نتایج جستجو برای: cytogenetic effects

تعداد نتایج: 1555618  

Journal: :Journal of medical genetics 1983
J L Watt D A Couzin

An extremely rare case of a child with three balanced reciprocal translocations involving six different autosomes is described. These abnormalities have apparently arisen de novo and seem to have only relatively minor phenotypic effects. The meiotic possibilities are discussed and cytogenetic markers suggest that the damage may have occurred in a paternal gamete.

Journal: :Journal of the College of Physicians and Surgeons--Pakistan : JCPSP 2008
Khurshid Ahmed Khan Ayesha Junaid Nauman Saleem Siddiqui Khalida Mukhtar Saleem Siddiqui

Imatinib mesylate is a BCR-ABL tyrosine kinase inhibitor used in the management of chronic myeloid leukemia. It is a safe and well-tolerated agent with a few manageable side effects. We are reporting a case of imatinib-related fatal bone marrow aplasia after complete cytogenetic response.

Journal: :Cancer research 1986
J L Wilmer G L Erexson A D Kligerman

Cyclophosphamide (CP) is metabolized to the reactive intermediates, phosphoramide mustard (PAM) and acrolein (AC), which have generally different molecular binding targets. Sodium 2-mercaptoethanesulfonic acid (MESNA) has been used clinically to alleviate hemorrhagic cystitis caused by CP chemotherapy, has exhibited anticarcinogenic effects in rats exposed to CP during a long-term bioassay, and...

Journal: :Blood 2003
Jorge E Cortes Moshe Talpaz Francis Giles Susan O'Brien Mary Beth Rios Jianqin Shan Guillermo Garcia-Manero Stefan Faderl Deborah A Thomas William Wierda Allessandra Ferrajoli Sima Jeha Hagop M Kantarjian

Cytogenetic clonal evolution (CE) is a known poor prognostic factor in Philadelphia chromosome-positive chronic myelogenous leukemia (Ph-positive CML). However, its prognostic relevance in the era of imatinib therapy is unknown. We investigated the independent prognostic relevance of CE in 498 patients with Ph-positive CML treated with imatinib for chronic or accelerated phases. One hundred twe...

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