نتایج جستجو برای: repeat

تعداد نتایج: 73564  

2012
Tatsuaki Kurosaki Shintaroh Ueda Takafumi Ishida Koji Abe Kinji Ohno Tohru Matsuura

Myotonic dystrophy type 2 (DM2) is a subtype of the myotonic dystrophies, caused by expansion of a tetranucleotide CCTG repeat in intron 1 of the zinc finger protein 9 (ZNF9) gene. The expansions are extremely unstable and variable, ranging from 75-11,000 CCTG repeats. This unprecedented repeat size and somatic heterogeneity make molecular diagnosis of DM2 difficult, and yield variable clinical...

2018
Roberto Simone Rubika Balendra Thomas G Moens Elisavet Preza Katherine M Wilson Amanda Heslegrave Nathan S Woodling Teresa Niccoli Javier Gilbert-Jaramillo Samir Abdelkarim Emma L Clayton Mica Clarke Marie-Therese Konrad Andrew J Nicoll Jamie S Mitchell Andrea Calvo Adriano Chio Henry Houlden James M Polke Mohamed A Ismail Chad E Stephens Tam Vo Abdelbasset A Farahat W David Wilson David W Boykin Henrik Zetterberg Linda Partridge Selina Wray Gary Parkinson Stephen Neidle Rickie Patani Pietro Fratta Adrian M Isaacs

Intronic GGGGCC repeat expansions in C9orf72 are the most common known cause of frontotemporal dementia (FTD) and amyotrophic lateral sclerosis (ALS), which are characterised by degeneration of cortical and motor neurons, respectively. Repeat expansions have been proposed to cause disease by both the repeat RNA forming foci that sequester RNA-binding proteins and through toxic dipeptide repeat ...

Journal: :Genetics 1992
K J Dornfeld D M Livingston

Using plasmids capable of undergoing intramolecular recombination, we have compared the rates and the molecular outcomes of recombination events in a wild-type and a rad52 strain of Saccharomyces cerevisiae. The plasmids contain his3 heteroalleles oriented in either an inverted or a direct repeat. Inverted repeat plasmids recombine approximately 20-fold less frequently in the mutant than in the...

2014
Beatriz A. Santillan Christopher Moye David Mittelman John H. Wilson

Trinucleotide repeats can be highly unstable, mutating far more frequently than point mutations. Repeats typically mutate by addition or loss of units of the repeat. CAG repeat expansions in humans trigger neurological diseases that include myotonic dystrophy, Huntington disease, and several spinocerebellar ataxias. In human cells, diverse mechanisms promote CAG repeat instability, and in mice,...

Journal: :Queueing Systems 2017

Journal: :Journal of Housing Economics 1991

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