نتایج جستجو برای: infarction size

تعداد نتایج: 677038  

Journal: :The American journal of Chinese medicine 2006
Li-Ping Yuan Zhi-Wu Chen Fan Li Liu-Yi Dong Fei-Hu Chen

This study was to investigate the effect of total flavones of rhododendra (TFR) on ischemic myocardial injury in rabbits. Rabbit ischemic myocardial injury was induced by occluding the anterior descent of the left artery (LAD). The ECG was recorded; the plasma creatine kinase (CK), nitric oxide (NO) and endothelin-1 (ET-1) levels were measured using spectrophotometry, Griess method and radioimm...

Journal: :The American journal of cardiology 2007
Stijntje D Roes Sebastian Kelle Theodorus A M Kaandorp Thomas Kokocinski Don Poldermans Hildo J Lamb Eric Boersma Ernst E van der Wall Eckart Fleck Albert de Roos Eike Nagel Jeroen J Bax

Currently, left ventricular (LV) ejection fraction (EF) and/or LV volumes are the established predictors of mortality in patients with coronary artery disease (CAD) and severe LV dysfunction. With contrast-enhanced magnetic resonance imaging (MRI), precise delineation of infarct size is now possible. The relative merits of LVEF/LV volumes and infarct size to predict long-term outcome are unknow...

2017
Danish Saleheen Philip C Haycock Wei Zhao Asif Rasheed Adam Taleb Atif Imran Shahid Abbas Faisal Majeed Saba Akhtar Nadeem Qamar Khan Shah Zaman Zia Yaqoob Tahir Saghir Syed Nadeem Hasan Rizvi Anis Memon Nadeem Hayyat Mallick Mohammad Ishaq Syed Zahed Rasheed Fazal-ur-Rehman Memon Khalid Mahmood Naveeduddin Ahmed Philippe Frossard Sotirios Tsimikas Joseph L Witztum Santica Marcovina Manjinder Sandhu Daniel J Rader John Danesh

BACKGROUND The lipoprotein(a) pathway is a causal factor in coronary heart disease. We used a genetic approach to distinguish the relevance of two distinct components of this pathway, apolipoprotein(a) isoform size and circulating lipoprotein(a) concentration, to coronary heart disease. METHODS In this mendelian randomisation study, we measured lipoprotein(a) concentration and determined apol...

2015
Elisa McAlindon Maria Pufulete Chris Lawton Gianni D. Angelini Chiara Bucciarelli-Ducci

AIMS The aim of this study was to evaluate seven methods for quantifying myocardial oedema [2 standard deviation (SD), 3 SD, 5 SD, full width at half maximum (FWHM), Otsu method, manual thresholding, and manual contouring] from T2-weighted short tau inversion recovery (T2w STIR) and also to reassess these same seven methods for quantifying acute infarct size following ST-segment myocardial infa...

Journal: :JACC. Cardiovascular interventions 2015
Steven K White Georg M Frohlich Daniel M Sado Viviana Maestrini Marianna Fontana Thomas A Treibel Shana Tehrani Andrew S Flett Pascal Meier Cono Ariti John R Davies James C Moon Derek M Yellon Derek J Hausenloy

OBJECTIVES This study aimed to determine whether remote ischemic conditioning (RIC) initiated prior to primary percutaneous coronary intervention (PPCI) could reduce myocardial infarct (MI) size in patients presenting with ST-segment elevation myocardial infarction. BACKGROUND RIC, using transient limb ischemia and reperfusion, can protect the heart against acute ischemia-reperfusion injury. ...

Journal: :Circulation 2009
Peter V Johnston Tetsuo Sasano Kevin Mills Robert Evers Shuo-Tsan Lee Rachel Ruckdeschel Smith Albert C Lardo Shenghan Lai Charles Steenbergen Gary Gerstenblith Richard Lange Eduardo Marbán

BACKGROUND Cardiosphere-derived cells (CDCs) isolated from human endomyocardial biopsies reduce infarct size and improve cardiac function in mice. Safety and efficacy testing in large animals is necessary for clinical translation. METHODS AND RESULTS Mesenchymal stem cells, which resemble CDCs in size and thrombogenicity, have been associated with infarction after intracoronary infusion. To m...

2016
Moslem Najafi Elnaz Shaseb Tahereh Eteraf Oskouei

Introduction: In the present study, potential differences between the effects of L-carnitine (LC) and acetyl-Lcarnitine (ALC) on ischemia/reperfusion (I/R)-induced myocardial infarction size were investigated. Methods: Male Wistar rats were randomly divided into five groups and then anesthetized by sodium pentobarbital. Hearts of the animals were removed and quickly mounted on a Langendorff app...

2015
Kenneth Mangion David Carrick Alexander R Payne John D McClure Maureen Mason Mark Petrie Margaret McEntegart Hany Eteiba Keith G Oldroyd Colin Berry

Background In the Preventive Angioplasty in Myocardial Infarction trial (PRAMI; ISRCTN73028481), immediate multivessel PCI (MV-PCI) of non-IRA (infarct related artery) lesions in patients with acute ST elevation myocardial infarction (STEMI) and multivessel coronary disease (MVD) improved long term prognosis. We assessed infarct distribution and size in a pre-specified cardiac magnetic resonanc...

2015
Shokoufeh Hajsadeghi Mitra Chitsazan Mandana Chitsazan Majid Haghjoo Nima Babaali Zahra Norouzzadeh Maryam Mohsenian

BACKGROUND Infarct size is an important surrogate end point for early and late mortality after acute myocardial infarction. Despite the high prevalence of metabolic syndrome in patients with atherosclerotic diseases, adequate data are still lacking regarding the extent of myocardial necrosis after acute myocardial infarction in these patients. OBJECTIVES In the present study we aimed to compa...

Journal: :Journal of the American College of Cardiology 1999
K W Mahaffey J A Puma N A Barbagelata M F DiCarli M A Leesar K F Browne P R Eisenberg R Bolli A C Casas V Molina-Viamonte C Orlandi R Blevins R J Gibbons R M Califf C B Granger

OBJECTIVES The Acute Myocardial Infarction STudy of ADenosine (AMISTAD) trial was designed to test the hypothesis that adenosine as an adjunct to thrombolysis would reduce myocardial infarct size. BACKGROUND Reperfusion therapy for acute myocardial infarction (MI) has been shown to reduce mortality, but reperfusion itself also may have deleterious effects. METHODS The AMISTAD trial was a pr...

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