virology 2008
Kristi L Norris Richard J Youle

Apoptosis is a host defense mechanism against viruses that can be subverted by viral gene products. Human cytomegalovirus encodes viral mitochondria-localized inhibitor of apoptosis (vMIA; also known as pUL37x1), which is targeted to mitochondria and functions as a potent cell death suppressor by binding to and inhibiting proapoptotic Bcl-2 family members Bax and Bak. vMIA expression also drama...

Journal: :American journal of physiology. Heart and circulatory physiology 2009
Deepa S De Silva Richard M Wilson Christoph Hutchinson Peter C Ip Anthony G Garcia Steve Lancel Masa Ito David R Pimentel Flora Sam

Aldosterone induces extracellular signal-regulated kinase (ERK)-dependent cardiac remodeling. Fenofibrate improves cardiac remodeling in adult rat ventricular myocytes (ARVM) partly via inhibition of aldosterone-induced ERK1/2 phosphorylation and inhibition of matrix metalloproteinases. We sought to determine whether aldosterone caused apoptosis in cultured ARVM and whether fenofibrate ameliora...

2015
Genkai Guo Yan Meng Wei Tan Yunfei Xia Chun Cheng Xiaolan Chen Zhifeng Gu

Previous studies indicated that bone marrow mesenchymal stem cells (BM-MSCs) from patients with systemic lupus erythematosus (SLE) exhibited the phenomenon of apoptosis. In this study, we aimed to investigate whether apoptosis of BM-MSCs from SLE patients were dysregulated. In this paper, endoplasmic reticulum stress (ERS) was evidenced by increased expression of phosphorylated protein kinase R...

Journal: :The Journal of Cell Biology 2000
Saadi Ghatan Stephen Larner Yoshito Kinoshita Michal Hetman Leena Patel Zhengui Xia Richard J. Youle Richard S. Morrison

Nitric oxide is a chemical messenger implicated in neuronal damage associated with ischemia, neurodegenerative disease, and excitotoxicity. Excitotoxic injury leads to increased NO formation, as well as stimulation of the p38 mitogen-activated protein (MAP) kinase in neurons. In the present study, we determined if NO-induced cell death in neurons was dependent on p38 MAP kinase activity. Sodium...

Journal: :Hypertension 2010
Takuya Kishi Yoshitaka Hirooka Satomi Konno Kiyohiro Ogawa Kenji Sunagawa

In the rostral ventrolateral medulla (RVLM), angiotensin II-derived superoxide anions, which increase sympathetic nerve activity, induce a pressor response by activating the p38 mitogen-activated protein kinase (p38 MAPK) and extracellular signal-regulated kinase (ERK) pathway. The small G protein Ras mediates a caspase-3-dependent apoptotic pathway through p38 MAPK, ERK, and c-Jun N-terminal k...

Journal: :American journal of physiology. Cell physiology 2014
Chao Huang Wenwen Cao Rujia Liao Jia Wang Yuzhe Wang Lijuan Tong Xiangfan Chen Weizhong Zhu Wei Zhang

Protein phosphatase 1 (PP1) and Ca2+/calmodulin-dependent protein kinase δ (CaMKIIδ) are upregulated in heart disorders. Alternative splicing factor (ASF), a major splice factor for CaMKIIδ splicing, can be regulated by both protein kinase and phosphatase. Here we determine the role of PP1 isoforms in ASF-mediated splicing of CaMKIIδ in cells. We found that 1) PP1γ, but not α or β isoform, enha...

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