نتایج جستجو برای: ctni

تعداد نتایج: 1252  

Journal: :Biophysical journal 2014
Yuanhua Cheng Steffen Lindert Peter Kekenes-Huskey Vijay S Rao R John Solaro Paul R Rosevear Rommie Amaro Andrew D McCulloch J Andrew McCammon Michael Regnier

During β-adrenergic stimulation, cardiac troponin I (cTnI) is phosphorylated by protein kinase A (PKA) at sites S23/S24, located at the N-terminus of cTnI. This phosphorylation has been shown to decrease KCa and pCa50, and weaken the cTnC-cTnI (C-I) interaction. We recently reported that phosphorylation results in an increase in the rate of early, slow phase of relaxation (kREL,slow) and a decr...

Journal: :Clinical chemistry 2013
Paul M McKie Denise M Heublein Christopher G Scott Mary Lou Gantzer Ramila A Mehta Richard J Rodeheffer Margaret M Redfield John C Burnett Allan S Jaffe

BACKGROUND High-sensitivity cardiac troponin (hs-cTn) assays are now available that can detect measurable troponin in significantly more individuals in the general population than conventional assays. The clinical use of these hs-cTn assays depends on the development of proper reference values. Therefore, our objective was to define hs-cTnI reference values and determinants in the general commu...

Journal: :PloS one 2016
Changlin Zhang Rong Liu Jiansong Yuan Jingang Cui Fenghuan Hu Weixian Yang Yan Zhang Youzhou Chen Shubin Qiao

BACKGROUND Both high-sensitivity cardiac troponin T and B-type natriuretic peptide are useful in detecting myocardial fibrosis, as determined by late gadolinium enhancement (LGE) cardiovascular magnetic resonance (CMR), in patients with non-obstructive hypertrophic cardiomyopathy. However, their values to predict myocardial fibrosis in hypertrophic obstructive cardiomyopathy (HOCM) remain uncle...

Journal: :Clinical chemistry 2000
D Wayand H Baum G Schätzle J Schärf D Neumeier

BACKGROUND In patients suffering from end-stage renal failure, cardiac troponin T (cTnT) and I (cTnI) may be increased in serum without other signs of acute myocardial damage. Whether these increases are specific to myocardial injury or nonspecific is not completely clear. METHODS We investigated time courses of cTnT and cTnI over 1 year and the clinical outcome over 2 years in 59 patients wi...

2017
Mark J Hamilton Yvonne Robb Sarah Cumming Helen Gregory Alexis Duncan Monika Rahman Anne McKeown Catherine McWilliam John Dean Alison Wilcox Maria E Farrugia Anneli Cooper Josephine McGhie Berit Adam Richard Petty Cheryl Longman Iain Findlay Alan Japp Darren G Monckton Martin A Denvir

OBJECTIVE High sensitivity plasma cardiac troponin-I (cTnI) is emerging as a strong predictor of cardiac events in a variety of settings. We have explored its utility in patients with myotonic dystrophy type 1 (DM1). METHODS 117 patients with DM1 were recruited from routine outpatient clinics across three health boards. A single measurement of cTnI was made using the ARCHITECT STAT Troponin I...

Journal: :Polish journal of veterinary sciences 2011
A Noszczyk-Nowak

The purpose of this study was to develop prognostic models for heart failure in dogs with dilated cardiomyopathy (DCM). The prospective study included 26 dogs with DCM and 58 healthy dogs. The ervation time median was 250 days (1-600 days). All the dogs were clinically examined, had echocardiography, electrocardiography, and morphological and biochemical blood sampling. Twenty four deaths were ...

Journal: :European journal of cardio-thoracic surgery : official journal of the European Association for Cardio-thoracic Surgery 1998
S Chocron J P Etievent

OBJECTIVE The definition of a reliable and generally accepted diagnostic standard for perioperative myocardial damage is desirable. Cardiac troponin I (cTnI) is highly specific for myocardial tissue and can be measured rapidly. The aim of our study was to evaluate the diagnostic potential of cTnI for myocardial lesions in patients undergoing coronary artery bypass surgery (CABG). METHODS A to...

Journal: :Heart 2005
T Nageh R A Sherwood B M Harris M R Thomas

OBJECTIVE To assess the role of cardiac troponin I (cTnI) in predicting outcome after percutaneous coronary intervention (PCI). METHODS AND RESULTS cTnI was measured immediately before and at 6, 14, and 24 hours after PCI in 316 consecutive patients with stable symptoms and native coronary artery disease. The study end point was the occurrence of major adverse cardiac events (MACE) at 30 days...

Journal: :Circulation 2005
Atsushi Sanbe Jeanne James Volkan Tuzcu Selman Nas Lisa Martin James Gulick Hanna Osinska Sadayappan Sakthivel Raisa Klevitsky Kenneth S Ginsburg Donald M Bers Bruce Zinman Edward G Lakatta Jeffrey Robbins

BACKGROUND Transgenic and gene-targeted models have focused on the mouse. Fundamental differences between the mouse and human exist in Ca2+ handling during contraction/relaxation and in alterations in Ca2+ flux during heart failure, with the rabbit more accurately reflecting the human system. METHODS AND RESULTS Cardiac troponin I (cTnI) mutations can cause familial hypertrophic cardiomyopath...

2015
Aurélien Frobert Jérémy Valentin Jean-Luc Magnin Erwin Riedo Stéphane Cook Marie-Noëlle Giraud

UNLABELLED Coronary artery ligations to induce myocardial infarction (MI) in mice and rats are widely used in preclinical investigation. However, myocardial ischemic damage and subsequent infarct size are highly variable. The lack of standardization of the model impairs the probability of effective translation to the clinic. Cardiac Troponin I (cTnI) is a major clinically relevant biomarker. ...

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