نتایج جستجو برای: il 22

تعداد نتایج: 347112  

Journal: :American journal of physiology. Gastrointestinal and liver physiology 2006
Stephan Brand Florian Beigel Torsten Olszak Kathrin Zitzmann Sören T Eichhorst Jan-Michel Otte Helmut Diepolder Andreas Marquardt Wolfgang Jagla Andreas Popp Stéphane Leclair Karin Herrmann Julia Seiderer Thomas Ochsenkühn Burkhard Göke Christoph J Auernhammer Julia Dambacher

IL-22 is produced by activated T cells and signals through a receptor complex consisting of IL-22R1 and IL-10R2. The aim of this study was to analyze IL-22 receptor expression, signal transduction, and specific biological functions of this cytokine system in intestinal epithelial cells (IEC). Expression studies were performed by RT-PCR. Signal transduction was analyzed by Western blot experimen...

Journal: :Diabetes 2014
Elise Dalmas Nicolas Venteclef Charles Caer Christine Poitou Isabelle Cremer Judith Aron-Wisnewsky Sébastien Lacroix-Desmazes Jagadeesh Bayry Srinivas V Kaveri Karine Clément Sébastien André Michèle Guerre-Millo

Proinflammatory cytokines are critically involved in the alteration of adipose tissue biology leading to deterioration of glucose homeostasis in obesity. Here we show a pronounced proinflammatory signature of adipose tissue macrophages in type 2 diabetic obese patients, mainly driven by increased NLRP3-dependent interleukin (IL)-1β production. IL-1β release increased with glycemic deterioration...

ژورنال: طب جانباز 2013
جمالی, داوود, رضایی, عباس, سجادی, معصومه, سروش, محمدرضا, شریف نیا, زرین, غضنفری, طوبی, پورفرزام, شهریار, کاوندی, الهه,

مقدمه: یکی از عوارض دیررس عمده ناشی از مواجهه با سولفورموستارد، عوارض ریوی است. به نظر می رسد که پاسخ های ایمنی و التهابی در ایجاد عوارض کلینیکی نقش دارند.IL-22 عضوی از سایتوکاین های خانوادهIL-10 است که نقش آن در بسیاری از بیماری های التهابی ریوی به اثبات رسیده است.هدف این مطالعه، بررسی رابطه سطح سرمی IL-22 و عوارض دیررس ریوی جانبازان شیمیایی مواجهه یافته با سولفور موستارد بود. مواد و روش ها: د...

2010
Robert Sabat Ellen Witte Sylke Schneider-Burrus Katrin Witte Kerstin Wolk Katarzyna Warszawska Conny Hoeflich Stefanie Kunz Annette Buss Hans Joachim Roewert Markus Krause Ansgar Lukowsky Hans-Dieter Volk Wolfram Sterry

Overexpression of the T cell cytokine IL-22 is linked to the development of some chronic diseases, but little is known about IL-22 deficiency in humans. As demonstrated in this study, acne inversa (AI; also designated as Hidradenitis suppurativa) lesions show a relative deficiency of IL-22 and IL-20, but not of IL-17A, IL-26, IFN-g, IL-24, or IL-1b. Moreover, AI lesions had reduced expression o...

Journal: :Frontiers in immunology 2015
Wei Shen Julie A. Hixon Mairi H. McLean Wen Qing Li Scott K. Durum

IL-22 has multiple activities ranging from tissue repair to inflammation. To characterize the pathogenicity and plasticity of cells that produce IL-22, a novel reporter mouse strain was generated. Homeostatic IL-22 reporter expression was observed in intestinal lymphoid cells identified as CD4 T cells and ILC3 cells. In a model of inflammatory bowel disease, CD4 T cells strongly expressed the I...

2016
Tegan N. Lavoie Wendy C. Carcamo Arun Wanchoo Ashok Sharma Afife Gulec Kathleen M. Berg Carol M. Stewart Cuong Q. Nguyen

TH17 cells and their associated signature cytokines, IL-17 and IL-22, are highly elevated in primary Sjögren's syndrome (pSjS). The levels of IL-22 present in sera showed significant correlations with many disease parameters, specifically hyposalivation, anti-SSB, anti-SSA/SSB, hypergammaglobulinemia and rheumatoid factor. The present study aims to examine the biological function of IL-22 on hu...

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