نتایج جستجو برای: osteoclastogenesis

تعداد نتایج: 2849  

Journal: :Arteriosclerosis, thrombosis, and vascular biology 2014
Masahiko Kurabayashi

Journal: :Bone 2015
Yusuke Kameda Masahiko Takahata Shintaro Mikuni Tomohiro Shimizu Hiroki Hamano Takashi Angata Shigetsugu Hatakeyama Masataka Kinjo Norimasa Iwasaki

Sialic acid-binding immunoglobulin-like lectin 15 (Siglec-15) is an immunoreceptor that regulates osteoclast development and bone resorption in association with an immunoreceptor tyrosine-based activation motif (ITAM) adaptor protein, DNAX-activating protein 12kDa (DAP12). Although Siglec-15 has an important role in physiologic bone remodeling by modulating RANKL signaling, it is unclear whethe...

2017
Ajiravudh Subarnbhesaj Mutsumi Miyauchi Chea Chanbora Aki Mikuriya Phuong Thao Nguyen Hisako Furusho Nurina Febriyanti Ayuningtyas Minoru Fujita Shigeaki Toratani Masaaki Takechi Shumpei Niida Takashi Takata

BACKGROUND Vascular endothelial growth factor (VEGF) is a highly specific signaling protein for vascular endothelial cells that plays a critical role in tumor growth and invasion through angiogenesis, and may contribute to cell migration and activation of pre-osteoclasts, osteoclasts and some tumor cells. OBJECTIVES We aimed to clarify the detailed roles of VEGF-Flt-1 signaling in bone invasi...

Journal: :Journal of immunology 2000
T Kaneda T Nojima M Nakagawa A Ogasawara H Kaneko T Sato H Mano M Kumegawa Y Hakeda

Differentiation of osteoclasts, the cells primarily responsible for bone resorption, is controlled by a variety of osteotropic hormones and cytokines. Of these factors, receptor activator of NF-kappaB (RANK) ligand (RANKL) has been recently cloned as an essential inducer of osteoclastogenesis in the presence of M-CSF. Here, we isolated a stroma-free population of monocyte/macrophage (M/Mphi)-li...

Journal: :Rheumatology 2015
Yan Ren Bo Yang Yufeng Yin Xiaomei Leng Ying Jiang Lei Zhang Yongzhe Li Xin Li Fengchun Zhang Wei He Xuan Zhang Xuetao Cao

OBJECTIVE CD200/CD200R1 signalling has an immunoregulatory effect on the activation threshold of the inflammatory immune response and maintains immune homeostasis. In this study we evaluated the status of CD200/CD200R1 interaction in patients with RA. METHODS The expression of CD200 and CD200R1 was examined by immunohistochemistry and flow cytometry and was compared between RA patients and he...

Journal: :Blood 2003
Takayuki Yamada Hidetoshi Yamazaki Toshiyuki Yamane Miya Yoshino Hiromi Okuyama Motokazu Tsuneto Tomomi Kurino Shin-Ichi Hayashi Seiji Sakano

Osteoclasts are derived from hematopoietic precursor cells belonging to the monocyte/macrophage lineage. Osteoclast development has been reported to be regulated by several molecules such as macrophage colony-stimulating factor (M-CSF), receptor activator of nuclear factor (NF)-kappaB ligand (RANKL), and a decoy receptor of RANKL, osteoprotegerin (OPG). Recently, it was demonstrated that the No...

2013
Zhiqiang Liu Jingda Xu Haiyan Li Yuhuan Zheng Jin He Huan Liu Yuping Zhong Yong Lu Bangxing Hong Mingjun Zhang Pei Lin Juan Du Jian Hou Jianfei Qian Larry W. Kwak Qing Yi Jing Yang

Multiple myeloma (MM) cells are responsible for aberrant osteoclast (OC) activation. However, when cocultured monocytes, but not OC precursors, with MM cells, we made a novel observation that MM cells inhibited receptor activator of nuclear factor κB ligand (RANKL)-induced increase of OC differentiation, OC gene expression, signaling pathways and bone resorption activity. Our results showed tha...

2017
Jin Hee Park Na Kyung Lee Soo Young Lee

Osteoclasts are bone-resorbing cells that are derived from hematopoietic precursor cells and require macrophage-colony stimulating factor and receptor activator of nuclear factor-κB ligand (RANKL) for their survival, proliferation, differentiation, and activation. The binding of RANKL to its receptor RANK triggers osteoclast precursors to differentiate into osteoclasts. This process depends on ...

2017
Nan Ma Di Yang Hirohiko Okamura Jumpei Teramachi Tomokazu Hasegawa Lihong Qiu Tatsuji Haneji

Periapical lesions are characterized by the destruction of periapical bone, and occur as a result of local inflammatory responses to root canal infection by microorganisms including Porphyromonas endodontalis (P. endodontalis). P. endodontalis and its primary virulence factor, lipopolysaccharide (LPS), are associated with the development of periapical lesions and alveolar bone loss. Interleukin...

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