نتایج جستجو برای: iranian novel mutation

تعداد نتایج: 1082005  

Journal: :international journal of molecular and cellular medicine 0
shahram torkamandi department of medical genetics, faculty of medicine, shahid beheshti university of medical sciences, tehran, iran.سازمان اصلی تایید شده: دانشگاه علوم پزشکی شهید بهشتی (shahid beheshti university of medical sciences) milad gholami department of medical genetics, faculty of medicine, shahid beheshti university of medical sciences, tehran, iran.سازمان اصلی تایید شده: دانشگاه علوم پزشکی شهید بهشتی (shahid beheshti university of medical sciences) javad mohammadi asl ahvaz jundishapour university of medical sciences, ahvaz, iran.سازمان های دیگر: . noor genetics laboratory, ahvaz, iran somaye rezaie department of neurology, imam hossein hospital, shahid beheshti university of medical sciences, tehran, iran.سازمان اصلی تایید شده: دانشگاه علوم پزشکی شهید بهشتی (shahid beheshti university of medical sciences) mohammad ali zaimy department of medical genetics, faculty of medicine, tehran university of medical sciences, tehran, iran.سازمان اصلی تایید شده: دانشگاه علوم پزشکی تهران (tehran university of medical sciences) mir davood omrani department of medical genetics, faculty of medicine, shahid beheshti university of medical sciences, tehran, iran.سازمان اصلی تایید شده: دانشگاه علوم پزشکی شهید بهشتی (shahid beheshti university of medical sciences)

hypohidrotic ectodermal dysplasia (hed) is a rare congenital disorder arising from deficient development of ectoderm-derived structures including skin, nails, glands and teeth. the phenotype of hed is associated with mutation in eda, edar, edaradd and nemo genes, all of them disruptingnf-κb signaling cascade necessary for initiation, formation and differentiation in the embryo and adult. here w...

2014
Kolsoum InanlooRahatloo Amir Farhang Zand Parsa Klaus Huse Paniz Rasooli Saeid Davaran Matthias Platzer Marcel Kramer Jian-Bing Fan Casey Turk Sasan Amini Frank Steemers Kevin Gunderson Mostafa Ronaghi Elahe Elahi

We aimed to identify the genetic cause of coronary artery disease (CAD) in an Iranian pedigree. Genetic linkage analysis identified three loci with an LOD score of 2.2. Twelve sequence variations identified by exome sequencing were tested for segregation with disease. A p.Val99Met causing mutation in ST6GALNAC5 was considered the likely cause of CAD. ST6GALNAC5 encodes sialyltransferase 7e. The...

Wolfram syndrome (WS) is a rare autosomal recessive neurodegenerative disease with variable symptoms, including neuropsychiatric manifestations. A 26-year-old man was reported with classic symptoms of WS and repetitive psychiatric hospitalizations and at least 16 suicidal attempts. The genetic study demonstrated a novel homozygous stop-codon mutation on the WFS1 gene. This special type of mutat...

2015
A Ghotaslou F Nadali B Chahardouli N Alizad Ghandforosh SH Rostami K Alimoghaddam A Ghavamzadeh

BACKGROUND Myeloproliferative disorders are a group of diseases characterized by increased proliferation of myeloid lineage. In addition to JAK2V617F mutation, several mutations in the c-MPL gene have been reported in patients with philadelphia-negative chronic myeloproliferative disorders that could be important in the pathogenesis of diseases. The aim of the present study was to investigate t...

2011
Javad Tavakkoly-Bazzaz Ozra Tabatabaei-Malazy Mohammad Tajmir-Riahi Daryoosh Javidi Manizhe Izadi Maryam Shahrabi-Farahani Parvin Amiri Mahsa M. Amoli

OBJECTIVE Klotho has an important role in development of coronary artery (CAD) disease. A functional variant of klotho gene (kl-vs) has been found as an independent risk factor for early-onset occult coronary artery disease (CAD) in previous studies. The Frequency of this variant was not known in Iranian population. We have examined the allele frequency of the kl-vs variant in a case-control st...

2011
Narges Zali Seyed Reza Mohebbi Sahar Esteghamat Mohsen Chiani Mahdi Montazer Haghighi Seyed Mohammad-Kazem Hosseini-Asl Faramarz Derakhshan Amir-Houshang Mohammad-Alizadeh Seyed-Ali Malek-Hosseini Mohammad Reza Zali

BACKGROUND Wilson disease (WD) is an autosomal recessive disorder. The WD gene, ATP7B, encodes a copper-transporting ATPase involved in the transport of copper into the plasma protein ceruloplasmin and in excretion of copper from the liver. ATP7B mutations cause copper to accumulate in the liver and brain. OBJECTIVES We examined the ATP7B mutation spectrum in Wilson disease patients in Iran. ...

Journal: :International journal of pediatric otorhinolaryngology 2012
Behzad Davarnia Mojgan Babanejad Zohreh Fattahi Nooshin Nikzat Niloofar Bazazzadegan Akbar Pirzade Reza Farajollahi Carla Nishimura Khadijeh Jalalvand Sanaz Arzhangi Kimia Kahrizi Richard J H Smith Hossein Najmabadi

OBJECTIVE Hereditary hearing impairment is a genetically heterogeneous disorder. In spite of this, mutations in the GJB2 gene, encoding connexin 26 (Cx26), are a major cause of nonsyndromic recessive hearing loss in many countries and are largely dependent on ethnic groups. The purpose of our study was to characterize the type and prevalence of GJB2 mutations among Azeri population of Iran. M...

Journal: :Genetic Testing and Molecular Biomarkers 2012

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