نتایج جستجو برای: xrcc5

تعداد نتایج: 319  

2015
Yao Xiao Jingshu Wang Yu Qin Yang Xuan Yunlu Jia Wenxian Hu Wendan Yu Meng Dai Zhenglin Li Canhui Yi Shilei Zhao Mei Li Sha Du Wei Cheng Xiangsheng Xiao Yiming Chen Taihua Wu Songshu Meng Yuhui Yuan Quentin Liu Wenlin Huang Wei Guo Shusen Wang Wuguo Deng

Cyclooxygenase-2 (COX-2) plays an important role in lung cancer development and progression. Using streptavidin-agarose pulldown and proteomics assay, we identified and validated Ku80, a dimer of Ku participating in the repair of broken DNA double strands, as a new binding protein of the COX-2 gene promoter. Overexpression of Ku80 up-regulated COX-2 promoter activation and COX-2 expression in l...

2015
SHUAI WANG ZHOU WANG YU YANG MO SHI ZHENGUO SUN

Ku80, a subunit of the heterodymeric Ku protein, is clearly implicated in nonhomologous end joining DNA repair, chemoresistance and radioresistance in malignant tumors. In the present study, the clinicopathological significance of Ku80 in esophageal squamous cell carcinoma (ESCC) was investigated. The expression levels of Ku80 were determined by reverse transcription-quantitative polymerase cha...

2008
Prafulla Raval Aleksei N. Kriatchko Sushil Kumar Patrick C. Swanson

Antigen receptor genes are assembled by a site-specific DNA rearrangement process called V(D)J recombination. This process proceeds through two distinct phases: a cleavage phase in which the RAG1 and RAG2 proteins introduce DNA double-strand breaks at antigen receptor gene segments, and a joining phase in which the resulting DNA breaks are processed and repaired via the non-homologous end-joini...

2008
Wael Y. Mansour Sabine Schumacher Raphael Rosskopf Tim Rhein Filip Schmidt-Petersen Fruszina Gatzemeier Friedrich Haag Kerstin Borgmann Henning Willers Jochen Dahm-Daphi

In mammalian cells, DNA double-strand breaks (DSBs) are repaired by three pathways, nonhomologous end-joining (NHEJ), gene conversion (GC) and single-strand annealing (SSA). These pathways are distinct with regard to repair efficiency and mutagenic potential and must be tightly controlled to preserve viability and genomic stability. Here, we employed chromosomal reporter constructs to character...

Journal: :Journal of immunology 2002
William Rodgers Stephen J Jordan J Donald Capra

The autoantigen Ku, composed of subunits Ku70 and Ku86, is necessary for repair of DNA double-strand breaks by nonhomologous end joining. Similarly, Ku participates in repair of DNA double-strand breaks that occur during V(D)J recombination, and it is therefore required for the development of B and T lymphocytes. Although previous studies have identified the DNA-binding activities of Ku, little...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2000
Y Gu J Sekiguchi Y Gao P Dikkes K Frank D Ferguson P Hasty J Chun F W Alt

Mammalian nonhomologous DNA end joining employs Ku70, Ku80, DNA-dependent protein kinase catalytic subunit (DNA-PKcs), XRCC4, and DNA ligase IV (Lig4). Herein, we show that Ku70 and Ku80 deficiency but not DNA-PKcs deficiency results in dramatically increased death of developing embryonic neurons in mice. The Ku-deficient phenotype is qualitatively similar to, but less severe than, that associa...

2011
Hideaki Ogiwara Takashi Kohno

Non-homologous end joining (NHEJ) is a major pathway for the repair of DNA double strand break (DSBs) with incompatible DNA ends, which are often generated by ionizing irradiation. In vitro reconstitution studies have indicated that NHEJ of incompatible DNA ends requires not only the core steps of synapsis and ligation, employing KU80/DNA-PKcs and LIG4, but also additional DNA end processing st...

Journal: :Cancer research 2008
Valerie B Holcomb Francis Rodier YongJun Choi Rita A Busuttil Hannes Vogel Jan Vijg Judith Campisi Paul Hasty

Ku80 facilitates DNA repair and therefore should suppress cancer. However, ku80(-/-) mice exhibit reduced cancer, although they age prematurely and have a shortened life span. We tested the hypothesis that Ku80 deletion suppresses cancer by enhancing cellular tumor-suppressive responses to inefficiently repaired DNA damage. In support of this hypothesis, Ku80 deletion ameliorated tumor burden i...

Journal: :FEBS letters 2000
K Song D Jung Y Jung S G Lee I Lee

Ku, a heterodimer of 70- and 80-kDa subunits, plays a general role in the metabolism of DNA ends in eukaryotic cells, including double-strand DNA break repair, V(D)J recombination, and maintenance of telomeres. We have utilized the yeast two-hybrid system to identify Ku70-interacting proteins other than Ku80. Two reactive clones were found to encode the dimerization domain of TRF2, a mammalian ...

2014
Carole Beck Christian Boehler Josée Guirouilh Barbat Marie-Elise Bonnet Giuditta Illuzzi Philippe Ronde Laurent R. Gauthier Najat Magroun Anbazhagan Rajendran Bernard S. Lopez Ralph Scully François D. Boussin Valérie Schreiber Françoise Dantzer

The repair of toxic double-strand breaks (DSB) is critical for the maintenance of genome integrity. The major mechanisms that cope with DSB are: homologous recombination (HR) and classical or alternative nonhomologous end joining (C-NHEJ versus A-EJ). Because these pathways compete for the repair of DSB, the choice of the appropriate repair pathway is pivotal. Among the mechanisms that influenc...

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