نتایج جستجو برای: genome instability

تعداد نتایج: 315656  

2014
Yu Lei Vivian W. Y. Lui Jennifer R. Grandis Ann Marie Egloff

Head and Neck Squamous Cell Carcinoma (HNSCC) encompasses malignancies that arise in the mucosa of the upper aerodigestive tract. Recent high throughput DNA sequencing revealed HNSCC genes mutations that contribute to several cancer cell characteristics, including dysregulation of cell proliferation and death, intracellular proinflammatory signaling, and autophagy. The PYRIN-domain containing N...

2016
Talya Shacham Ronit Galron Ofer Bihari Sivan Kanner Ari Barzilai

Maintenance of genome stability in the face of DNA damage is essential for cellular homeostasis and prevention of cancer and brain degeneration. The DNA damage response (DDR) is a complex response that is rapidly activated when a DNA lesion occurs in chromosomal DNA. Mutations affecting the proteins involved in the DDR can lead to genomic instability syndromes that involve tissue degeneration, ...

2016
J. Sidney Ang Supipi Duffy Romulo Segovia Peter C. Stirling Philip Hieter

Mutations that cause genome instability are considered important predisposing events that contribute to initiation and progression of cancer. Genome instability arises either due to defects in genes that cause an increased mutation rate (mutator phenotype), or defects in genes that cause chromosome instability (CIN). To extend the catalog of genome instability genes, we systematically explored ...

Journal: :Journal of immunology 2012
Lucia Kato Andre Stanlie Nasim A Begum Maki Kobayashi Masatoshi Aida Tasuku Honjo

An ortholog of activation-induced cytidine deaminase (AID) was, evolutionarily, the first enzyme to generate acquired immune diversity by catalyzing gene conversion and probably somatic hypermutation (SHM). AID began to mediate class switch recombination (CSR) only after the evolution of frogs. Recent studies revealed that the mechanisms for generating immune and genetic diversity share several...

2009
Tobias Dietschy Igor Shevelev Javier Pena-Diaz Daniela Hühn Sandra Kuenzle Raymond Mak Mohammad Fahad Miah Daniel Hess Monika Fey Michael O. Hottiger Pavel Janscak Igor Stagljar

Introduction Genome instability plays a major role in the development and progression of cancer. All organisms have developed pathways to mitigate DNA damage by employing enzymes that are involved in all DNA metabolic processes, including replication, recombination and repair (Tuteja and Tuteja, 2001). The fundamental importance of these enzymes, such as DNA helicases and acetyltransferases, is...

2016
Rebecca E. McIntyre Jérôme Nicod Carla Daniela Robles-Espinoza John Maciejowski Na Cai Jennifer Hill Ruth Verstraten Vivek Iyer Alistair G. Rust Gabriel Balmus Richard Mott Jonathan Flint David J. Adams

In mammals the regulation of genomic instability plays a key role in tumor suppression and also controls genome plasticity, which is important for recombination during the processes of immunity and meiosis. Most studies to identify regulators of genomic instability have been performed in cells in culture or in systems that report on gross rearrangements of the genome, yet subtle differences in ...

Journal: :Seminars in cancer biology 2010
Miriam K Konkel Mark A Batzer

It is now commonly agreed that the human genome is not the stable entity originally presumed. Deletions, duplications, inversions, and insertions are common, and contribute significantly to genomic structural variations (SVs). Their collective impact generates much of the inter-individual genomic diversity observed among humans. Not only do these variations change the structure of the genome; t...

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