نتایج جستجو برای: dr1 antigen

تعداد نتایج: 200871  

Journal: :Journal of immunology 2003
Yi Zhang Pascal Chaux Vincent Stroobant Alexander M M Eggermont Jurgen Corthals Bernard Maillère Kris Thielemans Marie Marchand Thierry Boon Pierre Van Der Bruggen

"Cancer-germline" genes such as those of the MAGE family are expressed in many tumors and in male germline cells, but are silent in normal tissues. They encode shared tumor-specific Ags, which have been used in therapeutic vaccination trials of cancer patients. MAGE-3 is expressed in 74% of metastatic melanoma and in 50% of carcinomas of esophagus, head and neck, bladder, and lung. We report he...

Journal: :PLoS ONE 2007
Michaela Lucas Axel Ulsenheimer Katja Pfafferot Malte H.J. Heeg Silvana Gaudieri Norbert Grüner Andri Rauch J. Tilman Gerlach Maria-Christina Jung Reinhart Zachoval Gerd R. Pape Winfried Schraut Teresa Santantonio Hans Nitschko Martin Obermeier Rodney Phillips Thomas J. Scriba Nasser Semmo Cheryl Day Jonathan N. Weber Sarah Fidler Robert Thimme Anita Haberstroh Thomas F. Baumert Paul Klenerman Helmut M. Diepolder

BACKGROUND CD4+ T cell help is critical in maintaining antiviral immune responses and such help has been shown to be sustained in acute resolving hepatitis C. In contrast, in evolving chronic hepatitis C CD4+ T cell helper responses appear to be absent or short-lived, using functional assays. METHODOLOGY/PRINCIPAL FINDINGS Here we used a novel HLA-DR1 tetramer containing a highly targeted CD4...

Journal: :Diabetes 1991
H A Erlich R L Griffith T L Bugawan R Ziegler C Alper G Eisenbarth

Genetic susceptibility to insulin-dependent diabetes mellitus (IDDM) is associated with the HLA-DR3 and DR4 haplotypes. The HLA-DR2 haplotype is negatively associated with IDDM, an association that has been interpreted as dominant protection. Here, we describe the molecular analysis of the HLA class II genes in an unusual family with three HLA-DR1/2 siblings, all of whom have IDDM. With polymer...

Journal: :Journal of the American Academy of Dermatology 2015

Journal: :Journal of leukocyte biology 1999
R Thomas K P MacDonald A R Pettit L L Cavanagh J Padmanabha S Zehntner

Rheumatoid arthritis (RA) is a chronic autoimmune inflammatory disease in which unknown arthrogenic autoantigen is presented to CD4+ T cells. The strong association of the disease with an epitope within the HLA-DR chain shared between various alleles of HLA-DR4 and DR1 emphasizes the importance of antigen presentation. This immune response predominantly occurs in the synovial tissue and fluid o...

2011
Mette Damkjær Bartels Lars Hestbjerg Hansen Kit Boye Søren J. Sørensen Henrik Westh

In methicillin resistant Staphylococcus aureus (MRSA), the arginine catabolic mobile element (ACME) was initially described in USA300 (t008-ST8) where it is located downstream of the staphylococcal cassette chromosome mec (SCCmec). A common health-care associated MRSA in Copenhagen, Denmark (t024-ST8) is clonally related to USA300 and is frequently PCR positive for the ACME specific arcA-gene. ...

Journal: :Journal of immunology 2002
Sigrid Hannier Christina Bitegye Stéphane Demotz

To study the requirements for activation of human Th1 and Th2 cells, soluble peptide/DR1 complexes were prepared from naturally expressed DR1 protein. When immobilized, this material induced T cell activation, as revealed by CD25 up-regulation. Unexpectedly, Th2 cells required a higher density of peptide/DR1 complexes than Th1 cells to initiate CD25 up-regulation. Similar findings were obtained...

2003
U. Shankarkumar D. Mohanty

Two hundred and eighty nine unrelated Marathas residing in Mumbai, Maharastra, (Western India) were studied for HLA A, B, C and DR locus antigen profiles. The HLA antigen maximum likelihood gene frequencies of HLA A1, A2, A9 (24), A11, A19 (33), B5, B7, B35, B40 (61), Cw3, Cw6, DR2, DR5, DR7, DQ1 and DQ2 were increased while that of HLA A3, A10 (26), A36, B8, B13, B16 (38), B18, B21, B22 (55), ...

Journal: :Blood 1997
S I Mannering J L McKenzie D B Fearnley D N Hart

Chronic myeloid leukemia (CML) is characterized by a specific translocation of the c-abl oncogene on chromosome 9 to the break point cluster region (bcr) on chromosome 22, t(9;22) (q34;q11). This translocation results in the expression of a 210-kD bcr-abl protein fusion gene product. The juxtaposition of the bcr and abl genes produces a novel junctional amino acid sequence, which may be present...

Journal: :The Astronomical Journal 2016

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