نتایج جستجو برای: c myc

تعداد نتایج: 1062338  

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2005
Xiao-Yong Zhang Lauren M DeSalle Jagruti H Patel Anthony J Capobianco Duonan Yu Andrei Thomas-Tikhonenko Steven B McMahon

The c-myc oncogene is among the most commonly overexpressed genes in human cancer. c-myc encodes a basic helix-loop-helix/leucine zipper (bHLH/LZ) transcription factor (c-MYC) that activates a cascade of downstream targets that ultimately mediate cellular transformation. Although a large number of genes are regulated by c-MYC, only a few have been functionally linked to c-MYC-mediated transform...

2017
Antonio Cigliano Maria G. Pilo Lei Li Gavinella Latte Marta Szydlowska Maria M. Simile Panagiotis Paliogiannis Li Che Giovanni M. Pes Giuseppe Palmieri Maria C. Sini Antonio Cossu Alberto Porcu Gianpaolo Vidili Maria A. Seddaiu Rosa M. Pascale Silvia Ribback Frank Dombrowski Xin Chen Diego F. Calvisi

Deregulated activity of the c-Myc protooncogene is a frequent molecular event underlying mouse and human hepatocarcinogenesis. Nonetheless, the mechanisms sustaining c-Myc oncogenic activity in liver cancer remain scarcely delineated. Recently, we showed that the mammalian target of rapamycin complex 1 (mTORC1) cascade is induced and necessary for c-Myc dependent liver tumor development and pro...

2009
Kristin Robinson Nichaya Asawachaicharn Denise A. Galloway Carla Grandori

c-Myc interacts with components of the pre-replication complex and directly regulates DNA replication [1]. However the consequences of this novel c-Myc function on cell cycle dynamics and replication-associated damage are unknown. Here, we show that c-Myc overexpression in primary human fibroblasts markedly accelerates S-phase while c-Myc deficient fibroblasts exhibit a prolonged S-phase. We al...

2012
Oscar M. Pello Raphael Chèvre Damya Laoui Alba De Juan Fidel Lolo María Jesús Andrés-Manzano Manuel Serrano Jo A. Van Ginderachter Vicente Andrés

Although tumor-associated macrophages (TAMs) are involved in tumor growth and metastasis, the mechanisms controlling their pro-tumoral activities remain largely unknown. The transcription factor c-MYC has been recently shown to regulate in vitro human macrophage polarization and be expressed in macrophages infiltrating human tumors. In this study, we exploited the predominant expression of LysM...

Journal: :Cancer research 2000
L F Barr S E Campbell G B Diette E W Gabrielson S Kim H Shim C V Dang

The c-myc oncogene is frequently amplified in cells grown from lung tumors and has been linked to the malignancy of these cancers. In support of this, c-myc transfection enhances the in vitro proliferation and soft agar cloning of human small cell lung cancer (SCLC) cells. In this study, we surprisingly found that c-myc expression suppressed the formation of tumors by SCLC cells in athymic nude...

Journal: :Cancer research 1995
S Kimura T Maekawa K Hirakawa A Murakami T Abe

This study was designed using c-myc antisense oligodeoxynucleoside phosphorothioate (AS PS-ODN) to evaluate how alterations of c-myc expression in HL-60 human myeloid leukemia cells could influence the induction of apoptosis. Unexpectedly both the continuous down-regulation of c-myc expression caused by exposure to c-myc AS PS-ODN and up-regulation after its withdrawal influenced apoptosis. We ...

Journal: :American journal of clinical pathology 2012
Tonia Cenci Maurizio Martini Nicola Montano Quintino G D'Alessandris Maria Laura Falchetti Daniela Annibali Mauro Savino Federico Bianchi Francesco Pierconti Sergio Nasi Roberto Pallini Luigi Maria Larocca

Although the c-Myc oncogene is frequently deregulated in human cancer, its involvement in the pathogenesis of glioblastoma is not clear. We conducted immunohistochemical analysis of the expression of c-Myc, polycomb ring finger oncogene (BMI1), and acetylation of the lysine 9 (H3K9Ac) of histone 3 in 48 patients with glioblastoma who underwent surgery followed by radiotherapy and temozolomide t...

2016
Rui Gao Lan Wang Hao Cai Jingjing Zhu Long Yu

RNF12/RLIM is a RING domain-containing E3 ubiquitin ligase whose function has only begun to be elucidated recently. Although RLIM was reported to play important roles in some biological processes such as imprinted X-chromosome inactivation and regulation of TGF-β pathway etc., other functions of RLIM are largely unknown. Here, we identified RLIM as a novel E3 ubiquitin ligase for c-Myc, one of ...

Journal: :Cancer research 1988
J Boultwood F S Wyllie E D Williams D Wynford-Thomas

In view of the frequent reports of the increased expression of myc oncogenes in several neuroendocrine tumor types, we have investigated c- and N-myc expression in human medullary carcinoma, a malignant tumor derived from the neuroendocrine "C"-cell subpopulation of the thyroid gland. In situ nucleic acid hybridization was used to permit analysis not only of tumors but also of normal C-cells wh...

2009
Lei Yu Michael J. Hitchler Wenqing Sun Ehab H. Sarsour Prabhat C. Goswami Aloysius J. Klingelhutz Frederick E. Domann

AP-2alpha and c-MYC are important transcription factors involved in multiple cellular processes. They each display the paradoxical capacities to stimulate both cell proliferation and apoptosis under different conditions. In the present study we found that over expression of c-MYC was associated with accumulation of reactive oxygen species (ROS) and apoptosis in human keratinocytes, both of whic...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید