نتایج جستجو برای: amyloid β

تعداد نتایج: 204360  

Journal: :BMC Genomics 2013

Journal: :Journal of Biological Chemistry 2001

Journal: :Nature Reviews Neuroscience 2011

2015
Jatinder Singh Sangha Owen Wally Arjun H. Banskota Roumiana Stefanova Jeff T. Hafting Alan T. Critchley Balakrishnan Prithiviraj George Perry

We report here the protective effects of a methanol extract from a cultivated strain of the red seaweed, Chondrus crispus, against β-amyloid-induced toxicity, in a transgenic Caenorhabditis elegans, expressing human Aβ1-42 gene. The methanol extract of C. crispus (CCE), delayed β-amyloid-induced paralysis, whereas the water extract (CCW) was not effective. The CCE treatment did not affect the t...

Journal: :AJNR. American journal of neuroradiology 2014
K Kantarci

Development of molecular imaging agents for fibrillar β-amyloid positron-emission tomography during the past decade has brought molecular imaging of Alzheimer disease pathology into the spotlight. Large cohort studies with longitudinal follow-up in cognitively normal individuals and patients with mild cognitive impairment and Alzheimer disease indicate that β-amyloid deposition can be detected ...

Journal: :Nihon Naika Gakkai Zasshi 2011

Journal: :Neurology 2016

Journal: :Current Opinion in Neurology 2009

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2012
Tristan Bolmont Arno Bouwens Christophe Pache Mitko Dimitrov Corinne Berclaz Martin Villiger Bettina M Wegenast-Braun Theo Lasser Patrick C Fraering

We demonstrate label-free imaging of cerebral β-amyloidosis ex vivo and in a living mouse model of Alzheimer's disease using extended-focus Fourier domain optical coherence microscopy (xfOCM). xfOCM provides 3D, high-resolution images of individual β-amyloid plaques in the brain parenchyma and vasculature and requires no staining of the alzheimeric sample under investigation. xfOCM also opens t...

2011
Hien T. Tran Laura Sanchez Thomas J. Esparza David L. Brody

Traumatic brain injury (TBI) is a major environmental risk factor for Alzheimer's disease. Intracellular accumulations of amyloid-β and tau proteins have been observed within hours following severe TBI in humans. Similar abnormalities have been recapitulated in young 3xTg-AD mice subjected to the controlled cortical impact model (CCI) of TBI and sacrificed at 24 h and 7 days post injury. This s...

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