نتایج جستجو برای: protease inhibitor

تعداد نتایج: 245560  

Journal: :The lancet. HIV 2016
Kathleen Squires Cissy Kityo Sally Hodder Margaret Johnson Evgeny Voronin Debbie Hagins Anchalee Avihingsanon Ellen Koenig Shuping Jiang Kirsten White Andrew Cheng Javier Szwarcberg Huyen Cao

BACKGROUND Women are under-represented in HIV antiretroviral therapy (ART) studies. Guidelines for selection of ART as initial therapy in patients with HIV-1 infection do not contain sex-specific treatment. We aimed to assess the safety and efficacy of the single tablet integrase inhibitor regimen containing elvitegravir, cobicistat, emtricitabine, and tenofovir disoproxil fumarate compared wit...

Journal: :Annals of internal medicine 1999
A R Zolopa R W Shafer A Warford J G Montoya P Hsu D Katzenstein T C Merigan B Efron

BACKGROUND Tests for resistance to HIV drugs are available for clinical use; however, their predictive value has not been fully assessed. OBJECTIVES To determine HIV-1 genotypic predictors of a virologic response to saquinavir-ritonavir therapy in patients in whom at least one previous protease inhibitor-containing regimen had failed and to compare the predictive value of baseline genotype wi...

Journal: :The Journal of biological chemistry 1987
J Sugatani M Miwa D J Hanahan

The serine protease inhibitors diethyl p-nitrophenyl phosphate and phenylmethylsulfonyl fluoride (chemical modifiers of serine residue) and N-acetyl-l-tryptophan ethyl ester (competitive inhibitor of chymotryptic protease) inhibited 1-O-alkyl-2-acetyl-sn-glycero-3-phosphocholine (AGEPC; platelet-activating factor)-induced platelet aggregation and secretion. The inhibition was dependent on the p...

Journal: :Angewandte Chemie 2021

We present the results of classical and QM/MM simulations for inhibition SARS-CoV-2 3CL protease by a hydroxymethylketone inhibitor, PF-00835231. In noncovalent complex carbonyl oxygen atom warhead is placed in oxyanion hole formed residues 143 to 145, while P1–P3 groups are accommodated active site with interactions similar those observed peptide substrate. According alchemical free energy cal...

Journal: :Antimicrobial agents and chemotherapy 2002
Rami Kantor W Jeffrey Fessel Andrew R Zolopa Dennis Israelski Nancy Shulman Jose G Montoya Michael Harbour Jonathan M Schapiro Robert W Shafer

In order to track the evolution of primary protease inhibitor (PI) resistance mutations in human immunodeficiency virus type 1 (HIV-1) isolates, baseline and follow-up protease sequences were obtained from patients undergoing salvage PI therapy who presented initially with isolates containing a single primary PI resistance mutation. Among 78 patients meeting study selection criteria, baseline p...

Mohammad Taghi Goodarzi,

Alpha-1-proteinase inhibitor (API) is one of the acute phase proteins. Following an inflammatory stimuli the concentration of API increased up to four folds. Accompanying these quantitative changes, there is qualitative alterations in the structure of carbohydrate moiety (glycosylation). To determine the alterations in the glycosylation of API in inflammation, API was isolated from the sera of...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2003
Haitao Yang Maojun Yang Yi Ding Yiwei Liu Zhiyong Lou Zhe Zhou Lei Sun Lijuan Mo Sheng Ye Hai Pang George F Gao Kanchan Anand Mark Bartlam Rolf Hilgenfeld Zihe Rao

A newly identified severe acute respiratory syndrome coronavirus (SARS-CoV), is the etiological agent responsible for the outbreak of SARS. The SARS-CoV main protease, which is a 33.8-kDa protease (also called the 3C-like protease), plays a pivotal role in mediating viral replication and transcription functions through extensive proteolytic processing of two replicase polyproteins, pp1a (486 kD...

Journal: :Applied and environmental microbiology 2013
Páraic O Cuív Rajesh Gupta Hareshwar P Goswami Mark Morrison

Clostridium thermocellum encodes a cellulosomal, modular, and thermostable serine protease inhibitor (serpin), PinA. PinA stability but not inhibitory activity is affected by the Fn(III) and Doc(I) domains, and PinA is a broad inhibitor of subtilisin-like proteases and may play a key role in protecting the cellulosome from protease attack.

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