نتایج جستجو برای: mir 34a

تعداد نتایج: 45629  

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2011
Massimiliano Agostini Paola Tucci Joern R Steinert Ruby Shalom-Feuerstein Matthieu Rouleau Daniel Aberdam Ian D Forsythe Kenneth W Young Andrea Ventura Carla P Concepcion Yoon-Chi Han Eleonora Candi Richard A Knight Tak W Mak Gerry Melino

The p53 family member TAp73 is a transcription factor that plays a key role in many biological processes, including neuronal development. In particular, we have shown that p73 drives the expression of miR-34a, but not miR-34b and c, in mouse cortical neurons. miR-34a in turn modulates the expression of synaptic targets including synaptotagmin-1 and syntaxin-1A. Here we show that this axis is re...

2015
Tarek Kamal Motawi Olfat Gamil Shaker Shohda Assem El-Maraghy Mahmoud Ahmed Senousy

MicroRNAs are messengers during interferon-virus interplay and are involved in antiviral immunity, however, little is known about interferon-related microRNAs regarding their detection in serum and their potential use as non-invasive diagnostic and prognostic biomarkers in chronic hepatitis C (CHC). To elucidate some of the molecular aspects underlying failure of pegylated interferon-α/ribaviri...

2017
Xiaoti Lin Weiyu Chen Fengqin Wei Binhua P. Zhou Mien-Chie Hung Xiaoming Xie

Rationale: Cancer stem cells (CSCs) have been implicated as the seeds of therapeutic resistance and metastasis, due to their unique abilities of self-renew, wide differentiation potentials and resistance to most conventional therapies. It is a proactive strategy for cancer therapy to eradicate CSCs. Methods: Tumor tissue-derived breast CSCs (BCSC), including XM322 and XM607, were isolated by fl...

Objective(s): microRNAs are small non-coding molecules that regulate gene expression in various biological processes. T-cell acute lymphoblastic leukemia (T-ALL) is a malignancy accompanied with genetic aberrations and accounts for 20% of children’s and adult’s ALL. Notch signaling pathway dysregulation occurs in 60% of T-ALL cases. In the present study, we aimed to de...

2011
Pasqualino de Antonellis Chiara Medaglia Emilio Cusanelli Immacolata Andolfo Lucia Liguori Gennaro De Vita Marianeve Carotenuto Annamaria Bello Fabio Formiggini Aldo Galeone Giuseppe De Rosa Antonella Virgilio Immacolata Scognamiglio Manuela Sciro Giuseppe Basso Johannes H. Schulte Giuseppe Cinalli Achille Iolascon Massimo Zollo

BACKGROUND Through negative regulation of gene expression, microRNAs (miRNAs) can function as oncosuppressors in cancers, and can themselves show altered expression in various tumor types. Here, we have investigated medulloblastoma tumors (MBs), which arise from an early impairment of developmental processes in the cerebellum, where Notch signaling is involved in many of the cell-fate-determini...

Journal: :Journal of cellular physiology 2014
Tindara Franchina Valeria Amodeo Giuseppe Bronte Giuseppina Savio Giuseppina R R Ricciardi Maria Picciotto Antonio Russo Antonio Giordano Vincenzo Adamo

Pemetrexed has been widely used in patients with advanced non-small cell lung cancer (NSCLC). The clinical relevance of polymorphisms of folate pathway genes for pemetrexed metabolism have not been fully elucidated yet. The aim of this study was to evaluate the expression levels of circulating miR-22, miR-24, and miR-34a, possibly involved in folate pathway, in NSCLC patients treated with pemet...

Journal: :Circulation research 2015
Yanfei Yang Hui-Wen Cheng Yiling Qiu David Dupee Madyson Noonan Yi-Dong Lin Sudeshna Fisch Kazumasa Unno Konstantina-Ioanna Sereti Ronglih Liao

RATIONALE In response to injury, the rodent heart is capable of virtually full regeneration via cardiomyocyte proliferation early in life. This regenerative capacity, however, is diminished as early as 1 week postnatal and remains lost in adulthood. The mechanisms that dictate postinjury cardiomyocyte proliferation early in life remain unclear. OBJECTIVE To delineate the role of miR-34a, a re...

Journal: :Carcinogenesis 2009
Elena Gallardo Alfons Navarro Nuria Viñolas Ramon M Marrades Tania Diaz Bernat Gel Angels Quera Eva Bandres Jesus Garcia-Foncillas Jose Ramirez Mariano Monzo

MicroRNAs (miRNAs) have been identified as promising prognostic markers in non-small-cell lung cancer (NSCLC) since they play an important role in oncogenesis. The miR-34 family is composed of three miRNAs (miR-34a, miR-34b and miR-34c) that are part of the p53 network and whose expression is directly induced by p53 in response to DNA damage or oncogenic stress. We have analyzed the impact of m...

Journal: :Molecular cell 2007
Tsung-Cheng Chang Erik A Wentzel Oliver A Kent Kalyani Ramachandran Michael Mullendore Kwang Hyuck Lee Georg Feldmann Munekazu Yamakuchi Marcella Ferlito Charles J Lowenstein Dan E Arking Michael A Beer Anirban Maitra Joshua T Mendell

The p53 tumor suppressor protein is a critical regulator of the cellular response to cancer-initiating insults such as genotoxic stress. In this report, we demonstrate that microRNAs (miRNAs) are important components of the p53 transcriptional network. Global miRNA expression analyses identified a cohort of miRNAs that exhibit p53-dependent upregulation following DNA damage. One such miRNA, miR...

2016
Martin Hart Stefanie Rheinheimer Petra Leidinger Christina Backes Jennifer Menegatti Tobias Fehlmann Friedrich Grässer Andreas Keller Eckart Meese

Circulating miRNAs have been associated with numerous human diseases. The lack of understanding the functional roles of blood-born miRNAs limits, however, largely their value as disease marker. In a systems biology analysis we identified miR-34a as strongly associated with pathogenesis. Genome-wide analysis of miRNAs in blood cell fractions highlighted miR-34a as most significantly up-regulated...

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