نتایج جستجو برای: exome

تعداد نتایج: 8594  

2016
Elise Ruark Márton Münz Matthew Clarke Anthony Renwick Emma Ramsay Anna Elliott Sheila Seal Gerton Lunter Nazneen Rahman

We present an easy-to-use, open-source Optimised Exome analysis tool, OpEx (http://icr.ac.uk/opex) that accurately detects small-scale variation, including indels, to clinical standards. We evaluated OpEx performance with an experimentally validated dataset (the ICR142 NGS validation series), a large 1000 exome dataset (the ICR1000 UK exome series), and a clinical proband-parent trio dataset. T...

2014
Stephan Pabinger Andreas Dander Maria Fischer René Snajder Michael Sperk Mirjana Efremova Birgit Krabichler Michael R. Speicher Johannes Zschocke Zlatko Trajanoski

Recent advances in genome sequencing technologies provide unprecedented opportunities to characterize individual genomic landscapes and identify mutations relevant for diagnosis and therapy. Specifically, whole-exome sequencing using next-generation sequencing (NGS) technologies is gaining popularity in the human genetics community due to the moderate costs, manageable data amounts and straight...

2017
Yoshiji Yamada Jun Sakuma Ichiro Takeuchi Yoshiki Yasukochi Kimihiko Kato Mitsutoshi Oguri Tetsuo Fujimaki Hideki Horibe Masaaki Muramatsu Motoji Sawabe Yoshinori Fujiwara Yu Taniguchi Shuichi Obuchi Hisashi Kawai Shoji Shinkai Seijiro Mori Tomio Arai Masashi Tanaka

We performed exome-wide association studies to identify single nucleotide polymorphisms that either influence fasting plasma glucose level or blood hemoglobin A1c content or confer susceptibility to type 2 diabetes mellitus in Japanese. Exome-wide association studies were performed with the use of Illumina Human Exome-12 DNA Analysis or Infinium Exome-24 BeadChip arrays and with 11,729 or 8635 ...

2015
Abdul Elkadri Cornelia Thoeni Sophie J. Deharvengt Ryan Murchie Conghui Guo James D. Stavropoulos Christian R. Marshall Paul Wales Robert H.J. Bandsma Ernest Cutz Chaim M. Roifman David Chitayat Yaron Avitzur Radu V. Stan Aleixo M. Muise

BACKGROUND & AIMS METHODS Severe intestinal diseases observed in very young children are often the result of monogenic defects. We used whole exome sequencing (WES) to examine the genetic cause in a patient with a distinct severe form of protein losing enteropathy (PLE) characterized by hypoproteinemia, hypoalbuminemia, and hypertriglyceridemia. METHODS WES was performed at the Centre for App...

2017
Erica D. Smith Kelly Radtke Mari Rossi Deepali N. Shinde Sourat Darabi Dima El‐Khechen Zöe Powis Katherine Helbig Kendra Waller Dorothy K. Grange Sha Tang Kelly D. Farwell Hagman

Ascertaining a diagnosis through exome sequencing can provide potential benefits to patients, insurance companies, and the healthcare system. Yet, as diagnostic sequencing is increasingly employed, vast amounts of human genetic data are produced that need careful curation. We discuss methods for accurately assessing the clinical validity of gene-disease relationships to interpret new research f...

2014
Elizabeth J. Brown Martin R. Pollak Moumita Barua

The haploid human genome is composed of three billion base pairs, about one percent of which consist of exonic regions, the coding sequence for functional proteins, also now known as the 'exome'. The development of next-generation sequencing makes it possible from a technical and economic standpoint to sequence an individual's exome but at the cost of generating long lists of gene variants that...

2015
Byungho Lim Jihyeob Mun Jeong-Hwan Kim Chan Wook Kim Seon Ae Roh Dong-Hyung Cho Yong Sung Kim Seon-Young Kim Jin Cheon Kim

To characterize the mutation profiles of colorectal cancer (CRC) primary tumors (PTs) and liver metastases (CLMs), we performed both whole-exome and RNA sequencing. Ten significantly mutated genes, including BMI1, CARD11, and NRG1, were found in 34 CRCs with CLMs. We defined three mutation classes (Class 1 to 3) based on the absence or presence of mutations during liver metastasis. Most mutatio...

2016
Eleanor G Seaby Rodney D Gilbert Reuben J Pengelly Gaia Andreoletti Antonia Clarke Sarah Ennis

This report illustrates the difficulties in diagnosing complex cases and demonstrates how whole exome sequencing can resolve complex phenotypes.

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