نتایج جستجو برای: wiskott

تعداد نتایج: 1772  

Journal: :Biochemistry 1999
H N Higgs L Blanchoin T D Pollard

The 70 C-terminal amino acids of Wiskott-Aldrich syndrome protein (WASp WA) activate the actin nucleation activity of the Arp2/3 complex. WASp WA binds both the Arp2/3 complex and actin monomers, but the mechanism by which it activates the Arp2/3 complex is not known. We characterized the effect of WASp WA on actin polymerization in the absence and presence of the human Arp2/3 complex. WASp WA ...

Journal: :Science 2002
Amy Hin Yan Tong Becky Drees Giuliano Nardelli Gary D Bader Barbara Brannetti Luisa Castagnoli Marie Evangelista Silvia Ferracuti Bryce Nelson Serena Paoluzi Michele Quondam Adriana Zucconi Christopher W V Hogue Stanley Fields Charles Boone Gianni Cesareni

Peptide recognition modules mediate many protein-protein interactions critical for the assembly of macromolecular complexes. Complete genome sequences have revealed thousands of these domains, requiring improved methods for identifying their physiologically relevant binding partners. We have developed a strategy combining computational prediction of interactions from phage-display ligand consen...

Journal: :Blood 2001
E Haddad J L Zugaza F Louache N Debili C Crouin K Schwarz A Fischer W Vainchenker J Bertoglio

In studies aimed at further characterizing the cellular immunodeficiency of the Wiskott-Aldrich syndrome (WAS), we found that T lymphocytes from WAS patients display abnormal chemotaxis in response to the T-cell chemoattractant stromal cell-derived factor (SDF)-1. The Wiskott- Aldrich syndrome protein (WASP), together with the Rho family GTPase Cdc42, control stimulus-induced actin cytoskeleton...

Journal: :Blood 2002
Hervé Falet Karin M Hoffmeister Ralph Neujahr John H Hartwig

Arp2/3 complex is believed to induce de novo nucleation of actin filaments at the edge of motile cells downstream of WASp family proteins. In this study, the signaling pathways leading to Arp2/3 complex activation, actin assembly, and shape change were investigated in platelets isolated from patients with Wiskott-Aldrich Syndrome (WAS), that is, who lack WASp, and in WASp-deficient mouse platel...

Journal: :Blood 2004
Sonja Majstoravich Jinyi Zhang Susan Nicholson-Dykstra Stefan Linder Wilhelm Friedrich Katherine A Siminovitch Henry N Higgs

Short microvilli cover the surfaces of circulating mammalian lymphocytes. The surfaces of monocytes and neutrophils are very different, containing ruffles as their predominant structure. In this study, we present the first quantitative characterization of lymphocyte microvilli. From analysis of scanning electron micrographs, we find that median microvillar length and surface density range from ...

2016
Hiroaki Kurushima Jin Yoshimura Jeong-Kyu Kim Jong-Kuk Kim Yutaka Nishimoto Katsuhiko Sayama Manabu Kato Kenta Watanabe Eisuke Hasegawa Derek A Roff Akira Shimizu

Many cryptic species have been discovered in various taxonomic groups based on molecular phylogenetic analyses and mating experiments. Some sympatric cryptic species share equivalent resources, which contradicts the competitive exclusion principle. Two major theories have been proposed to explain the apparent lack of competitive exclusion, i.e. niche-based coexistence and neutral model, but a c...

2003
Judith Goodship Joan Carter Teresa Espanol Yvonne Boyd

Wiskott-Aldrich syndrome (WAS) is an X-linked immunodeficiency disorder with no clinical or immunologic abnormalities in carrier famales. The defective gene has been localized to proximal Xp. Carrier females have nonrandom use of the X chromosome in granulocytes, lymphocytes, and monocytes. We have used the probe M27p. which detects both a variable number tandem repeat polymorphism and methylat...

2016
Rajesh B Patil Chandrakala Shanmukhaiah Farah Jijina Shailesh Bamborde Nilesh Wasekar Manoj Toshniwal Aniket Mohite Vinod Patil

Objective. The aim of this paper is to report the case of Wiskott-Aldrich syndrome (WAS) that presented with unusual laboratory features. Clinical Presentation and Intervention. Male neonate admitted with symptoms related to thrombocytopenia, whose initial diagnosis was considered as neonatal alloimmune thrombocytopenia and JMML (juvenile myelomonocytic leukemia) but subsequently diagnosis was ...

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