نتایج جستجو برای: nondystrophic myotonia

تعداد نتایج: 1472  

Journal: :Deutsche Zeitschrift für Nervenheilkunde 1916

Journal: :The Journal of General Physiology 1996
J B Patlak

The linkages between a protein 's primary sequence, three-dimensional structure, and the detailed manifestations of its function are well appreciated. Exploring and understanding these linkages have proven remarkably complex, however. While some functions may be localized exclusively to one protein region, others may be distributed or even diffusely coded in the structure. The kinetics of the t...

Journal: :Journal of neurology, neurosurgery, and psychiatry 1969
R C Hughes W B Matthews

2014
Silvia Corrochano Roope Männikkö Peter I. Joyce Philip McGoldrick Jessica Wettstein Glenda Lassi Dipa L. Raja Rayan Gonzalo Blanco Colin Quinn Andrianos Liavas Arimantas Lionikas Neta Amior James Dick Estelle G. Healy Michelle Stewart Sarah Carter Marie Hutchinson Liz Bentley Pietro Fratta Andrea Cortese Roger Cox Steve D. M. Brown Valter Tucci Henning Wackerhage Anthony A. Amato Linda Greensmith Martin Koltzenburg Michael G. Hanna Abraham Acevedo-Arozena

Mutations in the skeletal muscle channel (SCN4A), encoding the Nav1.4 voltage-gated sodium channel, are causative of a variety of muscle channelopathies, including non-dystrophic myotonias and periodic paralysis. The effects of many of these mutations on channel function have been characterized both in vitro and in vivo. However, little is known about the consequences of SCN4A mutations downstr...

2013
Jasper M. Morrow Emma Matthews Dipa L. Raja Rayan Arne Fischmann Christopher D.J. Sinclair Mary M. Reilly John S. Thornton Michael G. Hanna Tarek A. Yousry

We assessed the presence, frequency and pattern of MRI abnormalities in non-dystrophic myotonia patients. We reviewed T1-weighted and STIR (short-tau-inversion-recovery) 3T MRI sequences of lower limb muscles at thigh and calf level in 21 patients with genetically confirmed non-dystrophic myotonia: 11 with CLCN1 mutations and 10 with SCN4A mutations, and 19 healthy volunteers. The MRI examinati...

Journal: :American journal of human genetics 1996
V Mailänder R Heine F Deymeer F Lehmann-Horn

Mutations within CLCN1, the gene encoding the major skeletal muscle chloride channel, cause either dominant Thomsen disease or recessive Becker-type myotonia, which are sometimes difficult to discriminate, because of reduced penetrance or lower clinical expressivity in females. We screened DNA of six unrelated Becker patients and found four novel CLCN1 mutations (Gln-74-Stop, Tyr-150-Cys, Tyr-2...

2011
Christoph Fahlke

The generation of action potentials in excitable cells requires selective ion channels that open and close upon changes in membrane potential. Initially, cell excitabil-ity was mainly studied in neuronal axons, and in this particular cell compartment, electrical excitability is almost exclusively governed by cation channels. For many years, voltage-dependent anion channels were thought to be of...

Journal: :Journal of medical genetics 1992
D Viljoen P Beighton

Schwartz-Jampel syndrome is a rare autosomal recessive disorder. Joint contractures, generalised myotonia, skeletal anomalies, and facial dysmorphism are common features; malignant hyperthermia is a potentially lethal complication during anaesthesia.

Journal: :British journal of anaesthesia 1985
P Nightingale T E Healy K McGuinness

Atracurium was used to produce neuromuscular blockade in a patient with dystrophia myotonica. Operating conditions were excellent and myotonia was not seen. Sensitivity to atracurium was not seen and spontaneous recovery was not prolonged.

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