نتایج جستجو برای: il 23r

تعداد نتایج: 139911  

2015
Theresa V. Nguyen Okechukwu Ukairo Salman R. Khetani Michael McVay Chitra Kanchagar Wolfgang Seghezzi Gulesi Ayanoglu Onyi Irrechukwu Raymond Evers

Elevated levels of proinflammatory cytokines associated with infection and inflammation can modulate cytochrome P450 enzymes, leading to potential disease-drug interactions and altered small-molecule drug disposition. We established a human-derived hepatocyte-Kupffer cell (Hep:KC) coculture model to assess the indirect cytokine impact on hepatocytes through stimulation of KC-mediated cytokine r...

2016
Igor Z Barjaktarevic Ronald G Crystal Robert J Kaner

Rationale. Matrix metalloproteinase-9 (MMP-9) expression is upregulated in alveolar macrophages (AM) of HIV1(+) smokers who develop emphysema. Knowing that lung epithelial lining fluid (ELF) of HIV1(+) smokers contains increased levels of inflammatory cytokines compared to HIV1(-) smokers, we hypothesized that upregulation of lung cytokines in HIV1(+) smokers may be functionally related to incr...

2017
Paolo Scollo Afrah Fahad Alkhuriji Mauro Costa Simona Cavani Valerio Pisaturo Martina Di Luca Alessandro Geroldi Fausta Sozzi Paola Anserini Urska Bizjak Ogrinc Sabina Sencar Lucia Pasquini

Background: Interleukin 17 (IL-17) is a pro-inflammatory cytokine mainly secreted by activated TH-17 cells and involved in inflammatory immune responses. Interleukin 23 (IL-23) sustains the development of inflammation and is considered a check point. Aim: the aim of the present study was to investigate the association between serum IL-17A, IL-17F and serum IL23receptor variants in Saudi women w...

2013
Qinghong Wang Jijun Zhou Bei Zhang Zhiqiang Tian Jun Tang Yanhua Zheng Zemin Huang Yi Tian Zhengcai Jia Yan Tang Jennifer C. van Velkinburgh Qing Mao Xiuwu Bian Yifang Ping Bing Ni Yuzhang Wu

IL-23 regulates myriad processes in the innate and adaptive immune systems, and is a critical mediator of the proinflammatory effects exerted by Th17 cells in many diseases. In this study, we investigated whether and how hepatitis B virus (HBV) causes liver damage directly through the IL-23 signaling pathway. In biopsied liver tissues from HBV-infected patients, expression of both IL-23 and IL-...

Journal: :Cell 2015
Teruyuki Sano Wendy Huang Jason A. Hall Yi Yang Alessandra Chen Samuel J. Gavzy June-Yong Lee Joshua W. Ziel Emily R. Miraldi Ana I. Domingos Richard Bonneau Dan R. Littman

RORγt(+) Th17 cells are important for mucosal defenses but also contribute to autoimmune disease. They accumulate in the intestine in response to microbiota and produce IL-17 cytokines. Segmented filamentous bacteria (SFB) are Th17-inducing commensals that potentiate autoimmunity in mice. RORγt(+) T cells were induced in mesenteric lymph nodes early after SFB colonization and distributed across...

Journal: :Journal of immunology 2011
Jeffrey M Wilson Courtney C Kurtz Steven G Black William G Ross Mohammed S Alam Joel Linden Peter B Ernst

Adenosine is an endogenous metabolite produced during hypoxia or inflammation. Previously implicated as an anti-inflammatory mediator in CD4(+) T cell regulation, we report that adenosine acts via dendritic cell (DC) A(2B) adenosine receptor (A(2B)AR) to promote the development of Th17 cells. Mouse naive CD4(+) T cells cocultured with DCs in the presence of adenosine or the stable adenosine mim...

2017
Seyed Ali Ghaffari Maryam Nemati Hossain Hajghani Hossainali Ebrahimi Abdolkarim Sheikhi Abdollah Jafarzadeh

Background: Interleukin (IL)-17/IL-23 axis performs a prominent role in the pathogenesis of several autoimmune disorders. This study aimed to investigate the concentrations of IL-17 in patients with multiple sclerosis (MS) and its relationship with gender, medication, disease forms and single nucleotide polymorphisms (SNP) in IL-23R gene, including rs11209026 and rs1004819. Methods: The blood s...

2014
Elisabeth Glitzner Ana Korosec Patrick M Brunner Barbara Drobits Nicole Amberg Helia B Schonthaler Tamara Kopp Erwin F Wagner Georg Stingl Martin Holcmann Maria Sibilia

Several subtypes of APCs are found in psoriasis patients, but their involvement in disease pathogenesis is poorly understood. Here, we investigated the contribution of Langerhans cells (LCs) and plasmacytoid DCs (pDCs) in psoriasis. In human psoriatic lesions and in a psoriasis mouse model (DKO* mice), LCs are severely reduced, whereas pDCs are increased. Depletion of pDCs in DKO* mice prior to...

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