نتایج جستجو برای: nonsyndromic deafness

تعداد نتایج: 9132  

Journal: :Biochimica et biophysica acta 2003
Juraj Gregan Lut Van Laer Louis D Lieto Guy Van Camp Stephen E Kearsey

A mutation in human DFNA5 is associated with autosomal dominant nonsyndromic hearing impairment. The function of DFNA5 protein remains unknown and no experimental model has been described so far. Here we describe fission yeast Schizosaccharomyces pombe as a model organism for studying the function of heterologously expressed DFNA5. We have expressed wild-type as well as mutant DFNA5 alleles und...

Journal: :Archives of otolaryngology--head & neck surgery 2005
Abram P Vore Eugene H Chang Jane E Hoppe Merlin G Butler Shawnia Forrester Michael C Schneider Luke L H Smith Daniel W Burke Colleen A Campbell Richard J H Smith

OBJECTIVE To analyze the physical manifestations and genetic features of 2 families segregating X-linked deafness, which is most commonly reported to be caused by mutations of the POU domain gene POU3F4 at the DFN3 locus. DESIGN Computed tomographic study of the temporal bone in probands from each family, followed by mutation screening and deletion mapping of POU3F4 in family members. SETTI...

Journal: :Physiological genomics 2009
Pu Dai Andrew K Stewart Fouad Chebib Ann Hsu Julia Rozenfeld Deliang Huang Dongyang Kang Va Lip Hong Fang Hong Shao Xin Liu Fei Yu Huijun Yuan Margaret Kenna David T Miller Yiping Shen Weiyan Yang Israel Zelikovic Orah S Platt Dongyi Han Seth L Alper Bai-Lin Wu

Mutations of the human SLC26A4/PDS gene constitute the most common cause of syndromic and nonsyndromic hearing loss. Definition of the SLC26A4 mutation spectrum among different populations with sensorineural hearing loss is important for development of optimal genetic screening services for congenital hearing impairment. We screened for SLC26A4 mutations among Chinese and U.S. subjects with hea...

Journal: :Investigative ophthalmology & visual science 2009
David S Williams Tomas S Aleman Concepción Lillo Vanda S Lopes Louise C Hughes Edwin M Stone Samuel G Jacobson

PURPOSE To investigate the expression of harmonin in the mouse retina, test for ultrastructural and physiological mutant phenotypes in the retina of an Ush1c mutant mouse, and define in detail the retinal phenotype in human USH1C. METHODS Antibodies were generated against harmonin. Harmonin isoform distribution was examined by Western blot analysis and immunocytochemistry. Retinas of deaf cir...

2015
Mariella Simon Elodie M. Richard Xinjian Wang Mohsin Shahzad Vincent H. Huang Tanveer A. Qaiser Prasanth Potluri Sarah E. Mahl Antonio Davila Sabiha Nazli Saege Hancock Margret Yu Jay Gargus Richard Chang Nada Al-sheqaih William G. Newman Jose Abdenur Arnold Starr Rashmi Hegde Thomas Dorn Anke Busch Eddie Park Jie Wu Hagen Schwenzer Adrian Flierl Catherine Florentz Marie Sissler Shaheen N. Khan Ronghua Li Min-Xin Guan Thomas B. Friedman Doris K. Wu Vincent Procaccio Sheikh Riazuddin Douglas C. Wallace Zubair M. Ahmed Taosheng Huang Saima Riazuddin

Here we demonstrate association of variants in the mitochondrial asparaginyl-tRNA synthetase NARS2 with human hearing loss and Leigh syndrome. A homozygous missense mutation ([c.637G>T; p.Val213Phe]) is the underlying cause of nonsyndromic hearing loss (DFNB94) and compound heterozygous mutations ([c.969T>A; p.Tyr323*] + [c.1142A>G; p.Asn381Ser]) result in mitochondrial respiratory chain defici...

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