نتایج جستجو برای: khalil mardom bak

تعداد نتایج: 3778  

Journal: :Arab World English Journal For Translation and Literary Studies 2017

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2004
Damien Arnoult Laura M Bartle Anna Skaletskaya Delphine Poncet Naoufal Zamzami Peter U Park Juanita Sharpe Richard J Youle Victor S Goldmacher

We report that the cytomegalovirus-encoded cell death suppressor vMIA binds Bax and prevents Bax-mediated mitochondrial membrane permeabilization by sequestering Bax at mitochondria in the form of a vMIA-Bax complex. vMIA mutants with a defective mitochondria-targeting domain retain their Bax-binding function but not their ability to suppress mitochondrial membrane permeabilization or cell deat...

Journal: :The Journal of general virology 1999
M Thomas L Banks

Human papillomavirus (HPV) replication occurs in terminally differentiating epithelium, and requires the activation of cellular DNA replication proteins. Unscheduled DNA replication can result in the induction of apoptosis, and the viral E6 protein induces the degradation of p53 to prevent this. It has recently been shown that HPV-18 E6 can also stimulate the degradation of Bak, a pro-apoptotic...

Journal: :Proceedings of the American Mathematical Society 2004

Journal: :International Letters of Social and Humanistic Sciences 2015

2016
Yusuke Nasu Alexander Benke Satoko Arakawa Go J. Yoshida Genki Kawamura Suliana Manley Shigeomi Shimizu Takeaki Ozawa

Apoptosis plays a pivotal role in development and tissue homeostasis in multicellular organisms. Clustering of Bak proteins on the mitochondrial outer membrane is responsible for the induction of apoptosis by evoking a release of pro-apoptotic proteins from mitochondria into cytosol. However, how the protein cluster permeabilizes the mitochondrial membrane remains unclear because elucidation of...

Journal: :The Journal of Cell Biology 2007
Mitch Leslie

Apoptosis promoters spur cell division T wo cell-killing proteins also have a nurturing side, say Russell Jones, Craig Thompson (University of Pennsylvania, Philadelphia, PA), and colleagues. Although the proteins prod lymphocytes to commit suicide, they also prompt the cells to proliferate—and might do the same for a variety of cell types. The two proteins, Bax and Bak, are part of the apoptot...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2012
Biyun Ni Qi Ma Baowei Li Lixia Zhao Yong Liu Yushan Zhu Quan Chen

PURPOSE Bax and Bak are regarded as key mediators for cytochrome c (Cyt c) release and apoptosis. Loss of Bax or Bak is often reported in human cancers and renders resistance of these cancerous cells to chemotherapy. Here, we investigated that phenylarsine oxide (PAO) could induce Bax/Bak-independent apoptosis. EXPERIMENTAL DESIGN Annexin V/propidium iodide (PI) staining, terminal deoxynucleo...

Journal: :The Journal of biological chemistry 2015
Li Huang Junjie Han Danya Ben-Hail Luwei He Baowei Li Ziheng Chen Yueying Wang Yanlei Yang Lei Liu Yushan Zhu Varda Shoshan-Barmatz Hongwei Liu Quan Chen

The pro-apoptotic Bax and Bak proteins are considered central to apoptosis, yet apoptosis occurs in their absence. Here, we asked whether the mitochondrial protein VDAC1 mediates apoptosis independently of Bax/Bak. Upon screening a fungal secondary metabolite library for compounds inducing apoptosis in Bax/Bak-deficient mouse embryonic fibroblasts, we identified cyathin-R, a new cyathane diterp...

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