نتایج جستجو برای: braf

تعداد نتایج: 7505  

Journal: :Genetics and molecular research : GMR 2015
C L Shi Y Sun C Ding Y C Lv H D Qin

This study sought to investigate the correlations of V-raf murine sarcoma viral oncogene homolog B1 (BRAF) gene mutations with the clinicopathologic features of papillary thyroid carcinoma and central lymph node metastasis. We retrospectively analyzed the 2-year medical records of patients who underwent surgery for papillary thyroid carcinoma. After screening, the records of 126 patients who me...

2018
Molly C Peters Allyson Minton Otto Phanstiel Iv Susan K Gilmour

Mutant serine/threonine protein kinase B-Raf (BRAF) protein is expressed in over half of all melanoma tumors. Although BRAF inhibitors (BRAFi) elicit rapid anti-tumor responses in the majority of patients with mutant BRAF melanoma, the tumors inevitably relapse after a short time. We hypothesized that polyamines are essential for tumor survival in mutant BRAF melanomas. These tumors rely on bot...

2015
Luigi Fattore Debora Malpicci Emanuele Marra Francesca Belleudi Alessia Noto Claudia De Vitis Maria Elena Pisanu Pierpaolo Coluccia Rosa Camerlingo Giuseppe Roscilli Antoni Ribas Arianna Di Napoli Maria Rosaria Torrisi Luigi Aurisicchio Paolo Antonio Ascierto Rita Mancini Gennaro Ciliberto

Patients with metastatic melanoma bearing V600 mutations in BRAF oncogene clinically benefit from the treatment with BRAF inhibitors alone or in combination with MEK inhibitors. However, a limitation to such treatment is the occurrence of resistance. Tackling the adaptive changes helping cells survive from drug treatment may offer new therapeutic opportunities. Very recently the ErbB3 receptor ...

2017
Qian Li Jianjun Yuan Yan Wang Yuanpeng Zhai

The aim of the present study was to investigate the association between the BRAF V600E mutation and ultrasound features of the thyroid in papillary thyroid carcinoma (PTC). Fresh thyroid carcinoma tissue and paracarcinoma tissue were obtained from 34 patients undergoing surgery for PTC. The BRAF V600E mutation was detected by PCR amplification and direct DNA sequencing. The thyroid ultrasound r...

Journal: :Cancer discovery 2012
Kimberly Brown Dahlman Junfeng Xia Katherine Hutchinson Charles Ng Donald Hucks Peilin Jia Mohammad Atefi Zengliu Su Suzanne Branch Pamela L Lyle Donna J Hicks Viviana Bozon John A Glaspy Neal Rosen David B Solit James L Netterville Cindy L Vnencak-Jones Jeffrey A Sosman Antoni Ribas Zhongming Zhao William Pao

UNLABELLED Kinase inhibitors are accepted treatment for metastatic melanomas that harbor specific driver mutations in BRAF or KIT, but only 40% to 50% of cases are positive. To uncover other potential targetable mutations, we conducted whole-genome sequencing of a highly aggressive BRAF (V600) and KIT (W557, V559, L576, K642, and D816) wild-type melanoma. Surprisingly, we found a somatic BRAF(L...

Journal: :Cancer discovery 2015
Marian M Deuker Victoria Marsh Durban Wayne A Phillips Martin McMahon

UNLABELLED Phosphatidylinositide 3' (PI3')-lipid signaling cooperates with oncogenic BRAF(V600E) to promote melanomagenesis. Sustained PI3'-lipid production commonly occurs via silencing of the PI3'-lipid phosphatase PTEN or, less commonly, through mutational activation of PIK3CA, encoding the 110-kDa catalytic subunit of PI3'-kinase-α (PI3Kα). To define the PI3K catalytic isoform dependency of...

2013
Alexander Skorokhod Peter Helmbold Benedikt Brors Peter Schirmacher Alexander Enk Roland Penzel

Malignant melanoma is a highly-aggressive type of malignancy with considerable metastatic potential and frequent resistance to cytotoxic agents. BRAF mutant protein was recently recognized as therapeutic target in metastatic melanoma. We present a newly-developed U-BRAF approach – a universal pyrosequencing-based assay for mutation detection within activation segment in exon 15 of human braf. W...

Journal: :Science signaling 2015
Pol Margalef Carlota Colomer Alberto Villanueva Clara Montagut Mar Iglesias Beatriz Bellosillo Ramón Salazar María Martínez-Iniesta Anna Bigas Lluís Espinosa

KRAS mutations contribute to cell proliferation and survival in numerous cancers, including colorectal cancers (CRC). One pathway through which mutant KRAS acts is an inflammatory pathway that involves the kinase IKK and activates the transcription factor NF-κB. BRAF, a kinase that is downstream of KRAS, is mutated in a subset of CRC and is predictive of poor prognosis and therapeutic resistanc...

2012
Ryan B. Corcoran Hiromichi Ebi Alexa B. Turke Erin M. Coffee Michiya Nishino Alexandria P. Cogdill Ronald D. Brown Patricia Della Pelle Dora Dias-Santagata Kenneth E. Hung Keith T. Flaherty Adriano Piris Jennifer A. Wargo Jeffrey Settleman Mari Mino-Kenudson Jeffrey A. Engelman

BRAF mutations occur in 10% to 15% of colorectal cancers and confer adverse outcome in the metastatic setting. Although RAF inhibitors such as vemurafenib (PLX4032) have proven effective in the treatment of BRAF-mutant melanoma, they are surprisingly ineffective in BRAF-mutant colorectal cancers, and the reason for this disparity remains unclear. Compared with BRAF-mutant melanoma cells, BRAF-m...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2011
Lisa M Caronia John E Phay Manisha H Shah

BRAF, a cytoplasmic serine-threonine protein kinase, plays a critical role in cell signaling as an activator within the mitogen-activated protein kinase (MAPK) pathway. The most common BRAF mutation is the V600E transversion, which causes constitutive kinase activity. This mutation has been found in a multitude of human cancers, including both papillary thyroid cancer (PTC) and papillary-derive...

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