نتایج جستجو برای: a3243g mutation

تعداد نتایج: 291433  

ژورنال: :مجله دیابت و متابولیسم ایران 0
باقر لاریجانی bagher larijani endocrinology and metabolism research center, tehran university of medical sciencesمرکز تحقیقات غدد درون ریز و متابولیسم، دانشگاه علوم پزشکی تهران مسعود هوشمند masoud houshmand national institute for genetic engineering and biotechnologyپژوهشگاه ملی مهندسی ژنتیک و زیست فناوری قمر سلطان دراج ghamar soltan dorraj national institute for genetic engineering and biotechnologyپژوهشگاه ملی مهندسی ژنتیک و زیست فناوری فرزانه درویش زاده farzaneh darvishzadeh endocrinology and metabolism research center, tehran university of medical sciencesمرکز تحقیقات غدد درون ریز و متابولیسم، دانشگاه علوم پزشکی تهران

میتوکندری یکی از ارگان های داخل سلولی است که dna مخصوص به خود دارد. تا کنون تعدادی بیماری ناشی از جهش mtdna گزارش شده است. یکی از این جهش ها، جهش a3243g و حذف 5kb می باشد. عواقب این جهش های ژنی طیفی از بیماری ها را بوجود می آورد که یکی از آنها دیابت نوع 2 است. هدف از این مطالعه تعیین جهش a3243gtrnaleu و حذف 5kb در بیماران دیابتی نوع 2 می باشد. روش ها: dna 130 بیمار دیابت نوع 2 که میزان انسولین ...

Journal: :Human molecular genetics 2009
Michelina Iacovino Caroline Granycome Hiroshi Sembongi Monika Bokori-Brown Ronald A Butow Ian J Holt Joseph M Bateman

Maintenance of an intact mitochondrial genome is essential for oxidative phosphorylation in all eukaryotes. Depletion of mitochondrial genome copy number can have severe pathological consequences due to loss of respiratory capacity. In Saccharomyces cerevisiae, several bifunctional metabolic enzymes have been shown to be required for mitochondrial DNA (mtDNA) maintenance. For example, Ilv5 is r...

Journal: :Journal of neurology, neurosurgery, and psychiatry 1996
G M Fabrizi E Cardaioli G S Grieco T Cavallaro A Malandrini L Manneschi M T Dotti A Federico G Guazzi

OBJECTIVE To verify the phenotype to genotype correlations of mitochondrial DNA (mtDNA) related disorders in an atypical maternally inherited encephalomyopathy. METHODS Neuroradiological, morphological, biochemical, and molecular genetic analyses were performed on the affected members of a pedigree harbouring the heteroplasmic A to G transition at nucleotide 3243 of the mitochondrial tRNALeu(...

2015
Frederico Lisboa Nogueira Livia Villela Costa Nadine Márcia de Faria Henry Houlden Daniel Dutra Romualdo Silva

Results We report a case of a 44-year's old woman admitted to the Internal Medicine ward with a two weeks history of signs and symptoms of community-acquired pneumonia and heart failure. She was known to have DM since the third decade of life, hearing loss since the adolescence in addition to hypertension and was in use of metformin, losartan, simvastatin and NPH insulin, with recurrent episode...

Journal: :Archives of neurology 2005
Liana G Apostolova Matthew White Steven A Moore Patricia H Davis

BACKGROUND Myopathy, encephalopathy, lactic acidosis, and stroke-like episodes (MELAS) syndrome typically manifests in adults younger than 40 years with encephalopathy, stroke-like episodes, and lactic acidosis. Magnetic resonance imaging (MRI) abnormalities typically involve the cortical gray and the adjacent subcortical white matter. OBJECTIVE To describe a 58-year-old woman diagnosed with ...

2017
Dar-Shong Lin Shu-Huei Kao Che-Sheng Ho Yau-Huei Wei Pi-Lien Hung Mei-Hsin Hsu Tsu-Yen Wu Tuan-Jen Wang Yuan-Ren Jian Tsung-Han Lee Ming-Fu Chiang

Mitochondrial encephalomyopathy, lactic acidosis, and stroke-like episodes (MELAS) syndrome is most commonly caused by the A3243G mutation of mitochondrial DNA. The capacity to utilize fatty acid or glucose as a fuel source and how such dynamic switches of metabolic fuel preferences and transcriptional modulation of adaptive mechanism in response to energy deficiency in MELAS syndrome have not ...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید