نتایج جستجو برای: spastic paraplegia

تعداد نتایج: 11676  

Journal: :The Lancet. Neurology 2008
Sara Salinas Christos Proukakis Andrew Crosby Thomas T Warner

Hereditary spastic paraplegia (HSP) describes a heterogeneous group of genetic neurodegenerative disorders in which the most severely affected neurons are those of the spinal cord. These disorders are characterised clinically by progressive spasticity and weakness of the lower limbs, and pathologically by retrograde axonal degeneration of the corticospinal tracts and posterior columns. In recen...

Journal: :Journal of neurology, neurosurgery, and psychiatry 1977
J G McLeod J A Morgan C Reye

Motor and sensory conduction studies have been performed in 10 patients from three families with uncomplicated familial spastic paraplegia whose ages ranged from 4 to 41 years. In all cases the values fell within the control range. The findings may be contrasted with those in Friedreich's ataxia and some other spinocerebellar degenerations in which peripheral nerve abnormalities are present.

2014
Min-Yu Lan Yung-Yee Chang Tu-Hseuh Yeh Szu-Chia Lai Chia-Wei Liou Hung-Chou Kuo Yih-Ru Wu Rong-Kuo Lyu Jen-Wen Hung Ying-Chao Chang Chin-Song Lu

BACKGROUND Hereditary spastic paraplegias (HSPs) are a group of neurodegenerative diseases characterized by progressive spasticity and weakness of the lower limbs. SPG4, SPG3A and SPG31 are the three leading causes of autosomal dominant (AD) HSPs. METHODS A total of 20 unrelated AD-HSP families were recruited for clinical and genetic assessment. Detection of mutations in SPG4, SPG3A and SPG31...

Journal: :Journal of medical genetics 1987
J S Fitzsimmons P R Guilbert

Male uniovular twins presented at the age of 20 years with spastic paraplegia which had been slowly progressing over the years. Both have skeletal anomalies of their hands and feet with brachydactyly, cone shaped epiphyses, and an abnormal metaphyseal phalangeal pattern profile. In addition, they have a non-specific dysarthria and low-normal intellectual ability.

2017
Mustafa Y. Ahmed Aisha Al-Khayat Fathiya Al-Murshedi Amna Al-Futaisi Barry A. Chioza J. Pedro Fernandez-Murray Jay E. Self Claire G. Salter Gaurav V. Harlalka Lettie E. Rawlins Sana Al-Zuhaibi Faisal Al-Azri Fatma Al-Rashdi Amaury Cazenave-Gassiot Markus R. Wenk Fatema Al-Salmi Michael A. Patton David L. Silver Emma L. Baple Christopher R. McMaster Andrew H. Crosby

Mutations in genes involved in lipid metabolism have increasingly been associated with various subtypes of hereditary spastic paraplegia, a highly heterogeneous group of neurodegenerative motor neuron disorders characterized by spastic paraparesis. Here, we report an unusual autosomal recessive neurodegenerative condition, best classified as a complicated form of hereditary spastic paraplegia, ...

2016
Himanshu K. Mishra Iryna Prots Steven Havlicek Zacharias Kohl Francesc Perez‐Branguli Tom Boerstler Lukas Anneser Georgia Minakaki Holger Wend Martin Hampl Marina Leone Martina Brückner Jochen Klucken Andre Reis Leah Boyer Gerhard Schuierer Jürgen Behrens Angelika Lampert Felix B. Engel Fred H. Gage Jürgen Winkler Beate Winner

OBJECTIVE Mutations in the Spastic Paraplegia Gene11 (SPG11), encoding spatacsin, cause the most frequent form of autosomal recessive (AR) complex hereditary spastic paraplegia (HSP) and juvenile onset amyotrophic lateral sclerosis (ALS5). When SPG11 is mutated, patients frequently present with spastic paraparesis, a thin corpus callosum, and cognitive impairment. We previously delineated a neu...

Journal: :Journal of neurology, neurosurgery, and psychiatry 1974
W M Behan M Maia

Uncomplicated Strümpell's disease (Strümpell's familial spastic paraplegia) with a dominant mode of inheritance is recorded in six families. The neuropathological findings in two cases from these families are given, bringing the total of similar histologically documented reports in the literature to 11. It is concluded that, although exact classification and identification of the many different...

Journal: :Human molecular genetics 2014
Beate Winner Maria C Marchetto Jürgen Winkler Fred H Gage

While motor neuron diseases are currently incurable, induced pluripotent stem cell research has uncovered some disease-relevant phenotypes. We will discuss strategies to model different aspects of motor neuron disease and the specific neurons involved in the disease. We will then describe recent progress to investigate common forms of motor neuron disease: amyotrophic lateral sclerosis, heredit...

2017
Pingting Liu Baichun Jiang Jian Ma Pengfei Lin Yinshuai Zhang Changshun Shao Wenjie Sun Yaoqin Gong

The S113R mutation (c.339T>G) (MIM #603690.0001) in SLC33A1 (MIM #603690), an ER membrane acetyl-CoA transporter, has been previously identified in individuals with hereditary spastic paraplegia type 42 (SPG42; MIM #612539). SLC33A1 has also been shown to inhibit the bone morphogenetic protein (BMP) signaling pathway in zebrafish. To better understand the function of SLC33A1, we generated and c...

2014
Francesc Pérez-Brangulí Himanshu K. Mishra Iryna Prots Steven Havlicek Zacharias Kohl Domenica Saul Christine Rummel Jonatan Dorca-Arevalo Martin Regensburger Daniela Graef Elisabeth Sock Juan Blasi Teja W. Groemer Ursula Schlötzer-Schrehardt Jürgen Winkler Beate Winner

Hereditary spastic paraplegias are a group of inherited motor neuron diseases characterized by progressive paraparesis and spasticity. Mutations in the spastic paraplegia gene SPG11, encoding spatacsin, cause an autosomal-recessive disease trait; however, the precise knowledge about the role of spatacsin in neurons is very limited. We for the first time analyzed the expression and function of s...

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