نتایج جستجو برای: tp53 protein

تعداد نتایج: 1240012  

2015
Brandon-Luke L. Seagle Kevin H. Eng Monica Dandapani Judy Y. Yeh Kunle Odunsi Shohreh Shahabi

The objective of this study was to determine if ovarian cancer patients with a TP53 mutation grouped by location of the mutation within the p53 protein structure exhibit differential survival outcomes. Data from patients with high grade serous ovarian cancer (HGS OvCa) (N = 316) or breast cancer (BrCa) (N = 981) sequenced by The Cancer Genome Atlas (TCGA) was studied by Kaplan-Meier and Cox pro...

2016
Ming Li Xiao-Lei Zhang Fang Deng Li-Ting Qian Shui-Ping Meng Wu-Lin Shan Bao-Long Wang

Human papilloma virus (HPV) infection has previously been reported to be associated with TP53 and TP16 expression in Japanese and Taiwanese patients with lung cancer, but data for advanced non-small cell lung cancer (NSCLC) patients is limited. The present study examined the association between HPV infection and TP53 and TP16 expression in Chinese patients with advanced NSCLC. HPV DNA was detec...

Journal: :Blood 2012
Zijun Y Xu-Monette Lin Wu Carlo Visco Yu Chuan Tai Alexander Tzankov Wei-min Liu Santiago Montes-Moreno Karen Dybkaer April Chiu Attilio Orazi Youli Zu Govind Bhagat Kristy L Richards Eric D Hsi X Frank Zhao William W L Choi Xiaoying Zhao J Han van Krieken Qin Huang Jooryung Huh Weiyun Ai Maurilio Ponzoni Andrés J M Ferreri Fan Zhou Brad S Kahl Jane N Winter Wei Xu Jianyong Li Ronald S Go Yong Li Miguel A Piris Michael B Møller Roberto N Miranda Lynne V Abruzzo L Jeffrey Medeiros Ken H Young

TP53 mutation is an independent marker of poor prognosis in patients with diffuse large B-cell lymphoma (DLBCL) treated with cyclophosphamide, hydroxydaunorubicin, vincristine, and prednisone (CHOP) therapy. However, its prognostic value in the rituximab immunochemotherapy era remains undefined. In the present study of a large cohort of DLBCL patients treated with rituximab plus CHOP (R-CHOP), ...

2017
Pierre-Samuel Gillardin Géraldine Descamps Sophie Maiga Benoit Tessoulin Hanane Djamai Benedetta Lucani David Chiron Philippe Moreau Steven Le Gouill Martine Amiot Catherine Pellat-Deceunynck Agnès Moreau-Aubry

(1) Background: TP53 deficiency remains a major adverse event in Multiple Myeloma (MM) despite therapeutic progresses. As it is not possible to target TP53 deficiency with pharmacological agents, we explored the possibility of activating another p53 family member, p73, which has not been well studied in myeloma. (2) Methods: Using human myeloma cell lines (HMCLs) with normal or abnormal TP53 st...

2017
Akinobu Ota Haruhisa Nakao Yumi Sawada Sivasundaram Karnan Md Wahiduzzaman Tadahisa Inoue Yuji Kobayashi Takaya Yamamoto Norimitsu Ishii Tomohiko Ohashi Yukiomi Nakade Ken Sato Kiyoaki Itoh Hiroyuki Konishi Yoshitaka Hosokawa Masashi Yoneda

Splice variants of certain genes impact on genetic biodiversity in mammals. The tumor suppressor TP53 gene (encoding p53) plays an important role in the regulation of tumorigenesis in hepatocellular carcinoma (HCC). Δ40p53α is a naturally occurring p53 isoform that lacks the N-terminal transactivation domain, yet little is known about the role of Δ40p53α in the development of HCC. Here, we firs...

Journal: :Genetics and molecular research : GMR 2008
G R Pinto F K N Yoshioka R L L Silva C A Clara M J Santos J R W Almeida R R Burbano J A Rey C Casartelli

The TP53 tumor suppressor gene codifies a protein responsible for preventing cells with genetic damage from growing and dividing by blocking cell growth or apoptosis pathways. A common single nucleotide polymorphism (SNP) in TP53 codon 72 (Arg72Pro) induces a 15-fold decrease of apoptosis-inducing ability and has been associated with susceptibility to human cancers. Recently, another TP53 SNP a...

2013
Yonggeun Cho Juwon Kim Yoonjung Kim Joon Jeong Kyung-A Lee

Li-Fraumeni syndrome (LFS) is a rare, inherited syndrome associated with increased risk of various early-onset tumors. Since the introduction of classic LFS criteria, various criteria have been proposed to include patients with incomplete LFS features, which make up Li-Fraumeni-like syndromes (LFL). Germline missense mutations of TP53 are the primary cause of LFS and LFL. Mutations mostly resid...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2004
Viola M M van Houten C René Leemans J Alain Kummer Janny Dijkstra Dirk J Kuik Michiel W M van den Brekel Gordon B Snow Ruud H Brakenhoff

PURPOSE Approximately 10-30% of surgically treated head and neck cancer patients develop local recurrences while the resection margins are histologically tumor free. These recurrences may arise from cancer cells left behind but not detected by the pathologist, or they may develop from precursor lesions adjacent to the tumor that were not completely resected. We have investigated whether TP53-mu...

2016
Huaying Hu Jingping Liu Xinbin Liao Shuju Zhang Haibo Li Renbin Lu Xianfeng Li Wei Lin Minji Liu Zanxian Xia Guoliang Qing Jia-Da Li

Li Fraumeni syndrome (LFS) is a rare familial cancer predisposition syndrome with autosomal-dominant inheritance, occurring as frequently as one in 5,000-20,000 individuals. However, no LFS case has been reported from mainland China although it constitutes one quarter of population on earth. In this study, we identified, to our best knowledge, the first Li Fraumeni syndrome family in China. Six...

2017
Fiebo J.C. Ten Kate Lucia Suzuki Lambert C.J. Dorssers Winand N.M. Dinjens David T.W. Jones Daan Nieboer Michael Doukas J. Jan B. Van Lanschot Bas P.L. Wijnhoven Leendert H.J. Looijenga Katharina Biermann

Introduction TP53 mutations are considered to be the driving factor in the initiation of esophageal adenocarcinoma (EAC). However, the impact of this gene and its encoded protein as a prognostic marker has not been definitely established yet. Methods In total, 204 chemoradiotherapy (CRT)-naive patients with EAC were included for p53 protein expression evaluation by immunohistochemistry (IHC) ...

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