نتایج جستجو برای: lung injury

تعداد نتایج: 605251  

Journal: :American journal of physiology. Lung cellular and molecular physiology 2009
Raj Wadgaonkar Vipul Patel Natalia Grinkina Carol Romano Jing Liu Yutong Zhao Saad Sammani Joe G N Garcia Viswanathan Natarajan

Two mammalian sphingosine kinase (SphK) isoforms, SphK1 and SphK2, possess identical kinase domains but have distinct kinetic properties and subcellular localizations, suggesting each has one or more specific roles in sphingosine-1-phosphate (S1P) generation. Although both kinases use sphingosine as a substrate to generate S1P, the mechanisms controlling SphK activation and subsequent S1P gener...

2009
Huili Zhang Madhav Bhatia

Accumulating evidence has suggested that hydrogen sulfide (H2S) naturally occurs during cysteine metabolism in many types of mammalian cells. Since H2S exhibits vasodilator activity and plays an important role in nervous system and inflammatory diseases, it is currently considered to be the third gaseous mediator. Recently, more and more attention has been paid to the biological functions of H2...

2010
B. Kilpatrick P. Slinger

Key points † Mechanical ventilation can have adverse effects on pulmonary function by several distinct mechanisms. † Patients undergoing one-lung ventilation or cardiopulmonary bypass are at increased risk of developing acute lung injury (ALI). † Protective ventilatory strategies may prevent or reduce ALI. † There is a lack of randomized controlled trials to guide optimal intra-operative ventil...

2012
Jae Hyang Lim Hirofumi Jono Kensei Komatsu Chang-Hoon Woo Jiyun Lee Masanori Miyata Takashi Matsuno Xiangbin Xu Yuxian Huang Wenhong Zhang Soo Hyun Park Yu-Il Kim Yoo-Duk Choi Huahao Shen Kyung-Sun Heo Haodong Xu Patricia Bourne Tomoaki Koga Haidong Xu Chen Yan Binghe Wang Lin-Feng Chen Xin-Hua Feng Jian-Dong Li

Lung injury, whether induced by infection or caustic chemicals, initiates a series of complex wound-healing responses. If uncontrolled, these responses may lead to fibrotic lung diseases and loss of function. Thus, resolution of lung injury must be tightly regulated. The key regulatory proteins required for tightly controlling the resolution of lung injury have yet to be identified. Here we sho...

Journal: :Journal of applied toxicology : JAT 2006
Shannon D McClintock Laszlo M Hoesel Salil K Das Gerd O Till Thomas Neff Robin G Kunkel Milton G Smith Peter A Ward

Airway instillation into rats of 2-chloroethyl ethyl sulfide (CEES), the half molecule of sulfur mustard compound, results in acute lung injury, as measured by the leak of plasma albumin into the lung. Morphologically, early changes in the lung include alveolar hemorrhage and fibrin deposition and the influx of neutrophils. Following lung contact with CEES, progressive accumulation of collagen ...

2014
Cassiano Felippe Gonçalves-de-Albuquerque Patrícia Burth Adriana Ribeiro Silva Isabel Matos Medeiros de Moraes Flora Magno de Jesus Oliveira Ricardo Erthal Santelli Aline Soares Freire Gerson Silva de Lima Emilson Domingos da Silva Camila Ignácio da Silva Verônica Morandi Patrícia Torres Bozza Mauricio Younes-Ibrahim Hugo Caire de Castro Faria Neto Mauro Velho de Castro Faria

BACKGROUND Leptospiral glycolipoprotein (GLP) is a potent and specific Na/K-ATPase inhibitor. Severe pulmonary form of leptospirosis is characterized by edema, inflammation and intra-alveolar hemorrhage having a dismal prognosis. Resolution of edema and inflammation determines the outcome of lung injury. Na/K-ATPase activity is responsible for edema clearance. This enzyme works as a cell recept...

Journal: :American journal of physiology. Lung cellular and molecular physiology 2006
Brett A Simon R Blaine Easley Dmitry N Grigoryev Shwu-Fan Ma Shui Q Ye Tera Lavoie Rubin M Tuder Joe G N Garcia

Human acute lung injury is characterized by heterogeneous tissue involvement, leading to the potential for extremes of mechanical stress and tissue injury when mechanical ventilation, required to support critically ill patients, is employed. Our goal was to establish whether regional cellular responses to these disparate local mechanical conditions could be determined as a novel approach toward...

Journal: :American journal of respiratory cell and molecular biology 2000
S A McDowell K Gammon C J Bachurski J S Wiest J E Leikauf D R Prows G D Leikauf

Acute lung injury, an often fatal condition, can result from a wide range of insults leading to a complex series of biologic responses. Despite extensive research, questions remain about the interplay of the factors involved and their role in acute lung injury. We proposed that assessing the temporal and functional relationships of differentially expressed genes after pulmonary insult would rev...

2018
Julie Wagner Karl M Strosing Sashko G Spassov Ziwei Lin Helen Engelstaedter Sabine Tacke Alexander Hoetzel Simone Faller

Mechanical ventilation is a life-saving clinical treatment but it can induce or aggravate lung injury. New therapeutic strategies, aimed at reducing the negative effects of mechanical ventilation such as excessive production of reactive oxygen species, release of pro-inflammatory cytokines, and transmigration as well as activation of neutrophil cells, are needed to improve the clinical outcome ...

2017
José Roberto Fioretto Susiane Oliveira Klefens Rafaelle Fernandes Pires Cilmery Suemi Kurokawa Mario Ferreira Carpi Rossano César Bonatto Marcos Aurélio Moraes Carlos Fernando Ronchi

OBJECTIVE To compare the effects of high-frequency oscillatory ventilation and conventional protective mechanical ventilation associated with the prone position on oxygenation, histology and pulmonary oxidative damage in an experimental model of acute lung injury. METHODS Forty-five rabbits with tracheostomy and vascular access were underwent mechanical ventilation. Acute lung injury was indu...

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