نتایج جستجو برای: دستگاه خودپرداز atm

تعداد نتایج: 44586  

Journal: :Breast Cancer Research 1999

Journal: :Atlas of Genetics and Cytogenetics in Oncology and Haematology 2015

Journal: :genetics in the 3rd millennium 0
fatemeh ahangari zohreh fattahi mahsa fadaee raheleh vazehan ayda abolhassani elham parsimehr

hereditary ataxias are heterogeneous group of neurodegenerative disorders classified mainly into more than 40 autosomal dominant cerebellar ataxias and 50 recessive ataxias. large amount of nearly uncommon subtypes with extensive phenotypic overlap and relatively high rate of abnormal repetitive sequence expansions, such as trinucleotide repeat expansions make diagnostic genetic testing complic...

2007
Mahesh Subramanyan

This report discusses the performance and sources of protocol overhead in ATM networks. It rst introduces ATM followed by a description of the inhouse ATM network. Our primary goal was to study the performance tradeoo of chosing TCP/IP versus ATM API in a local ATM network.

2015
Wei-Ta Chen Nancy D Ebelt Travis H Stracker Blerta Xhemalce Carla L Van Den Berg Kyle M Miller Joaquin M Espinosa

Ataxia-telangiectasia mutated (ATM) protein kinase regulates the DNA damage response (DDR) and is associated with cancer suppression. Here we report a cancer-promoting role for ATM. ATM depletion in metastatic cancer cells reduced cell migration and invasion. Transcription analyses identified a gene network, including the chemokine IL-8, regulated by ATM. IL-8 expression required ATM and was re...

Journal: :Cancer research 1998
P Starostik T Manshouri S O'Brien E Freireich H Kantarjian M Haidar S Lerner M Keating M Albitar

The gene mutated in ataxia telangiectasia, ATM, on human chromosome 11q22-q23 is implicated in cell cycle control and DNA repair. Ataxia telangiectasia patients as well as ATM-deficient mice are immune deficient and develop lymphoproliferative disease. Abnormalities in 11q22.3-q23.1 have also been described in B-cell chronic lymphocytic leukemia (B-CLL). We analyzed B-CLL samples for loss of he...

2014
Satoko Iwahori Daisuke Kohmon Junya Kobayashi Yuhei Tani Takashi Yugawa Kenshi Komatsu Tohru Kiyono Nozomi Sugimoto Masatoshi Fujita

Ataxia-telangiectasia mutated (ATM) plays crucial roles in DNA damage responses, especially with regard to DNA double-strand breaks (DSBs). However, it appears that ATM can be activated not only by DSB, but also by some changes in chromatin architecture, suggesting potential ATM function in cell cycle control. Here, we found that ATM is involved in timely degradation of Cdt1, a critical replica...

Journal: :Annals of oncology : official journal of the European Society for Medical Oncology 2014
R C Bueno R A Canevari R A R Villacis M A C Domingues J R F Caldeira R M Rocha S A Drigo S R Rogatto

BACKGROUND Ataxia telangiectasia-mutated (ATM) gene downexpression has been reported in sporadic breast carcinomas (BC); however, the prognostic value and mechanisms of ATM deregulation remain unclear. PATIENTS AND METHODS ATM and miRNAs (miR-26a, miR-26b, miR-203, miR-421, miR-664, miR-576-5p and miR-18a) expression levels were evaluated by quantitative real-time PCR (RT-qPCR) in 52 BC and 3...

2000
Sihui Zhou Aruna Seneviratne Terry Percival

Location management is one of the most important aspects in the realization of mobility in wireless networks. Following developments of ATM (Asynchronous Transfer Mode) technology, more core networks become ATM-based. Therefore, the demand of mobility on wireless networks with ATM backbones is increasing. In recent years, several location management schemes have been proposed for classical wire...

Journal: :Molecular and cellular biology 1998
Y Xu E M Yang J Brugarolas T Jacks D Baltimore

Disruption of the mouse Atm gene, whose human counterpart is consistently mutated in ataxia-telangiectasia (A-T) patients, creates an A-T mouse model exhibiting most of the A-T-related systematic and cellular defects. While ATM plays a major role in signaling the p53 response to DNA strand break damage, Atm-/- p53(-/-) mice develop lymphomas earlier than Atm-/- or p53(-/-) mice, indicating that...

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