نتایج جستجو برای: compritol ato 888

تعداد نتایج: 3638  

Journal: :FEBS letters 2012
Guodong Zhang Jing Liu Ye Zhang Jinglei Qu Ling Xu Huachuan Zheng Yunpeng Liu Xiujuan Qu

Various molecular mechanisms are involved in the efficacy of arsenic trioxide (ATO) against malignant hematologic and some solid tumors. FLICE-like inhibitory protein (FLIP) is an inhibitor of apoptosis mediated by death receptors. In this study, we identified a new link between the down-regulation of cellular FLIP(L) and ATO-induced autophagy. ATO induced the degradation of FLIP(L) in K562 and...

2015
Ezhilarasi Chendamarai Saravanan Ganesan Ansu Abu Alex Vandana Kamath Sukesh C. Nair Arun Jose Nellickal Nancy Beryl Janet Vivi Srivastava Kavitha M. Lakshmi Auro Viswabandya Aby Abraham Mohammed Aiyaz Nandita Mullapudi Raja Mugasimangalam Rose Ann Padua Christine Chomienne Mammen Chandy Alok Srivastava Biju George Poonkuzhali Balasubramanian Vikram Mathews

There is limited data on the clinical, cellular and molecular changes in relapsed acute promyeloytic leukemia (RAPL) in comparison with newly diagnosed cases (NAPL). We undertook a prospective study to compare NAPL and RAPL patients treated with arsenic trioxide (ATO) based regimens. 98 NAPL and 28 RAPL were enrolled in this study. RAPL patients had a significantly lower WBC count and higher pl...

Journal: :Acta biochimica et biophysica Sinica 2011
Guangfen Xiao Xueyuan Tang Chenjiao Yao Chenghong Wang

The novel chrysin analog 8-bromo-7-methoxychrysin (BrMC) has been reported to induce apoptosis of various cancer cell lines. Arsenic trioxide (ATO) treatment induces clinical remission in acute promyelocytic leukemia patients. The combination of ATO with other agents has been shown to improve therapeutic effectiveness in vitro and in vivo. In this report, the mechanism of apoptosis induced by t...

2016
Xiaofan Li Xiaoyan Guan Fang Li Yuanzhong Chen Nainong Li

OBJECTIVE To investigate the cytotoxic effects of suberanilohydroxamic acid (vorinostat) in combination with arsenic trioxide (ATO) on the human NB4 cell line in vitro. METHODS The rates of cell proliferation following treatment with vorinostat with or without ATO were measured. Flow cytometry of Annexin-V/propidium iodide double-stained cells was used to measure apoptosis. Acridine Orange an...

Journal: :Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology 2017
Jing-Yi Zhang Gui-Bo Sun Yun Luo Min Wang Wei Wang Yu-Yang Du Ying-Li Yu Xiao-Bo Sun

BACKGROUND/AIMS This study aimed to investigate whether Salvianolic acid A (Sal A) conferred cardiac protection against Arsenic trioxide (ATO)-induced cardiotoxicity in H9c2 cells by inhibiting MAPK pathways activation. METHODS H9c2 cardiac cells were exposed to 10 µM ATO for 24 h to induce cytotoxicity. The cells were pretreated with Sal A for 4 h before exposure to ATO. Cell viability was d...

2014
Omar Patiño-Rodríguez Irma Torres-Roque Maricela Martínez-Delgado Abraham Escobedo-Moratilla José Pérez-Urizar

Recent clinical research has shown that atorvastatin (ATO) in combination with cholesterol absorption inhibitor ezetimibe (EZE) significantly reduces LDL-C level in patients with hypercholesterolemia, showing a superior lipid-lowering efficacy compared to statin alone. With no information currently available on the interaction between the two drugs, a pharmacokinetic study was conducted to inve...

Journal: :Molecular pharmacology 2012
Amelia M Huehls Jill M Wagner Catherine J Huntoon Larry M Karnitz

Floxuridine (5-fluorodeoxyuridine, FdUrd), a U.S. Food and Drug Administration-approved drug and metabolite of 5-fluorouracil, causes DNA damage that is repaired by base excision repair (BER). Thus, poly(ADP-ribose) polymerase (PARP) inhibitors, which disrupt BER, markedly sensitize ovarian cancer cells to FdUrd, suggesting that this combination may have activity in this disease. It remains unc...

ژورنال: :فصلنامه پژوهشی خون 0
سعید حسنی s. hasani دانشکده پیراپزشکی دانشگاه علوم پزشکی تهران فرهاد ذاکر f. zaker دانشیار دانشکده پیراپزشکی دانشگاه علوم پزشکی تهرانسازمان اصلی تایید شده: دانشگاه علوم پزشکی تهران (tehran university of medical sciences) علی ذکری a. zekri دانشکده پزشکی دانشگاه علوم پزشکی تهرانسازمان اصلی تایید شده: دانشگاه علوم پزشکی تهران (tehran university of medical sciences) اعظم زغل a. zaghal دانشکده پزشکی دانشگاه علوم پزشکی تهرانسازمان اصلی تایید شده: دانشگاه علوم پزشکی تهران (tehran university of medical sciences) کامران علی مقدم k. alimoghaddam دانشکده پزشکی دانشگاه علوم پزشکی تهرانسازمان اصلی تایید شده: دانشگاه علوم پزشکی تهران (tehran university of medical sciences) اردشیر قوام زاده a. ghavamzadeh دانشکده پزشکی دانشگاه علوم پزشکی تهرانسازمان اصلی تایید شده: دانشگاه علوم پزشکی تهران (tehran university of medical sciences) سید حمیداله غفاری

چکید ه   سابقه و هدف   داروی آرسنیک تری اکسید( ato ) که در درمان لوسمی پرومیلوسیتی حاد( apl ) استفاده می شود، دارای عوارض جانبی شدیدی می باشد. به منظور افزایش اثرات ضد سرطانی و استفاده از دوزهای پایین تر ato ، اثر ترکیب آن با داروی آزیدوتیمیدین (azt) در القای آپوپتوز روی سلول های nb4 (رده apl ) مورد بررسی قرار گرفت.   مواد و روش ها   در یـک مطالعـه تجربـی، بعـد از کشـت و تیمار سلول ها به مدت 48...

2013
Suk-Gu Yoon Hee-Jeong Cheong Sook-Ja Kim Kyoung Ha Kim Sang-Cheol Lee Namsu Lee Hee Sook Park Jong-Ho Won

PURPOSE Leukemic promyelocytes have the unique ability to undergo differentiation after exposure to retinoic acid and both differentiation and apoptosis after exposure to arsenic trioxide (ATO). Recent studies have shown that inhibition of Src family kinases (SFKs) resulted in enhancement of retinoic acid-induced myeloid differentiation. MATERIALS AND METHODS In this study, we investigated th...

2014
LING-YUN ZHOU FANG-YUAN CHEN LI-JING SHEN HAI-XIA WAN JI-HUA ZHONG

Acute leukemia is a malignant clonal hematopoietic stem cell disease. In the current study, the effects of arsenic trioxide (ATO) on the ecotropic viral integration site-1 (EVI-1) gene were investigated in the THP1 cell line. THP-1 cells were treated with different concentrations of ATO (0, 1, 3 and 5 μM) for 24, 48 or 72 h, then tested for cell viability by CCK-8 kit, cell morphology by cytosp...

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