Ideally, a successful kidney transplantation corrects the abnormalities in mineral metabolism that, during uraemia, lead to secondary hyperparathyroidism (HPT) and renal osteodystrophy. This includes reversal of uraemia, abolition of hypocalcaemia, hyperphosphataemia and acidosis, and restoration of calcitriol production and reversal of skeletal resistance to parathyroid hormone (PTH) and vitam...