نتایج جستجو برای: sirt

تعداد نتایج: 742  

2017
Cheng Zeng Xiaoyan Hu Weiyang He Yanfeng Wang Ling Li Yan Xiong Qifa Ye

Hypothermic machine perfusion (HMP) effectively reduces ischemia‑reperfusion injury (IRI) in livers donated after circulatory death (DCD) when compared with cold storage (CS). However, the underlying mechanisms remain unclear. The current study aimed to investigate the cellular mechanisms by which HMP ameliorates the inflammatory response during IRI. Adult male Sprague‑Dawley rat livers were ex...

2014
Mohamed K. Hassan Hidemichi Watari Alaa-eldin Salah-eldin Ahmed S. Sultan Zainab Mohamed Yoichiro Fujioka Yusuke Ohba Noriaki Sakuragi

This study describes the sensitization mechanism to thermal stress by histone deacetylase inhibitors (HDACIs) in lung cancer cells and shows that Ku70, based on its acetylation status, mediates the protection of lung cancer from hyperthermia (42.5°C, 1-6 hrs). Ku70 regulates apoptosis by sequestering pro-apoptotic Bax. However, its role in thermal stress is not fully understood. The findings sh...

Journal: :Journal of nuclear medicine : official publication, Society of Nuclear Medicine 2010
Hojjat Ahmadzadehfar Amir Sabet Kim Biermann Marianne Muckle Holger Brockmann Christiane Kuhl Kai Wilhelm Hans-Jürgen Biersack Samer Ezziddin

UNLABELLED Selective internal radiation therapy (SIRT), a catheter-based liver-directed modality for treating primary and metastatic liver cancer, requires appropriate planning to maximize its therapeutic response and minimize its side effects. (99m)Tc-macroaggregated albumin (MAA) scanning should precede the therapy to detect any extrahepatic shunting to the lung or gastrointestinal tract. Our...

Journal: :Molecular cancer therapeutics 2014
Kartick C Pramanik Neel M Fofaria Parul Gupta Sanjay K Srivastava

Here, we investigated the potential mechanism of capsaicin-mediated apoptosis in pancreatic cancer cells. Capsaicin treatment phosphorylated c-jun-NH2-kinase (JNK); forkhead box transcription factor, class O (FOXO1); and BIM in BxPC-3, AsPC-1, and L3.6PL cells. The expression of BIM increased in response to capsaicin treatment. Capsaicin treatment caused cleavage of caspase-3 and PARP, indicati...

2014
Kartick C. Pramanik Neel M. Fofaria Parul Gupta Sanjay K. Srivastava

Here, we investigated the potential mechanism of capsaicin-mediated apoptosis in pancreatic cancer cells. Capsaicin treatment phosphorylated c-jun-NH2-kinase (JNK); forkhead box transcription factor, class O (FOXO1); and BIM in BxPC-3, AsPC-1, and L3.6PL cells. The expression of BIM increased in response to capsaicin treatment. Capsaicin treatment caused cleavage of caspase-3 and PARP, indicati...

2017
Ya-Feng Li Gayani Nanayakkara Yu Sun Xinyuan Li Luqiao Wang Ramon Cueto Ying Shao Hangfei Fu Candice Johnson Jiali Cheng Xiongwen Chen Wenhui Hu Jun Yu Eric T Choi Hong Wang Xiao-Feng Yang

BACKGROUND It is well established that caspase-1 exerts its biological activities through its downstream targets such as IL-1β, IL-18, and Sirt-1. The microarray datasets derived from various caspase-1 knockout tissues indicated that caspase-1 can significantly impact the transcriptome. However, it is not known whether all the effects exerted by caspase-1 on transcriptome are mediated only by i...

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