نتایج جستجو برای: tram flap
تعداد نتایج: 20039 فیلتر نتایج به سال:
Breast fat necrosis is a benign inflammatory process. Although it is often considered idiopathic, breast fat necrosis is usually caused by trauma, and many idiopathic cases are probably due to microtrauma. Trauma leading to breast fat necrosis can result from accidents or clinical treatment. Among accidental agents, injury from safety belts in traffic accidents is common. Iatrogenic agents incl...
TRAM-34, a clotrimazole analog characterized as a potent and selective inhibitor of intermediate-conductance, calcium-activated K(+) (IKCa) channels, has been used extensively in vitro and in vivo to study the biological roles of these channels. The major advantage of TRAM-34 over clotrimazole is the reported lack of inhibition of the former drug on cytochrome P450 (CYP) activity. CYPs, a large...
Breast reconstruction is now a part of the overall treatment of breast cancer. Its main drawback is rather the suspicion to conceal any probable recurrence. Fat necrosis is a particularly important finding because it can be mistaken for a local recurrence. Alternatively, local recurrences may be dismissed as areas of fat necrosis. Fat necrosis is a relatively minor complication of TRAM flap bre...
This work furthers the work in Transaction Agent Modelling (TrAM) by merging its conceptual catalogue based on the REA (Resources-Events-Agents) accounting model with Sowa’s 1984 conceptual catalogue. The merged catalogue features in a preliminary implementation of TrAM using the Amine software tool, which also offers the model-checking support that is core to TrAM. This automated process demon...
TLR signal via Toll-IL-1R (TIR) homology domain-containing adaptor proteins. One of these adaptors, Toll-IL-1R domain-containing adaptor inducing IFN-beta-related adaptor molecule (TRAM), has been shown to be essential for TLR4 signaling in TRAM(-/-) mice and cell lines. Previously, we showed that MyD88 or Mal dominant-negative constructs did not inhibit LPS induction of cytokines in primary hu...
MyD88, a Toll/interleukin-1 receptor homology (TIR) domain-containing adaptor protein, mediates signals from the Toll-like receptors (TLR) or IL-1/IL-18 receptors to downstream kinases. In MyD88-dependent TLR4 signaling, the function of MyD88 is enhanced by another TIR domain-containing adaptor, Mal/TIRAP, which brings MyD88 to the plasma membrane and promotes its interaction with the cytosolic...
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