نتایج جستجو برای: acute infantile gaucher disease

تعداد نتایج: 1877238  

Journal: :Clinical genetics 2011
A K Hawkins

1. Galvin JE, Lee VM, Trojanowski JQ. Synucleinopathies: clinical and pathological implications. Arch Neurol 2001: 58 (2): 186–190. 2. Mazzulli JR, Xu YH, Sun Y et al. Gaucher disease glucocerebrosidase and α-synuclein form a bidirectional pathogenic loop in synucleinopathies. Cell 2011: 146 (1): 37–52. 3. Cookson MR. A feedforward loop links Gaucher and Parkinson’s diseases? Cell 2011: 146 (1)...

Journal: :Blood 1993
A Zimran C E Hollak A Abrahamov M H van Oers M Kelly E Beutler

Intravenous enzyme replacement therapy (Alglucerase; Ceredase; Genzyme Corp, Boston, MA) is an effective and safe treatment for patients with type 1 Gaucher disease. In an attempt to reduce its high cost, a "low-dose high-frequency" protocol (30 U/kg/mo, 3 times a week) was introduced and found to be as effective as the original high-dose protocol (60 U/kg every 2 weeks). Because receiving freq...

2014
Marissa Orenstein Deborah Barbouth Olaf A Bodamer Neal J Weinreb

BACKGROUND Gaucher disease, an autosomal recessive condition due to deficiency of lysosomal glucocerebrosidase, is a multisystemic disease, with variable age of onset, severity and progression. It is classified into subtypes delineated by the absence (type 1) or presence (type 2 and 3) of primary nervous system involvement. The ethnically diverse, largely immigrant population in South Florida h...

Journal: :Journal of lipid research 2005
Ying Sun Brian Quinn David P Witte Gregory A Grabowski

Gaucher disease is a common lysosomal storage disease caused by a defect of acid beta-glucosidase (GCase). The optimal in vitro hydrolase activity of GCase requires saposin C, an activator protein that derives from a precursor, prosaposin. To develop additional models of Gaucher disease and to test in vivo effects of saposin deficiencies, mice expressing low levels (4--45% of wild type) of pros...

2015
Stefano Raffaele Giannubilo Angela Pasculli Elisa Tidu Andrea Ciavattini

BACKGROUND Gaucher disease is a lysosomal storage disorder due to deficiency of glucocerebrosidase enzyme. In this study, a case of enzyme-treated woman during her pregnancy was reported. CASE PRESENTATION A 27-year old woman with type I Gaucher disease was managed for pregnancy until delivery. She underwent elective splenectomy at age 26 years and was treated with 19-38 units/kg of imiglucer...

2014
Samira Shizuko Parreão Oi Dario Itapary Nicolau Sebastião Kelson Alves dos Santos Marcos Antonio Custódio Neto da Silva Graça Maria de Castro Viana Maria do Desterro Soares Brandão Nascimento

BACKGROUND Gaucher disease is an inborn, autosomal recessive error of the metabolism which belongs to the group of lysosomal storage disorders. OBJECTIVE This work reports on the treatment of Gaucher disease in several members of the same family from the countryside of Maranhão. METHODS This was an observational, retrospective and prospective, descriptive case study about the efficacy of en...

2015
Jun Mitsui Takashi Matsukawa Hidenao Sasaki Ichiro Yabe Masaaki Matsushima Alexandra Dürr Alexis Brice Hiroshi Takashima Akio Kikuchi Masashi Aoki Hiroyuki Ishiura Tsutomu Yasuda Hidetoshi Date Budrul Ahsan Atsushi Iwata Jun Goto Yaeko Ichikawa Yasuo Nakahara Yoshio Momose Yuji Takahashi Kenju Hara Akiyoshi Kakita Mitsunori Yamada Hitoshi Takahashi Osamu Onodera Masatoyo Nishizawa Hirohisa Watanabe Mizuki Ito Gen Sobue Kinya Ishikawa Hidehiro Mizusawa Kazuaki Kanai Takamichi Hattori Satoshi Kuwabara Kimihito Arai Shigeru Koyano Yoshiyuki Kuroiwa Kazuko Hasegawa Tatsuhiko Yuasa Kenichi Yasui Kenji Nakashima Hijiri Ito Yuishin Izumi Ryuji Kaji Takeo Kato Susumu Kusunoki Yasushi Osaki Masahiro Horiuchi Tomoyoshi Kondo Shigeo Murayama Nobutaka Hattori Mitsutoshi Yamamoto Miho Murata Wataru Satake Tatsushi Toda Alessandro Filla Thomas Klockgether Ullrich Wüllner Garth Nicholson Sid Gilman Caroline M Tanner Walter A Kukull Mathew B Stern Virginia M-Y Lee John Q Trojanowski Eliezer Masliah Phillip A Low Paola Sandroni Laurie J Ozelius Tatiana Foroud Shoji Tsuji

OBJECTIVE Glucocerebrosidase gene (GBA) variants that cause Gaucher disease are associated with Parkinson disease (PD) and dementia with Lewy bodies (DLB). To investigate the role of GBA variants in multiple system atrophy (MSA), we analyzed GBA variants in a large case-control series. METHODS We sequenced coding regions and flanking splice sites of GBA in 969 MSA patients (574 Japanese, 223 ...

Journal: :The Journal of clinical investigation 1992
J A Nolta X J Yu I Bahner D B Kohn

Gaucher disease, a lysosomal glycolipid storage disorder, results from the genetic deficiency of an acidic glucosidase, glucocerebrosidase (GC). The beneficial effects of allogeneic bone marrow transplantation (BMT) for Gaucher disease suggest that GC gene transduction and the transplantation of autologous hematopoietic stem cells (gene therapy) may similarly alleviate symptoms. We have constru...

Journal: :QJM : monthly journal of the Association of Physicians 2003
B Dayan D Elstein A Zimran G Nesher

BACKGROUND Gaucher disease, the most common sphingolipid storage disease, results in accumulation of glucocerebroside in macrophages or "Gaucher cells". In a preliminary screening of 109 patients with type I disease, when asked specifically about dry mouth, approximately one quarter claimed to suffer from this symptom. AIM To ascertain whether decreased salivary output is a feature of Gaucher...

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