نتایج جستجو برای: p15ink4b gene

تعداد نتایج: 1141459  

Journal: :Cancer research 1999
M Cool P Jolicoeur

Loss of heterozygosity (LOH) analysis was performed on 62 mammary tumors that were induced in (BALB/c x C57BL/6)F1 mouse mammary tumor virus/neu transgenic mice. Eighty-six simple sequence length polymorphism markers were used to cover all of the somatic chromosomes. Frequency of LOH was observed to be significant for chromosomes 4 (50%), 19 (32%), and 8 (21%). On chromosome 4, at least three d...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2007
Chi-Hwa Wu Jan van Riggelen Alper Yetil Alice C Fan Pavan Bachireddy Dean W Felsher

Oncogene-induced senescence is an important mechanism by which normal cells are restrained from malignant transformation. Here we report that the suppression of the c-Myc (MYC) oncogene induces cellular senescence in diverse tumor types including lymphoma, osteosarcoma, and hepatocellular carcinoma. MYC inactivation was associated with prototypical markers of senescence, including acidic beta-g...

Journal: :The Journal of Cell Biology 2004
Masakiyo Sakaguchi Masahiro Miyazaki Hiroyuki Sonegawa Mariko Kashiwagi Motoi Ohba Toshio Kuroki Masayoshi Namba Nam-ho Huh

Growth regulation of epithelial cells is of major concern because most human cancers arise from them. We demonstrated previously a novel signal pathway involving S100C/A11 for high Ca2+-induced growth inhibition of normal human keratinocytes (Sakaguchi, M., M. Miyazaki, M. Takaishi, Y. Sakaguchi, E. Makino, N. Kataoka, H. Yamada, M. Namba, and N.H. Huh. 2003. J. Cell Biol. 163:825-835). This pa...

Journal: :Genes & development 2017
Marieke Aarts Athena Georgilis Meryam Beniazza Patrizia Beolchi Ana Banito Thomas Carroll Marizela Kulisic Daniel F Kaemena Gopuraja Dharmalingam Nadine Martin Wolf Reik Johannes Zuber Keisuke Kaji Tamir Chandra Jesús Gil

Expression of the transcription factors OCT4, SOX2, KLF4, and cMYC (OSKM) reprograms somatic cells into induced pluripotent stem cells (iPSCs). Reprogramming is a slow and inefficient process, suggesting the presence of safeguarding mechanisms that counteract cell fate conversion. One such mechanism is senescence. To identify modulators of reprogramming-induced senescence, we performed a genome...

2016
Jie Ting Zhang Zhi Hui Weng Kam Sze Tsang Lai Ling Tsang Hsiao Chang Chan Xiao Hua Jiang Gilbert Bernier

The biologic studies of human neural crest stem cells (hNCSCs) are extremely challenging due to the limited source of hNCSCs as well as ethical and technical issues surrounding isolation of early human embryonic tissues. On the other hand, vast majority of studies on MycN have been conducted in human tumor cells, thus, the role of MycN in normal human neural crest development is completely unkn...

Journal: :Therapeutics and Clinical Risk Management 2007
Hussain I Saba

Myelodysplastic syndromes (MDS) are a group of heterogeneous clonal hematopoietic stem cell disorders characterized by ineffective hematopoiesis, peripheral blood cytopenias and a propensity to transform into acute myeloid leukemia. There are few treatment options available for patients with MDS. Studies into the molecular biology of MDS have demonstrated abnormal patterns of DNA methylation th...

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