نتایج جستجو برای: e cadherin gene expression

تعداد نتایج: 2537700  

Journal: :The Journal of Cell Biology 2001
Cara J. Gottardi Ellen Wong Barry M. Gumbiner

E-cadherin is a tumor suppressor protein with a well-established role in cell-cell adhesion. Adhesion could contribute to tumor suppression either by physically joining cells or by facilitating other juxtacrine signaling events. Alternatively, E-cadherin tumor suppressor activity could result from binding and antagonizing the nuclear signaling function of beta-catenin, a known proto-oncogene. T...

2017
Joe M. Segal Christopher M. Ward

We have previously shown that E-cadherin regulates the naive pluripotent state of mouse embryonic stem cells (mESCs) by enabling LIF-dependent STAT3 phosphorylation, with E-cadherin null mESCs exhibiting over 3000 gene transcript alterations and a switch to Activin/Nodal-dependent pluripotency. However, elucidation of the exact mechanisms associated with E-cadherin function in mESCs is compound...

Journal: :The Journal of clinical investigation 2012
Chenfang Dong Yadi Wu Jun Yao Yifan Wang Yinhua Yu Piotr G Rychahou B Mark Evers Binhua P Zhou

Breast cancers are highly heterogeneous but can be grouped into subtypes based on several criteria, including level of expression of certain markers. Claudin-low breast cancer (CLBC) is associated with early metastasis and resistance to chemotherapy, while gene profiling indicates it is characterized by the expression of markers of epithelial-mesenchymal transition (EMT) - a phenotypic conversi...

Journal: :International journal of oncology 2014
Nicoletta Fortunati Francesca Marano Andrea Bandino Roberto Frairia Maria Graziella Catalano Giuseppe Boccuzzi

Triple-negative breast cancer (TNBC) is a very aggressive type of tumour and its aggressiveness is linked to E-cadherin downregulation. In estrogen-sensitive breast cancer, high levels of E-cadherin fit with high levels of ERα and MTA3 (a component of the transcription Mi-2/NuRD complex with intrinsic DAC activity). In TNBC the E-cadherin downregulation could be due to epigenetic silencing of t...

مقدمه: ژن پروتئینE-cadherin  با متاستاز و پیش آگهی در سرطان همراه می باشد و متیلاسیون پروموتر ژن منجر به کاهش بیان ژن می گردد. اینترلوکین 17 به عنوان یک سایتوکاین التهابی، می تواند نقش موثری در ایجاد متیلاسیون داشته باشد. در این تحقیق ما به بررسی نقش پلی مورفیسم اینترلوکین 17 بر متیلاسیون پروموتر ژن E-cadherin در سرطان پستان پرداخته ایم. مواد و روش ها: در این تحقیق موردی-شاهدی، از بافت سرطانی ...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2004
Yutaka Shimada Seiji Yamasaki Yosuke Hashimoto Tetsuo Ito Jun-ichiro Kawamura Toshiya Soma Yoshinori Ino Yukihiro Nakanishi Michiie Sakamoto Setsuo Hirohashi Masayuki Imamura

PURPOSE Dysadherin, a cancer-associated cell membrane glycoprotein, has been reported to down-regulate E-cadherin expression and promote metastasis. To evaluate the role of dysadherin in gastric cancer, we examined dysadherin and E-cadherin expression in gastric cancer patients. EXPERIMENTAL DESIGN Dysadherin and E-cadherin expression were evaluated in 276 gastric cancer patients by immunohis...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2014
Hayato Nakagawa Yohko Hikiba Yoshihiro Hirata Joan Font-Burgada Kei Sakamoto Yoku Hayakawa Koji Taniguchi Atsushi Umemura Hiroto Kinoshita Kosuke Sakitani Yuji Nishikawa Kenji Hirano Tsuneo Ikenoue Hideaki Ijichi Debanjan Dhar Wataru Shibata Masao Akanuma Kazuhiko Koike Michael Karin Shin Maeda

E-cadherin is an important adhesion molecule whose loss is associated with progression and poor prognosis of liver cancer. However, it is unclear whether the loss of E-cadherin is a real culprit or a bystander in liver cancer progression. In addition, the precise role of E-cadherin in maintaining liver homeostasis is also still unknown, especially in vivo. Here we demonstrate that liver-specifi...

Journal: :International journal of oncology 2015
Tadashi Asakura Noriko Yamaguchi Kiyoshi Ohkawa Kiyotsugu Yoshida

Downregulation of E-cadherin (gene: CDH1) plays an important role in epithelial-mesenchymal transition (EMT), which is critical for normal development and disease states. As a result of long-term treatment of endometrial carcinoma Ishikawa cells with epoxomicin (EXM), the cells exhibited the phenotype for EXM-resistance (Ish/EXM cells). Moreover, CDH1 mRNA and its protein were suppressed and EM...

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