a r t i c l e i n f o Keywords: Fire risk Risk map Danger Damage potential Urban–wildland interface zone A forest fire risk model was designed and applied to a South American Mediterranean ecosystem, taking into consideration three analysis groups: fire risk; danger of fire spread, or propagation; and damage potential over economic threat values. The study area for development and validation of...

Field observations of maturely slipped faults show a generally broad zone of damage by cracking and granulation. Nevertheless, large shear deformation, and therefore heat generation, in individual earthquakes takes place with extreme localization to a zone <1–5 mm wide within a finely granulated fault core. Relevant fault weakening processes during large crustal events are therefore likely to b...

BACKGROUND AND PURPOSE Determining how cerebral cortex adapts to sudden focal damage is important for gaining a better understanding of stroke. In this study we used a computational model to examine the hypothesis that cortical map reorganization after a simulated infarct is critically dependent on perilesion excitability and to identify factors that influence the extent of poststroke reorganiz...

The use of cohesive zone models is an e cient way to treat the damage, especially when the crack path is known a priori. This is the case in the modeling of delamination in composite laminates. However, the simulations using cohesive zone models are expensive in a computational point of view. When using implicit time integration scheme or when solving static problems, the non-linearity related ...

The role secretory IgM has in protecting splenic tissue from LPS-induced damage was assessed in mice incapable of secreting IgM but able to express surface IgM and IgD. Within seconds after LPS challenge, 99% of the (131)I-labeled LPS was found in the liver and the spleen of both sIgM-deficient and wild-type mice. In the spleen FITC-labeled LPS was found on the surface of 2F8(+) scavenger recep...

The time course and acinar distribution of bromobenzene-induced hepatic necrosis was studied in the rat. Cellular damage, lipid infiltration, and changes in glycogen deposits were investigated by light microscopy 6, 16, 24, and 48 hr after bromobenzene (BZ) administration. Concomitantly, ultrastructural changes were followed by electron microscopy in each zone of the acinus. To insure accurate ...