نتایج جستجو برای: polyposis

تعداد نتایج: 11192  

Journal: :The Journal of pathology 2015
Robbert D A Weren Ramprasath Venkatachalam Jean-Baptiste Cazier Henner F Farin C Marleen Kets Richarda M de Voer Lilian Vreede Eugène T P Verwiel Monique van Asseldonk Eveline J Kamping Lambertus A Kiemeney Kornelia Neveling Katja K H Aben Luis Carvajal-Carmona Iris D Nagtegaal Hans K Schackert Hans Clevers Marc van de Wetering Ian P Tomlinson Marjolijn J L Ligtenberg Nicoline Hoogerbrugge Ad Geurts van Kessel Roland P Kuiper

Heritable genetic variants can significantly affect the lifetime risk of developing cancer, including polyposis and colorectal cancer (CRC). Variants in genes currently known to be associated with a high risk for polyposis or CRC, however, explain only a limited number of hereditary cases. The identification of additional genetic causes is, therefore, crucial to improve CRC prevention, detectio...

Journal: :Gut 2002
A-L Moisio H Järvinen P Peltomäki

BACKGROUND Familial adenomatous polyposis (FAP) is a rare autosomal dominantly inherited disease predisposing to colon cancer and caused by germline mutations in the APC (adenomatous polyposis coli) gene. AIMS We conducted a population based study to evaluate the prevalence and clinical implications of APC mutations among Finnish FAP kindreds. A possible founder effect in parallel with previo...

Journal: :Monaldi archives for chest disease = Archivio Monaldi per le malattie del torace 2009
G E Carpagnano P Carratú M Gelardi A Spanevello G Di Gioia T Condreva O Resta M P Foschino Barbaro

BACKGROUND AND AIM Nasal polyposis (NP) occurs in about 1-4% of the worldwide population. Increased plasma concentrations of different pro-inflammatory cytokines have been observed in NP, and might be related to the pathogenesis of this syndrome. The present study was designed to investigate IL-6 and IL-4 concentrations in nasal and oral exhaled breath condensate of patients with early and adva...

Journal: :The Journal of clinical investigation 1981
M Lipkin B S Reddy J Weisburger L Schechter

In previous studies subjects with familial polyposis, the autosomal dominant disease leading to colon cancer, excreted higher levels of fecal cholesterol than normal subjects, with decreased conversion to degradation products. Findings suggested fecal cholesterol degradation as a marker of hereditary predisposition to colon cancer. Current measurements now have shown that affected individuals a...

Journal: :Cancer immunology research 2015
Kristen L Dennis Abdulrahman Saadalla Nichole R Blatner Shuya Wang Vysak Venkateswaran Fotini Gounari Hilde Cheroutre Casey T Weaver Axel Roers Nejat K Egilmez Khashayarsha Khazaie

IL10 is attributed with immune-suppressive and anti-inflammatory properties, which could promote or suppress cancer in the gastrointestinal tract. Loss of IL10 exacerbates colonic inflammation, leading to colitis and cancer. Consistent with this, transfer of IL10-competent regulatory T cells (Treg) into mice with colitis or hereditary polyposis protects against disease, while IL10-deficient mic...

Journal: :Proceedings of the Royal Society of Medicine 1958

Journal: :JAMA: The Journal of the American Medical Association 1913

Journal: :Journal of Medical Genetics 1966

Journal: :Video Journal and Encyclopedia of GI Endoscopy 2013

Journal: :Clinical Gastroenterology and Hepatology 2020

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