نتایج جستجو برای: gnrhr
تعداد نتایج: 235 فیلتر نتایج به سال:
Departments of Anatomy Histology and Forensic Medicine and Clinical Physiopathology (AP,MM,GBV), Endocrinology (CC,PF,RS,CMR) and Andrology (MM,ML) Units, Genetic Unit (EP,FT), Departments of Internal Medicine (RGR), Preclinical and Clinical Pharmacology (PF), University of Florence, School of Medicine, Italy and Department of Experimental and Clinical Medicine (TB), University of Catanzaro, It...
The objective of this study was to determine the effects of an luteinizing hormone-releasing hormone (LHRH)-I antagonist, Cetrorelix, on human multiple myeloma (MM) cells and to elucidate the mechanisms of action. We showed that LHRH-I and LHRHR-I genes were expressed in MM cell lines and primary MM cells. Treatment with Cetrorelix inhibited growth and colony-forming ability of myeloma cells, i...
Aim: to review and analyze the use of gonadotropin-releasing hormone (GnRH) antagonists in endometriosis, adenomyosis, non-menstrual pelvic pain, uterine fibroids, prostate cancer, female infertility, precocious puberty. Methods: information used write this manuscript was obtained during a three-month period, between October May 2022, from specialized literature, written English Spanish, relate...
BACKGROUND Immunotherapy is fast emerging as one of the leading modes of treatment of cancer, in combination with chemotherapy and radiation. Use of immunotoxins, proteins bearing a cell-surface receptor-specific antibody conjugated to a toxin, enhances the efficacy of cancer treatment. The toxin Abrin, isolated from the Abrus precatorius plant, is a type II ribosome inactivating protein, has a...
Objective(s): Polycystic ovary syndrome (PCOS) is an endocrinopathy in women, which, unlike its impact on fertility and health of women, there is no clear understanding about the causal mechanisms of this pathogenesis. The aim of this review paper is to investigate the pathophysiological pathways affecting the PCOS etiology, based on functions of gonadotropins– and steroid hormones–related gene...
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