نتایج جستجو برای: keratinocyte growth factor

تعداد نتایج: 1464820  

Journal: :Cancer research 1991
A A Dlugosz S H Yuspa

Staurosporine is a potent but nonselective inhibitor of protein kinase C (PKC) and blocks responses to 12-O-tetradecanoylphorbol-13-acetate (TPA) in several cell types in vitro. In cultured primary mouse keratinocytes, however, staurosporine fails to inhibit TPA-mediated keratinocyte maturation and itself elicits responses that are similar to TPA (T. Sako et al., Cancer Res., 48: 4646-4650, 198...

2016
Neeraj Jain Pazhanichamy Kalailingam Kai Wei Tan Hui Bing Tan Ming Keat Sng Jeremy Soon Kiat Chan Nguan Soon Tan Thirumaran Thanabalu

Neural-Wiskott Aldrich Syndrome Protein (N-WASP) is expressed ubiquitously, regulates actin polymerization and is essential during mouse development. We have previously shown that N-WASP is critical for cell-ECM adhesion in fibroblasts. To characterize the role of N-WASP in fibroblast for skin development, we generated a conditional knockout mouse model in which fibroblast N-WASP was ablated us...

Journal: :Molecular pharmacology 2015
Hiroki Sakai Kenji Matsuura Yoshie Tanaka Takeshi Honda Teruo Nishida Makoto Inui

Re-epithelialization begins early during skin wound healing and is regulated by various growth factors and cytokines. Angiotensin II promotes the migration of keratinocytes and thereby contributes to wound healing. We investigated the mechanism by which angiotensin II stimulates human keratinocyte migration. Angiotensin II-induced keratinocyte migration was inhibited by an angiotensin II type 1...

2014
Hiroki Sakai Kenji Matsuura Yoshie Tanaka Takeshi Honda Teruo Nishida Makoto Inui

Re-epithelialization begins early during skin wound healing and is regulated by various growth factors and cytokines. Angiotensin II promotes the migration of keratinocytes and thereby contributes to wound healing. We investigated the mechanism by which angiotensin II stimulates human keratinocyte migration. Angiotensin II–induced keratinocyte migration was inhibited by an angiotensin II type 1...

Journal: :Journal of cell science 2003
Nicole Maas-Szabowski Anja Stärker Norbert E Fusenig

The human keratinocyte cell line HaCaT expresses essentially all epidermal differentiation markers but exhibits deficiencies in tissue organization as surface transplants in nude mice and even more so in organotypic co-cultures with fibroblasts. Whereas tissue differentiation by normal keratinocytes (NEKs) is regulated by stromal interactions, this mechanism is impaired in HaCaT cells. This reg...

Journal: :Journal of cell science 2012
Emilie Faure Françoise Garrouste Fabrice Parat Sylvie Monferran Ludovic Leloup Gilbert Pommier Hervé Kovacic Maxime Lehmann

α6β4 integrin is the main component of hemidesmosomes (HD) that stably anchor the epithelium to the underlying basement membrane. Epithelial cell migration requires HD remodelling, which can be promoted by epidermal growth factor (EGF). We previously showed that extracellular nucleotides inhibit growth factor-induced keratinocyte migration. Here, we investigate the effect of extracellular nucle...

2012
Zhenxiang Wang Ying Wang Farhang Farhangfar Monica Zimmer Yongxin Zhang

Wound healing is primarily controlled by the proliferation and migration of keratinocytes and fibroblasts as well as the complex interactions between these two cell types. To investigate the interactions between keratinocytes and fibroblasts and the effects of direct cell-to-cell contact on the proliferation and migration of keratinocytes, keratinocytes and fibroblasts were stained with differe...

Journal: :Journal of immunology 2005
Sho Tokumaru Koji Sayama Yuji Shirakata Hitoshi Komatsuzawa Kazuhisa Ouhara Yasushi Hanakawa Yoko Yahata Xiuju Dai Mikiko Tohyama Hiroshi Nagai Lujun Yang Shigeki Higashiyama Akihiko Yoshimura Motoyuki Sugai Koji Hashimoto

The closure of skin wounds is essential for resistance against microbial pathogens, and keratinocyte migration is an important step in skin wound healing. Cathelicidin hCAP18/LL-37 is an innate antimicrobial peptide that is expressed in the skin and acts to eliminate microbial pathogens. Because hCAP18/LL-37 is up-regulated at skin wound sites, we hypothesized that LL-37 induces keratinocyte mi...

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