نتایج جستجو برای: vegfr 2

تعداد نتایج: 2526325  

Journal: :Molecular cancer therapeutics 2013
Khalid A Mohamedali Yu Cao Lawrence H Cheung Walter N Hittelman Michael G Rosenblum

The serine protease granzyme B (GrB) induces apoptosis through both caspase-dependent and -independent multiple-cascade mechanisms. VEGF₁₂₁ binds to both VEGF receptor (VEGFR)-1 and VEGFR-2 receptors. We engineered a unique GrB/VEGF₁₂₁ fusion protein and characterized its properties in vitro and in vivo. Endothelial and tumor cell lines showed varying levels of sensitivity to GrB/VEGF₁₂₁ that c...

Journal: :Molecular cancer research : MCR 2005
Vivian Y Shin William K K Wu Kent-Man Chu Helen P S Wong Emily K Y Lam Emily K K Tai Marcel W L Koo Chi-Hin Cho

Blockade of angiogenesis is a promising strategy to suppress tumor growth, invasion, and metastasis. Vascular endothelial growth factor (VEGF), which binds to tyrosine kinase receptors [VEGF receptors (VEGFR) 1 and 2], is the mediator of angiogenesis and mitogen for endothelial cells. Cyclooxygenase-2 (COX-2) plays an important role in the promoting action of nicotine on gastric cancer growth. ...

Journal: :Cancer research 2003
Jean S Beebe Jitesh P Jani Elisabeth Knauth Peter Goodwin Carla Higdon Ann Marie Rossi Erling Emerson Martin Finkelstein Eugenia Floyd Shawn Harriman Jim Atherton Steve Hillerman Cathy Soderstrom Kou Kou Tom Gant Mark C Noe Barb Foster Farzan Rastinejad Matthew A Marx Tracey Schaeffer Pamela M Whalen W Gregory Roberts

Signaling through vascular endothelial growth factor (VEGF) receptors (VEGFRs) is a key pathway initiating endothelial cell proliferation and migration resulting in angiogenesis, a requirement for human tumor growth and metastasis. Abrogation of signaling through VEGFR by a variety of approaches has been demonstrated to inhibit angiogenesis and tumor growth. Small molecule inhibitors of VEGFR t...

2008
Xiaoyan Shi Ling Xi Danhui Weng Gang Chen Xiaohong Song Peng Wu Beibei Wang Juncheng Wei Shixuan Wang Jianfeng Zhou Ding Ma

To investigate the roles of VEGF-C, VEGFR-3 and cyclooxygenase-2 (COX-2) in tumor progression and lymph node metastasis. The expression of VEGF-C, VEGFR-3 and COX-2 were examined in 93 cases of surgical speciments of cervical diseases by immunohistochemical staining. The correlation between expression of these factors and tumor aggressiveness was evaluated. The expression levels of VEGF-C and C...

Journal: :Blood 2012
Maurice S Brozzo Sasa Bjelić Kaisa Kisko Thomas Schleier Veli-Matti Leppänen Kari Alitalo Fritz K Winkler Kurt Ballmer-Hofer

VEGFs activate 3 receptor tyrosine kinases, VEGFR-1, VEGFR-2, and VEGFR-3, promoting angiogenic and lymphangiogenic signaling. The extracellular receptor domain (ECD) consists of 7 Ig-homology domains; domains 2 and 3 (D23) represent the ligand-binding domain, whereas the function of D4-7 is unclear. Ligand binding promotes receptor dimerization and instigates transmembrane signaling and recept...

Journal: :Neurochemistry international 2012
Shuangmei Liu Changshui Xu Guilin Li Han Liu Jinyan Xie Guihua Tu Haiying Peng Shuyi Qiu Shangdong Liang

Neuropathic pain can arise from a lesion affecting the peripheral nervous system. Selective P2X(3) and P2X(2/3) receptors' antagonists effectively reduce neuropathic pain. VEGF inhibitors are effective for pain relief. The present study investigated the effects of Vatalanib (VEGF receptor-2 (VEGFR-2) inhibitor) on the neuropathic pain to address the interaction of VEGFR-2 and P2X(2/3) receptor ...

2007
Surovi Hazarika Ayotunde O. Dokun Yongjun Li Aleksander S. Popel Christopher D. Kontos Brian H. Annex

Deficient angiogenesis after ischemia may contribute to worse outcomes of peripheral arterial disease in patients with diabetes mellitus (DM). Vascular endothelial growth factor (VEGF) and its receptors promote angiogenesis. We hypothesized that in peripheral arterial disease, maladaptive changes in VEGF ligand/receptor expression could account for impaired angiogenesis in DM. Skeletal muscle f...

2004

The vascular system is the first functional organ that starts to develop in the embryo. The extraembryonic mesodermal cells, so-called hemangioblasts, aggregate in the yolk sac to form blood islands, where they differentiate into an external endothelial cell (EC) layer and an inner core of blood cells. The external ECs subsequently constitute the primary capillary plexus. Shortly thereafter, th...

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