نتایج جستجو برای: tram flap

تعداد نتایج: 20039  

2012
Sergi Ganau Lidia Tortajada Fernanda Escribano F. Javier Andreu

Breast fat necrosis is a benign inflammatory process. Although it is often considered idiopathic, breast fat necrosis is usually caused by trauma, and many idiopathic cases are probably due to microtrauma. Trauma leading to breast fat necrosis can result from accidents or clinical treatment. Among accidental agents, injury from safety belts in traffic accidents is common. Iatrogenic agents incl...

2013
Jay J. Agarwal Yi Zhu Qing-Yu Zhang Alexander A. Mongin Lindsay B. Hough

TRAM-34, a clotrimazole analog characterized as a potent and selective inhibitor of intermediate-conductance, calcium-activated K(+) (IKCa) channels, has been used extensively in vitro and in vivo to study the biological roles of these channels. The major advantage of TRAM-34 over clotrimazole is the reported lack of inhibition of the former drug on cytochrome P450 (CYP) activity. CYPs, a large...

2004
Fuat Yüksel Emir Silit Bahattin Çeliköz

Breast reconstruction is now a part of the overall treatment of breast cancer. Its main drawback is rather the suspicion to conceal any probable recurrence. Fat necrosis is a particularly important finding because it can be mistaken for a local recurrence. Alternatively, local recurrences may be dismissed as areas of fat necrosis. Fat necrosis is a relatively minor complication of TRAM flap bre...

2008
Richard Hill Simon Polovina

This work furthers the work in Transaction Agent Modelling (TrAM) by merging its conceptual catalogue based on the REA (Resources-Events-Agents) accounting model with Sowa’s 1984 conceptual catalogue. The merged catalogue features in a preliminary implementation of TrAM using the Amine software tool, which also offers the model-checking support that is core to TrAM. This automated process demon...

Journal: :Journal of immunology 2007
Sandra M Sacre Anna M C Lundberg Evangelos Andreakos Corinne Taylor Marc Feldmann Brian M Foxwell

TLR signal via Toll-IL-1R (TIR) homology domain-containing adaptor proteins. One of these adaptors, Toll-IL-1R domain-containing adaptor inducing IFN-beta-related adaptor molecule (TRAM), has been shown to be essential for TLR4 signaling in TRAM(-/-) mice and cell lines. Previously, we showed that MyD88 or Mal dominant-negative constructs did not inhibit LPS induction of cytokines in primary hu...

2012
Hidenori Ohnishi Hidehito Tochio Zenichiro Kato Norio Kawamoto Takeshi Kimura Kazuo Kubota Takahiro Yamamoto Tatsuyoshi Funasaka Hiroshi Nakano Richard W. Wong Masahiro Shirakawa Naomi Kondo

MyD88, a Toll/interleukin-1 receptor homology (TIR) domain-containing adaptor protein, mediates signals from the Toll-like receptors (TLR) or IL-1/IL-18 receptors to downstream kinases. In MyD88-dependent TLR4 signaling, the function of MyD88 is enhanced by another TIR domain-containing adaptor, Mal/TIRAP, which brings MyD88 to the plasma membrane and promotes its interaction with the cytosolic...

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